Vegfa/vegfr2 signaling is necessary for zebrafish islet vessel development, but is dispensable for beta-cell and alpha-cell formation.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
05 03 2019
Historique:
received: 06 11 2018
accepted: 29 01 2019
entrez: 7 3 2019
pubmed: 7 3 2019
medline: 24 9 2020
Statut: epublish

Résumé

The mechanisms underlying zebrafish pancreatic islet vascularization have not been well characterized. We sought to determine the angiogenic factors responsible for islet vascularization and assess whether an absence of endothelial cells affects beta-cell and alpha-cell formation. We used a double transgenic zebrafish Tg(fli1:EGFP; insa:tagRFP) to label endothelial cells and beta-cells, respectively. Beta-cells developed adjacent to endothelial cells and by 72 hours post fertilization (hpf) the zebrafish pancreatic islet was highly vascularized. Zebrafish beta-cells express vascular endothelial growth factors (vegf), vegfaa and vegfab. Double knockdown of vegfaa and vegfab or the primary Vegfa receptors (Vegfr2), kdr and kdrl, resulted in vessel deficient islets. While beta-cell and alpha-cell numbers remained unchanged in vessel deficient islets, insulina expression was downregulated relative to controls. Vegfaa/Vegfab-Vegfr2 signaling is necessary for proper islet vessel development, but not for the initial formation of beta-cells and alpha-cells.

Identifiants

pubmed: 30837605
doi: 10.1038/s41598-019-40136-1
pii: 10.1038/s41598-019-40136-1
pmc: PMC6401103
doi:

Substances chimiques

Vascular Endothelial Growth Factor A 0
Vegfaa protein, zebrafish 0
Zebrafish Proteins 0
Vascular Endothelial Growth Factor Receptor-2 EC 2.7.10.1
kdr protein, zebrafish EC 2.7.10.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3594

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Auteurs

Chiara M Toselli (CM)

Department of Cellular and Physiological Sciences, Life Sciences Institute, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada.

Brayden M Wilkinson (BM)

Department of Cellular and Physiological Sciences, Life Sciences Institute, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada.

Joshua Paterson (J)

Department of Cellular and Physiological Sciences, Life Sciences Institute, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada.

Timothy J Kieffer (TJ)

Department of Cellular and Physiological Sciences, Life Sciences Institute, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada. tim.kieffer@ubc.ca.
Department of Surgery, Life Sciences Institute, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada. tim.kieffer@ubc.ca.

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Classifications MeSH