Decidual Interleukin-22-Producing CD4+ T Cells (Th17/Th0/IL-22+ and Th17/Th2/IL-22+, Th2/IL-22+, Th0/IL-22+), Which Also Produce IL-4, Are Involved in the Success of Pregnancy.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
19 Jan 2019
Historique:
received: 18 12 2018
revised: 16 01 2019
accepted: 17 01 2019
entrez: 24 1 2019
pubmed: 24 1 2019
medline: 2 5 2019
Statut: epublish

Résumé

Trophoblast expressing paternal HLA-C resembles a semiallograft, and could be rejected by maternal T cells. IL-22 seems to be involved in allograft rejection and thus could be responsible for miscarriages. We examined the role of decidual IL-22-producing CD4+ T on human pregnancy. In those experiencing successful pregnancy and those experiencing unexplained recurrent abortion (URA), the levels of IL-22 produced by decidual CD4+ T cells are higher than those of peripheral blood T cells. We found a correlation of IL-22 and IL-4 produced by decidual CD4+ T cells in those experiencing successful pregnancy, not in those experiencing URA. The correlation of IL-22 and IL-4 was also found in the serum of successful pregnancy. A prevalence of CD4+ T cells producing IL-22 and IL-4 (Th17/Th2/IL-22+, Th17/Th0/IL-22+, Th17/Th2/IL-22+, and Th0/IL-22+ cells) was observed in decidua of those experiencing successful pregnancy, whereas Th17/Th1/IL-22+ cells, which do not produce IL-4, are prevalent in those experiencing URA. Th17/Th2/IL-22+ and Th17/Th0/IL-22+ cells are exclusively present at the embryo implantation site where IL-4, GATA-3, IL-17A, ROR-C, IL-22, and AHR mRNA are expressed. T-bet and IFN-γ mRNA are found away from the implantation site. There is no pathogenic role of IL-22 when IL-4 is also produced by decidual CD4+ cells. Th17/Th2/IL-22+ and Th17/Th0/IL-22+ cells seem to be crucial for embryo implantation.

Identifiants

pubmed: 30669479
pii: ijms20020428
doi: 10.3390/ijms20020428
pmc: PMC6359245
pii:
doi:

Substances chimiques

Biomarkers 0
Cytokines 0
Interleukins 0
Interleukin-4 207137-56-2

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : European NoE EMBIC
ID : LSHM-CT-2004-512040
Organisme : Intramural Scientific Research (ex-60%) of the University of Florence
ID : RICATEN18

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Auteurs

Federica Logiodice (F)

Department of Experimental and Clinical Medicine and DENOTHE Excellence Center, University of Florence, 50134 Florence, Italy. federica_logiodice@yahoo.it.

Letizia Lombardelli (L)

Department of Experimental and Clinical Medicine and DENOTHE Excellence Center, University of Florence, 50134 Florence, Italy. letizialombardelli@gmail.com.

Ornela Kullolli (O)

Department of Experimental and Clinical Medicine and DENOTHE Excellence Center, University of Florence, 50134 Florence, Italy. ornelahysa@hotmail.it.

Herman Haller (H)

Department of Gynecology and Obstetrics, Medical Faculty, University of Rijeka, 51000 Rijeka, Croatia. herman.haller@medri.uniri.hr.

Enrico Maggi (E)

Department of Experimental and Clinical Medicine and DENOTHE Excellence Center, University of Florence, 50134 Florence, Italy. enrico.maggi@unifi.it.
Immunology Area, IRCCS Bambino Gesù Children's Hospital, 00165 Rome, Italy. enrico.maggi@unifi.it.

Daniel Rukavina (D)

Department of Physiology and Immunology, Medical Faculty, University of Rijeka, 51000 Rijeka, Croatia. rimed@hazu.hr.

Marie-Pierre Piccinni (MP)

Department of Experimental and Clinical Medicine and DENOTHE Excellence Center, University of Florence, 50134 Florence, Italy. mppiccinni@hotmail.com.

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