Phase II randomised study of maintenance treatment with bevacizumab or bevacizumab plus metronomic chemotherapy after first-line induction with FOLFOXIRI plus Bevacizumab for metastatic colorectal cancer patients: the MOMA trial.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
03 2019
Historique:
received: 06 10 2018
revised: 21 12 2018
accepted: 30 12 2018
pubmed: 9 2 2019
medline: 22 5 2020
entrez: 9 2 2019
Statut: ppublish

Résumé

Alternating induction and maintenance phases is a common strategy in metastatic colorectal cancer (mCRC). Metronomic chemotherapy (metroCT) may represent a well-tolerated chemotherapy backbone for maximising bevacizumab effect during maintenance. The MOMA trial was designed to compare metroCT plus bevacizumab versus bevacizumab alone as maintenance following 4 months of induction with FOLFOXIRI plus bevacizumab. In this phase II study, patients with unresectable mCRC were randomised to receive up to 8 cycles of FOLFOXIRI plus bevacizumab, followed by bevacizumab (arm A) or the same regimen followed by bevacizumab plus metroCT (capecitabine 500 mg/three times per day and cyclophosphamide 50 mg/die, arm B) until disease progression. The primary end-point was progression-free survival (PFS). According to the Rubinstein and Korn's design, to detect a hazard ratio[HR] of 0.75 favouring arm B, with 1 sided-alpha and beta errors of 15% and 80%, 173 events and 222 patients were required. Between May 2012 and March 2015, 232 patients, mostly with RAS (65%) or BRAF (9%) mutant tumours, were randomised in 16 Italian centres. At a median follow-up of 47.8 months, 210 and 164 progression and death events were registered. The primary end-point was not met. Median PFS was 10.3 and 9.4 months in arm B and A, respectively (HR: 0.94 [70% confidence interval {CI}: 0.82-1.09], p = 0.680). No significant differences were reported in terms of overall survival (OS) (median OS arm B/A: 22.5/28 months; HR: 1.16 [95%CI: 0.99-1.37], p = 0.336). Response rate with FOLFOXIRI plus bevacizumab was 63% (arm B/A: 58%/68%). In the liver-limited subgroup, the secondary resection rate was 49% (arm B/A: 45%/55%). The addition of metroCT to maintenance with bevacizumab does not significantly improve PFS of mCRC patients. The activity of FOLFOXIRI plus bevacizumab is confirmed in a population with high prevalence of RAS/BRAF mutations treated with a 4-months induction. www.clinicaltrials.gov NCT02271464.

Sections du résumé

BACKGROUND
Alternating induction and maintenance phases is a common strategy in metastatic colorectal cancer (mCRC). Metronomic chemotherapy (metroCT) may represent a well-tolerated chemotherapy backbone for maximising bevacizumab effect during maintenance. The MOMA trial was designed to compare metroCT plus bevacizumab versus bevacizumab alone as maintenance following 4 months of induction with FOLFOXIRI plus bevacizumab.
PATIENTS AND METHODS
In this phase II study, patients with unresectable mCRC were randomised to receive up to 8 cycles of FOLFOXIRI plus bevacizumab, followed by bevacizumab (arm A) or the same regimen followed by bevacizumab plus metroCT (capecitabine 500 mg/three times per day and cyclophosphamide 50 mg/die, arm B) until disease progression. The primary end-point was progression-free survival (PFS). According to the Rubinstein and Korn's design, to detect a hazard ratio[HR] of 0.75 favouring arm B, with 1 sided-alpha and beta errors of 15% and 80%, 173 events and 222 patients were required.
RESULTS
Between May 2012 and March 2015, 232 patients, mostly with RAS (65%) or BRAF (9%) mutant tumours, were randomised in 16 Italian centres. At a median follow-up of 47.8 months, 210 and 164 progression and death events were registered. The primary end-point was not met. Median PFS was 10.3 and 9.4 months in arm B and A, respectively (HR: 0.94 [70% confidence interval {CI}: 0.82-1.09], p = 0.680). No significant differences were reported in terms of overall survival (OS) (median OS arm B/A: 22.5/28 months; HR: 1.16 [95%CI: 0.99-1.37], p = 0.336). Response rate with FOLFOXIRI plus bevacizumab was 63% (arm B/A: 58%/68%). In the liver-limited subgroup, the secondary resection rate was 49% (arm B/A: 45%/55%).
CONCLUSIONS
The addition of metroCT to maintenance with bevacizumab does not significantly improve PFS of mCRC patients. The activity of FOLFOXIRI plus bevacizumab is confirmed in a population with high prevalence of RAS/BRAF mutations treated with a 4-months induction.
TRIAL REGISTRATION
www.clinicaltrials.gov NCT02271464.

