Effect of Radiation on the Expression of CVD-related miRNAs, Inflammation and Endothelial Dysfunction of HUVECs.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Feb 2019
Historique:
received: 12 12 2018
revised: 21 01 2019
accepted: 22 01 2019
entrez: 4 2 2019
pubmed: 4 2 2019
medline: 21 3 2019
Statut: ppublish

Résumé

Radiotherapy (RT) can lead to cardiovascular disease (CVD). Evidence suggests that radiation modulates miRNA levels. Our purpose was to assess the acute response to radiation-induced modulation of the expression of miRNA-146a, miRNA-155, miRNA-221, and miRNA-222, inflammatory response and endothelial dysfunction on endothelial cells. Human umbilical vein endothelial cells (HUVECs) were exposed to 2 Gy RT, and intracellular levels of selected miRNAs were measured by real-time polymerase chain reaction at 2 and 24 h. Cytokine and adhesion molecule release were also assessed. Results showed that 2 Gy significantly increased the expression of miRNA-221 and miRNA-222, and reduced the level of miRNA-155 after 2 h; whereas miRNA-146a and miRNA-155 were significantly overexpressed and miRNA-222 was significantly down-regulated at 24 h. Interleukin-8 and soluble vascular cell adhesion molecule 1 levels were not affected by the studied RT. RT at 2 Gy modulated expression of selected miRNAs by endothelial cells after 2 and 24 h, which might be related to CVD development in patients who receive RT.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Radiotherapy (RT) can lead to cardiovascular disease (CVD). Evidence suggests that radiation modulates miRNA levels. Our purpose was to assess the acute response to radiation-induced modulation of the expression of miRNA-146a, miRNA-155, miRNA-221, and miRNA-222, inflammatory response and endothelial dysfunction on endothelial cells.
MATERIALS AND METHODS METHODS
Human umbilical vein endothelial cells (HUVECs) were exposed to 2 Gy RT, and intracellular levels of selected miRNAs were measured by real-time polymerase chain reaction at 2 and 24 h. Cytokine and adhesion molecule release were also assessed.
RESULTS RESULTS
Results showed that 2 Gy significantly increased the expression of miRNA-221 and miRNA-222, and reduced the level of miRNA-155 after 2 h; whereas miRNA-146a and miRNA-155 were significantly overexpressed and miRNA-222 was significantly down-regulated at 24 h. Interleukin-8 and soluble vascular cell adhesion molecule 1 levels were not affected by the studied RT.
CONCLUSION CONCLUSIONS
RT at 2 Gy modulated expression of selected miRNAs by endothelial cells after 2 and 24 h, which might be related to CVD development in patients who receive RT.

Identifiants

pubmed: 30711956
pii: 39/2/771
doi: 10.21873/anticanres.13174
doi:

Substances chimiques

Cytokines 0
DNA, Complementary 0
MIRN146 microRNA, human 0
MIRN155 microRNA, human 0
MIRN221 microRNA, human 0
MIRN222 microRNA, human 0
MicroRNAs 0
Vascular Cell Adhesion Molecule-1 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

771-780

Informations de copyright

Copyright© 2019, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Roser Esplugas (R)

Physiology Unit, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.
Laboratory of Toxicology and Environmental Health, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.

Montserrat Bellés (M)

Physiology Unit, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.
Laboratory of Toxicology and Environmental Health, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.

Noemí Serra (N)

Laboratory of Toxicology and Environmental Health, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.

Meritxell Arenas (M)

Radiation Oncology Department, Sant Joan University Hospital, IISPV, Rovira i Virgili University, Reus, Spain.

Víctor Hernández (V)

Department of Medical Physics, Sant Joan University Hospital, IISPV, Rovira i Virgili University, Reus, Spain.

Joan Carles Vallvé (JC)

Research Unit on Lipids and Atherosclerosis, Sant Joan University Hospital, IISPV, Rovira i Virgili University, Reus, Spain jc.vallve@urv.cat.

Victoria Linares (V)

Physiology Unit, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.
Laboratory of Toxicology and Environmental Health, School of Medicine, IISPV, Rovira i Virgili University, Reus, Spain.

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Classifications MeSH