Longitudinal changes of right ventricular deformation mechanics during trastuzumab therapy in breast cancer patients.


Journal

European journal of heart failure
ISSN: 1879-0844
Titre abrégé: Eur J Heart Fail
Pays: England
ID NLM: 100887595

Informations de publication

Date de publication:
04 2019
Historique:
received: 17 07 2018
accepted: 12 11 2018
pubmed: 28 2 2019
medline: 25 8 2020
entrez: 28 2 2019
Statut: ppublish

Résumé

Trastuzumab improves dramatically the prognosis of HER2-positive breast cancer patients, but it may lead to cardiotoxicity with left ventricular (LV) systolic dysfunction. Its effects on right ventricular (RV) function have not however been elucidated. We sought to assess LV and RV deformation mechanics during treatment with trastuzumab in breast cancer patients. We studied 101 consecutive women (mean age 54.3 ± 11.4 years) receiving trastuzumab for 12 months; 62 of them (61.4%) had previously received anthracyclines and 26 (25.7%) were receiving taxanes concurrently with trastuzumab. Comprehensive two-dimensional echocardiography with speckle tracking imaging of LV and RV global longitudinal strain (GLS) and RV free wall longitudinal strain (FWLS) analyses were performed at baseline and every 3 months up to treatment completion. Cardiotoxicity was defined as a decrease of baseline LV ejection fraction > 10 percentage units to a value < 50%. At 3 months, only LV GLS was significantly reduced (-19.5 ± 2.7 to -18.7 ± 2.8, P = 0.0410), while at 6 months, LV GLS, RV GLS and RV FWLS had significantly declined reaching their lowest values (-17.9 ± 6.1, P = 0.002, -19.6 ± 5.2, P = 0.003 and -19.7 ± 5.6, P = 0.004, respectively). Ten women (9.9%) developed cardiotoxicity. A RV GLS percent change of -14.8% predicted cardiotoxicity with 66.7% sensitivity and 70.8% specificity (area under the curve 0.68, 95% confidence interval 0.54-0.81), classifying correctly 90% of women with cardiotoxicity. This cut-off is quite similar to the 15% change of LV GLS previously suggested as predictive of cardiotoxicity. Deformation mechanics of both the left and right ventricle follow similar temporal pattern and degree of impairment during trastuzumab therapy, confirming the global and uniform effect of trastuzumab on myocardial function.

Sections du résumé

BACKGROUND
Trastuzumab improves dramatically the prognosis of HER2-positive breast cancer patients, but it may lead to cardiotoxicity with left ventricular (LV) systolic dysfunction. Its effects on right ventricular (RV) function have not however been elucidated. We sought to assess LV and RV deformation mechanics during treatment with trastuzumab in breast cancer patients.
METHODS AND RESULTS
We studied 101 consecutive women (mean age 54.3 ± 11.4 years) receiving trastuzumab for 12 months; 62 of them (61.4%) had previously received anthracyclines and 26 (25.7%) were receiving taxanes concurrently with trastuzumab. Comprehensive two-dimensional echocardiography with speckle tracking imaging of LV and RV global longitudinal strain (GLS) and RV free wall longitudinal strain (FWLS) analyses were performed at baseline and every 3 months up to treatment completion. Cardiotoxicity was defined as a decrease of baseline LV ejection fraction > 10 percentage units to a value < 50%. At 3 months, only LV GLS was significantly reduced (-19.5 ± 2.7 to -18.7 ± 2.8, P = 0.0410), while at 6 months, LV GLS, RV GLS and RV FWLS had significantly declined reaching their lowest values (-17.9 ± 6.1, P = 0.002, -19.6 ± 5.2, P = 0.003 and -19.7 ± 5.6, P = 0.004, respectively). Ten women (9.9%) developed cardiotoxicity. A RV GLS percent change of -14.8% predicted cardiotoxicity with 66.7% sensitivity and 70.8% specificity (area under the curve 0.68, 95% confidence interval 0.54-0.81), classifying correctly 90% of women with cardiotoxicity. This cut-off is quite similar to the 15% change of LV GLS previously suggested as predictive of cardiotoxicity.
CONCLUSIONS
Deformation mechanics of both the left and right ventricle follow similar temporal pattern and degree of impairment during trastuzumab therapy, confirming the global and uniform effect of trastuzumab on myocardial function.

Identifiants

pubmed: 30811091
doi: 10.1002/ejhf.1385
doi:

Substances chimiques

Anthracyclines 0
Antineoplastic Agents, Immunological 0
Taxoids 0
Trastuzumab P188ANX8CK

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

529-535

Informations de copyright

© 2019 The Authors. European Journal of Heart Failure © 2019 European Society of Cardiology.

Auteurs

Kalliopi Keramida (K)

Cardio-Oncology Clinic, Heart Failure Unit, Department of Cardiology, Attikon University Hospital, National and Kapodistrian University of Athens Medical School, Athens, Greece.

Dimitrios Farmakis (D)

Cardio-Oncology Clinic, Heart Failure Unit, Department of Cardiology, Attikon University Hospital, National and Kapodistrian University of Athens Medical School, Athens, Greece.
University of Cyprus Medical School, Nicosia, Cyprus.

Jose Bingcang (J)

Barts Health NHS Trust and University College London Hospitals, London, UK.

Samir Sulemane (S)

Hammersmith Hospital, Imperial College Healthcare NHS Trust, London, UK.

Stephanie Sutherland (S)

Mount Vernon Cancer Centre, West Hertfordshire Hospitals NHS Trust, London, UK.

Roma A Bingcang (RA)

Barts Health NHS Trust and University College London Hospitals, London, UK.

Kanchana Ramachandran (K)

Watford Hospital, West Hertfordshire Hospitals NHS Trust, London, UK.

Chara Tzavara (C)

Department of Hygiene, Epidemiology and Medical Statistics, Athens University Medical School, Athens, Greece.

Georgios Charalampopoulos (G)

2nd Radiology Department, University General Hospital 'ATTIKON', Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Dimitrios Filippiadis (D)

2nd Radiology Department, University General Hospital 'ATTIKON', Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Nikolaos Kouris (N)

Cardiology Department, General Hospital of Elefsina Thriassio, Athens, Greece.

Petros Nihoyannopoulos (P)

Hammersmith Hospital, Imperial College Healthcare NHS Trust, London, UK.
UK National Heart and Lung Institute, London, UK.

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