Selective inhibition of mitochondrial sodium-calcium exchanger protects striatal neurons from α-synuclein plus rotenone induced toxicity.
Journal
Cell death & disease
ISSN: 2041-4889
Titre abrégé: Cell Death Dis
Pays: England
ID NLM: 101524092
Informations de publication
Date de publication:
28 01 2019
28 01 2019
Historique:
received:
20
08
2018
accepted:
28
11
2018
revised:
14
11
2018
entrez:
30
1
2019
pubmed:
30
1
2019
medline:
22
4
2020
Statut:
epublish
Résumé
Progressive accumulation of α-synuclein (α-syn) and exposure to environmental toxins are risk factors that may both concur to Parkinson's disease (PD) pathogenesis. Electrophysiological recordings of field postsynaptic potentials (fEPSPs) and Ca
Identifiants
pubmed: 30692508
doi: 10.1038/s41419-018-1290-6
pii: 10.1038/s41419-018-1290-6
pmc: PMC6349907
doi:
Substances chimiques
Sodium-Calcium Exchanger
0
alpha-Synuclein
0
Rotenone
03L9OT429T
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
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