Rational drug design for androgen receptor and glucocorticoids receptor dual antagonist.


Journal

European journal of medicinal chemistry
ISSN: 1768-3254
Titre abrégé: Eur J Med Chem
Pays: France
ID NLM: 0420510

Informations de publication

Date de publication:
15 Mar 2019
Historique:
received: 09 10 2018
revised: 15 01 2019
accepted: 15 01 2019
pubmed: 4 2 2019
medline: 27 3 2019
entrez: 4 2 2019
Statut: ppublish

Résumé

Prostate cancer (PCa) is the most frequently diagnosed male malignant tumor and remains the second leading cause of male cancer mortality in the western countries. The second-generation antiandrogen enzalutamide (ENZa) can prolong survival time for patients with mCRPC. However, the overexpression of glucocorticoids receptor (GR) in mCRPC cells causes the resistance of antiandrogen and leads to the failure of androgen receptor (AR) targeting therapy. Herein, based on the chemical structures of antiandrogen and crystal structure of GR, we set up to develop GR/AR (GR and AR) dual antagonist by virtual screening and biological evaluation. We identified Z19 as a dual AR/GR antagonist. Z19 inhibited the transcription activity of both AR and GR, reducing both protein and mRNA level of the downstream proteins of GR and AR signaling, and provided a potential lead compound for the development of novel treatment agents of prostate cancer. Our work demonstrates that rational drug design is an efficient strategy in development of the GR/AR dual antagonist for the treatment of prostate cancer.

Identifiants

pubmed: 30711833
pii: S0223-5234(19)30046-7
doi: 10.1016/j.ejmech.2019.01.036
pii:
doi:

Substances chimiques

Androgen Receptor Antagonists 0
RNA, Messenger 0
Receptors, Androgen 0
Receptors, Glucocorticoid 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

232-242

Informations de copyright

Copyright © 2019 Elsevier Masson SAS. All rights reserved.

Auteurs

Meng Wu (M)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China; Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Yongli Xie (Y)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China; Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Xiangling Cui (X)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China; Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Chenchao Huang (C)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China.

Rongyu Zhang (R)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China.

Yang He (Y)

Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Xiaoyu Li (X)

Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Mingliang Liu (M)

Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China.

Shan Cen (S)

Institute of Medicinal Biotechnology, Chinese Academy of Medical Science, Beijing, China. Electronic address: shancen@imb.pumc.edu.cn.

Jinming Zhou (J)

Key Laboratory of the Ministry of Education for Advanced Catalysis Materials, Department of Chemistry, Zhejiang Normal University, 688 Yingbin Road, Jinhua, 321004, PR China. Electronic address: zhoujinming@zjnu.edu.cn.

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Classifications MeSH