Identifiants

pubmed: 30735919
pii: S0959-8049(19)30006-1
doi: 10.1016/j.ejca.2018.12.028
pii:
doi:

Substances chimiques

Bevacizumab 2S9ZZM9Q9V
Capecitabine 6804DJ8Z9U
Irinotecan 7673326042
Cyclophosphamide 8N3DW7272P
Leucovorin Q573I9DVLP
Fluorouracil U3P01618RT

Banques de données

ClinicalTrials.gov
['NCT02271464']

Types de publication

Clinical Trial, Phase II Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

175-182

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Chiara Cremolini (C)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Federica Marmorino (F)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Francesca Bergamo (F)

Medical Oncology Unit 1, Department of Clinical and Experimental Oncology, Veneto Institute of Oncology - IRCCS, Padova, Italy.

Giuseppe Aprile (G)

Department of Oncology, University and General Hospital, Udine, Italy; Department of Oncology, General Hospital, ULSS8 Berica - East District, 36100, Vicenza, Italy.

Lisa Salvatore (L)

U.O.C Oncologia, Fondazione Policlinico Universitario Agostino Gemelli IRCCS, Largo Agostino Gemelli 8, 00168 Roma, Italy.

Gianluca Masi (G)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Emanuela Dell'Aquila (E)

Department of Medical Oncology, University Campus Biomedico, Rome, Italy.

Carlotta Antoniotti (C)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Sabina Murgioni (S)

Medical Oncology Unit 1, Department of Clinical and Experimental Oncology, Veneto Institute of Oncology - IRCCS, Padova, Italy.

Giacomo Allegrini (G)

Unit of Medical Oncology, Livorno Hospital, Department of Medical Oncology, Azienda Toscana Nord Ovest, Livorno, Italy.

Beatrice Borelli (B)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Donatello Gemma (D)

Department of Medical Oncology, Hospital of Frosinone, Frosinone, Italy.

Mariaelena Casagrande (M)

Department of Oncology, University and General Hospital, Udine, Italy.

Cristina Granetto (C)

Department of Oncology, S. Croce and Carle Teaching Hospital, Cuneo, Italy.

Sara Delfanti (S)

Medical Oncology Unit, Fondazione IRCCS Policlinico San Matteo, Pavia, Italy.

Samantha Di Donato (S)

Medical Oncology Department, Nuovo Ospedale-Santo Stefano, Prato, Italy.

Marta Schirripa (M)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy; Medical Oncology Unit 1, Department of Clinical and Experimental Oncology, Veneto Institute of Oncology - IRCCS, Padova, Italy.

Elisa Sensi (E)

Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy.

Giuseppe Tonini (G)

Department of Medical Oncology, University Campus Biomedico, Rome, Italy.

Sara Lonardi (S)

Medical Oncology Unit 1, Department of Clinical and Experimental Oncology, Veneto Institute of Oncology - IRCCS, Padova, Italy.

Gabriella Fontanini (G)

Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy.

Luca Boni (L)

Clinical Trials Coordinating Center, Toscano Cancer Institute, University Hospital Careggi, Florence, Italy.

Alfredo Falcone (A)

Unit of Medical Oncology 2, Azienda Ospedaliero-Universitaria Pisana, Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy. Electronic address: alfredo.falcone@med.unipi.it.

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