Classic and Nonclassic Renin-Angiotensin Systems in the Critically Ill.
Acute Kidney Injury
/ drug therapy
Adult
Aged
Aged, 80 and over
Angiotensin I
/ therapeutic use
Angiotensin II
/ therapeutic use
Blood Pressure
/ physiology
Critical Illness
Female
Homeostasis
/ physiology
Humans
Male
Middle Aged
Peptide Fragments
/ therapeutic use
Renin-Angiotensin System
/ physiology
Respiratory Distress Syndrome
/ drug therapy
Shock, Septic
/ drug therapy
Vasoconstrictor Agents
/ therapeutic use
Acute kidney injury
Acute respiratory distress syndrome
Angiotensin
Angiotensin-converting enzyme
Inflammation
Renin
Sepsis
Septic shock
Journal
Critical care clinics
ISSN: 1557-8232
Titre abrégé: Crit Care Clin
Pays: United States
ID NLM: 8507720
Informations de publication
Date de publication:
Apr 2019
Apr 2019
Historique:
entrez:
21
2
2019
pubmed:
21
2
2019
medline:
14
6
2019
Statut:
ppublish
Résumé
Classic and nonclassic renin-angiotensin systems (RAS) are 2 sides of an ubiquitous endocrine/paracrine cascade regulating blood pressure and homeostasis. Angiotensin II and angiotensin-converting enzyme (ACE) levels are associated with severity of disease in the critically ill, and are central to the physiology and the pathogenesis of circulatory shock. Angiotensin (1-7) and ACE2 act as an endogenous counterregulatory arm to the angiotensin II/ACE axis. The tissue-based RAS has paracrine effects dissociated from those of the circulating RAS. Exogenous angiotensin II or ACE2 may improve the outcome of septic shock and acute respiratory distress syndrome, respectively.
Identifiants
pubmed: 30784605
pii: S0749-0704(18)30770-X
doi: 10.1016/j.ccc.2018.11.002
pmc: PMC7125612
pii:
doi:
Substances chimiques
Peptide Fragments
0
Vasoconstrictor Agents
0
Angiotensin II
11128-99-7
Angiotensin I
9041-90-1
angiotensin I (1-7)
IJ3FUK8MOF
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
213-227Informations de copyright
Copyright © 2018 Elsevier Inc. All rights reserved.
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