Bio-adrenomedullin as a marker of congestion in patients with new-onset and worsening heart failure.


Journal

European journal of heart failure
ISSN: 1879-0844
Titre abrégé: Eur J Heart Fail
Pays: England
ID NLM: 100887595

Informations de publication

Date de publication:
06 2019
Historique:
received: 29 10 2018
revised: 20 12 2018
accepted: 10 01 2019
pubmed: 8 3 2019
medline: 8 9 2020
entrez: 8 3 2019
Statut: ppublish

Résumé

Secretion of adrenomedullin (ADM) is stimulated by volume overload to maintain endothelial barrier function, and higher levels of biologically active (bio-) ADM in heart failure (HF) are a counteracting response to vascular leakage and tissue oedema. This study aimed to establish the value of plasma bio-ADM as a marker of congestion in patients with worsening HF. The association of plasma bio-ADM with clinical markers of congestion, as well as its prognostic value was studied in 2179 patients with new-onset or worsening HF enrolled in BIOSTAT-CHF. Data were validated in a separate cohort of 1703 patients. Patients with higher plasma bio-ADM levels were older, had more severe HF and more signs and symptoms of congestion (all P < 0.001). Amongst 20 biomarkers, bio-ADM was the strongest predictor of a clinical congestion score (r Plasma bio-ADM in patients with new-onset and worsening HF is associated with more severe HF and more oedema, orthopnoea, hepatomegaly and jugular venous pressure. We therefore postulate bio-ADM as a congestion marker, which might become useful to guide decongestive therapy.

Sections du résumé

BACKGROUND
Secretion of adrenomedullin (ADM) is stimulated by volume overload to maintain endothelial barrier function, and higher levels of biologically active (bio-) ADM in heart failure (HF) are a counteracting response to vascular leakage and tissue oedema. This study aimed to establish the value of plasma bio-ADM as a marker of congestion in patients with worsening HF.
METHODS AND RESULTS
The association of plasma bio-ADM with clinical markers of congestion, as well as its prognostic value was studied in 2179 patients with new-onset or worsening HF enrolled in BIOSTAT-CHF. Data were validated in a separate cohort of 1703 patients. Patients with higher plasma bio-ADM levels were older, had more severe HF and more signs and symptoms of congestion (all P < 0.001). Amongst 20 biomarkers, bio-ADM was the strongest predictor of a clinical congestion score (r
CONCLUSIONS
Plasma bio-ADM in patients with new-onset and worsening HF is associated with more severe HF and more oedema, orthopnoea, hepatomegaly and jugular venous pressure. We therefore postulate bio-ADM as a congestion marker, which might become useful to guide decongestive therapy.

Identifiants

pubmed: 30843353
doi: 10.1002/ejhf.1437
doi:

Substances chimiques

Biomarkers 0
Adrenomedullin 148498-78-6

Types de publication

Journal Article Multicenter Study Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

732-743

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2019 The Authors. European Journal of Heart Failure © 2019 European Society of Cardiology.

Auteurs

Biniyam G Demissei (BG)

Department of Cardiology, University of Groningen, University Medical Centre Groningen, Groningen, The Netherlands.

Joachim Struck (J)

Sphingotec GmbH, Hennigsdorf, Germany.

Andreas Bergmann (A)

Sphingotec GmbH, Hennigsdorf, Germany.

Stefan D Anker (SD)

Department of Cardiology (CVK), and Berlin-Brandenburg Center for Regenerative Therapies (BCRT), German Centre for Cardiovascular Research (DZHK) partner site Berlin, Charité Universitätsmedizin, Berlin, Germany.
Department of Cardiology and Pneumology, University Medical Center Göttingen (UMG), Göttingen, Germany.

Leong L Ng (LL)

Department of Cardiovascular Sciences, University of Leicester, Leicester, UK.
NIHR Leicester Biomedical Research Centre, Glenfield Hospital, Leicester, UK.

Kenneth Dickstein (K)

University of Bergen, Bergen, Norway.
Stavanger University Hospital, Stavanger, Norway.

Marco Metra (M)

Department of Medical and Surgical Specialties, Institute of Cardiology, Radiological Sciences and Public Health, University of Brescia, Brescia, Italy.

Nilesh J Samani (NJ)

Department of Cardiovascular Sciences, University of Leicester, Leicester, UK.
NIHR Leicester Biomedical Research Centre, Glenfield Hospital, Leicester, UK.

Simon P R Romaine (SPR)

Department of Cardiovascular Sciences, University of Leicester, Leicester, UK.
NIHR Leicester Biomedical Research Centre, Glenfield Hospital, Leicester, UK.

John Cleland (J)

National Heart & Lung Institute, Royal Brompton & Harefield Hospitals, Imperial College, London, UK.

Nicolas Girerd (N)

Inserm CIC1433, Université de Lorrain, CHU de Nancy, Nancy, France.

Chim C Lang (CC)

Division of Molecular and Clinical Medicine, School of Medicine, University of Dundee, Ninewells Hospital & Medical School, Dundee, UK.

Dirk J van Veldhuisen (DJ)

Department of Cardiology, University of Groningen, University Medical Centre Groningen, Groningen, The Netherlands.

Adriaan A Voors (AA)

Department of Cardiology, University of Groningen, University Medical Centre Groningen, Groningen, The Netherlands.

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