Type 1 Diabetes in STAT Protein Family Mutations: Regulating the Th17/Treg Equilibrium and Beyond.


Journal

Diabetes
ISSN: 1939-327X
Titre abrégé: Diabetes
Pays: United States
ID NLM: 0372763

Informations de publication

Date de publication:
02 2019
Historique:
received: 12 06 2018
accepted: 11 11 2018
entrez: 23 1 2019
pubmed: 23 1 2019
medline: 6 8 2019
Statut: ppublish

Résumé

Improvements in the immunological, molecular, and genetic technologies such as next-generation sequencing have led to an exponential increase in the number of monogenic immune dysregulatory syndromes diagnosed, where type 1 diabetes (T1D) forms part of the autoimmune manifestations. Here, we reviewed the mutations in the signal transducer and activator of transcription (STAT) protein family, namely gain-of-function (GOF) mutations in

Identifiants

pubmed: 30665954
pii: 68/2/258
doi: 10.2337/db18-0627
doi:

Substances chimiques

STAT1 Transcription Factor 0
STAT3 Transcription Factor 0

Types de publication

Journal Article

Langues

eng

Pagination

258-265

Informations de copyright

© 2019 by the American Diabetes Association.

Auteurs

Marco Fabbri (M)

Division of Immunology, Transplantation and Infectious Diseases, Diabetes Research Institute, IRCCS San Raffaele Scientific Institute, Milan, Italy.
Vita-Salute San Raffaele University, Milan, Italy.

Mikaela Frixou (M)

School of Medicine, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow, U.K.

Massimo Degano (M)

Biocrystallography Unit, Division of Immunology, Transplantation and Infectious Diseases, IRCCS San Raffaele Scientific Institute, Milan, Italy.

Georgia Fousteri (G)

Division of Immunology, Transplantation and Infectious Diseases, Diabetes Research Institute, IRCCS San Raffaele Scientific Institute, Milan, Italy fousteri.georgia@hsr.it.

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Classifications MeSH