Potassium channel activity controls breast cancer metastasis by affecting β-catenin signaling.
Animals
Antineoplastic Agents
/ pharmacology
Cell Movement
/ drug effects
Cell Proliferation
/ drug effects
Cresols
/ pharmacology
ERG1 Potassium Channel
/ genetics
Epithelial-Mesenchymal Transition
/ drug effects
Female
Humans
MCF-7 Cells
Mice
Neoplasm Metastasis
Phenylurea Compounds
/ pharmacology
Transplantation, Heterologous
Triple Negative Breast Neoplasms
/ drug therapy
Wnt Signaling Pathway
/ genetics
beta Catenin
/ antagonists & inhibitors
Journal
Cell death & disease
ISSN: 2041-4889
Titre abrégé: Cell Death Dis
Pays: England
ID NLM: 101524092
Informations de publication
Date de publication:
21 02 2019
21 02 2019
Historique:
received:
04
12
2018
accepted:
04
02
2019
revised:
01
02
2019
entrez:
23
2
2019
pubmed:
23
2
2019
medline:
21
4
2020
Statut:
epublish
Résumé
Potassium ion channels are critical in the regulation of cell motility. The acquisition of cell motility is an essential parameter of cancer metastasis. However, the role of K
Identifiants
pubmed: 30792401
doi: 10.1038/s41419-019-1429-0
pii: 10.1038/s41419-019-1429-0
pmc: PMC6385342
doi:
Substances chimiques
Antineoplastic Agents
0
CTNNB1 protein, human
0
Cresols
0
ERG1 Potassium Channel
0
Phenylurea Compounds
0
beta Catenin
0
1,3-bis(2-hydroxy-5-trifluoromethylphenyl)urea
0I579CNG0I
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
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