Mitochondrial superoxide disrupts the metabolic and epigenetic landscape of CD4


Journal

Redox biology
ISSN: 2213-2317
Titre abrégé: Redox Biol
Pays: Netherlands
ID NLM: 101605639

Informations de publication

Date de publication:
10 2019
Historique:
received: 04 01 2019
revised: 05 02 2019
accepted: 11 02 2019
pubmed: 2 3 2019
medline: 24 4 2020
entrez: 2 3 2019
Statut: ppublish

Résumé

While the role of mitochondrial metabolism in controlling T-lymphocyte activation and function is becoming more clear, the specifics of how mitochondrial redox signaling contributes to T-lymphocyte regulation remains elusive. Here, we examined the global effects of elevated mitochondrial superoxide (O

Identifiants

pubmed: 30819616
pii: S2213-2317(19)30022-9
doi: 10.1016/j.redox.2019.101141
pmc: PMC6859572
pii:
doi:

Substances chimiques

Cytokines 0
Superoxides 11062-77-4
Hydrogen Peroxide BBX060AN9V
Superoxide Dismutase EC 1.15.1.1

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

101141

Subventions

Organisme : NHLBI NIH HHS
ID : R00 HL123471
Pays : United States

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier B.V. All rights reserved.

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Auteurs

C M Moshfegh (CM)

Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, United States.

C W Collins (CW)

Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, United States.

V Gunda (V)

The Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE, United States.

A Vasanthakumar (A)

Section of Hematology/Oncology, Department of Medicine, The University of Chicago Medicine Comprehensive Cancer Center, Chicago, IL, United States.

J Z Cao (JZ)

Section of Hematology/Oncology, Department of Medicine, The University of Chicago Medicine Comprehensive Cancer Center, Chicago, IL, United States.

P K Singh (PK)

The Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE, United States.

L A Godley (LA)

Section of Hematology/Oncology, Department of Medicine, The University of Chicago Medicine Comprehensive Cancer Center, Chicago, IL, United States.

Adam J Case (AJ)

Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, United States. Electronic address: adam.case@unmc.edu.

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Classifications MeSH