Shift Work Patterns, Chronotype, and Epithelial Ovarian Cancer Risk.


Journal

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology
ISSN: 1538-7755
Titre abrégé: Cancer Epidemiol Biomarkers Prev
Pays: United States
ID NLM: 9200608

Informations de publication

Date de publication:
05 2019
Historique:
received: 13 10 2018
revised: 03 12 2018
accepted: 26 02 2019
pubmed: 8 3 2019
medline: 17 7 2020
entrez: 8 3 2019
Statut: ppublish

Résumé

Shift work causing circadian disruption is classified as a "probable carcinogen" and may contribute to the pathogenesis of hormone-sensitive cancers. This study investigated shift work exposure in relation to epithelial ovarian cancer (EOC) risk. In a population-based case-control study with 496 EOC cases and 906 controls, lifetime occupational histories were collected and used to calculate cumulative years of shift work exposure, average number of night shifts per month, and average number of consecutive night shifts per month. ORs and 95% confidence intervals (CI) for associations with EOC risk were estimated using logistic regression. Associations were also examined according to chronotype and menopausal status. More than half of the cases (53.4%) and controls (51.7%) worked evening and/or night shifts. There was no clear pattern of increasing EOC risk with increasing years of shift work; the adjusted OR of EOC comparing the highest shift work category versus never working shift work was 1.20 (95% CI, 0.89-1.63). This association was more pronounced among those self-identified as having a "morning" chronotype (OR, 1.64; 95% CI, 1.01-2.65). Associations did not greatly differ by menopausal status. These results do not strongly demonstrate a relationship between shift work and EOC risk. This study collected detailed shift work information and examined shift work patterns according to shift times and schedules. The findings highlight that chronotype should be considered in studies of shift work as an exposure.

Sections du résumé

BACKGROUND
Shift work causing circadian disruption is classified as a "probable carcinogen" and may contribute to the pathogenesis of hormone-sensitive cancers. This study investigated shift work exposure in relation to epithelial ovarian cancer (EOC) risk.
METHODS
In a population-based case-control study with 496 EOC cases and 906 controls, lifetime occupational histories were collected and used to calculate cumulative years of shift work exposure, average number of night shifts per month, and average number of consecutive night shifts per month. ORs and 95% confidence intervals (CI) for associations with EOC risk were estimated using logistic regression. Associations were also examined according to chronotype and menopausal status.
RESULTS
More than half of the cases (53.4%) and controls (51.7%) worked evening and/or night shifts. There was no clear pattern of increasing EOC risk with increasing years of shift work; the adjusted OR of EOC comparing the highest shift work category versus never working shift work was 1.20 (95% CI, 0.89-1.63). This association was more pronounced among those self-identified as having a "morning" chronotype (OR, 1.64; 95% CI, 1.01-2.65). Associations did not greatly differ by menopausal status.
CONCLUSIONS
These results do not strongly demonstrate a relationship between shift work and EOC risk.
IMPACT
This study collected detailed shift work information and examined shift work patterns according to shift times and schedules. The findings highlight that chronotype should be considered in studies of shift work as an exposure.

Identifiants

pubmed: 30842128
pii: 1055-9965.EPI-18-1112
doi: 10.1158/1055-9965.EPI-18-1112
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

987-995

Subventions

Organisme : CIHR
Pays : Canada

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

©2019 American Association for Cancer Research.

Auteurs

Lisa Leung (L)

Université de Montréal Hospital Research Centre (CRCHUM), Montreal, Quebec, Canada.
Department of Public Health Sciences, Queen's University, Kingston, Ontario, Canada.

Anne Grundy (A)

Université de Montréal Hospital Research Centre (CRCHUM), Montreal, Quebec, Canada.
Department of Social and Preventative Medicine, Université de Montréal, Montreal, Quebec, Canada.

Jack Siemiatycki (J)

Université de Montréal Hospital Research Centre (CRCHUM), Montreal, Quebec, Canada.
Department of Social and Preventative Medicine, Université de Montréal, Montreal, Quebec, Canada.

Jocelyne Arseneau (J)

Gynecologic Oncology Unit, McGill University Health Centre, Montreal, Quebec, Canada.

Lucy Gilbert (L)

Gynecologic Oncology Unit, McGill University Health Centre, Montreal, Quebec, Canada.

Walter H Gotlieb (WH)

Gynecologic Oncology and Colposcopy, Sir Mortimer B. Davis-Jewish General Hospital, Montreal, Quebec, Canada.

Diane M Provencher (DM)

Université de Montréal Hospital Research Centre (CRCHUM), Montreal, Quebec, Canada.
Division of Gynecologic Oncology, Department of Obstetrics and Gynecology, Centre hospitalier de l'Université de Montréal, Montreal, Quebec, Canada.

Kristan J Aronson (KJ)

Department of Public Health Sciences, Queen's University, Kingston, Ontario, Canada.
Division of Cancer Care and Epidemiology, Queen's Cancer Research Institute, Kingston, Ontario, Canada.

Anita Koushik (A)

Université de Montréal Hospital Research Centre (CRCHUM), Montreal, Quebec, Canada. anita.koushik@umontreal.ca.
Department of Social and Preventative Medicine, Université de Montréal, Montreal, Quebec, Canada.

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