Efficacy of primary treatment with immunoglobulin plus ciclosporin for prevention of coronary artery abnormalities in patients with Kawasaki disease predicted to be at increased risk of non-response to intravenous immunoglobulin (KAICA): a randomised controlled, open-label, blinded-endpoints, phase 3 trial.


Journal

Lancet (London, England)
ISSN: 1474-547X
Titre abrégé: Lancet
Pays: England
ID NLM: 2985213R

Informations de publication

Date de publication:
16 Mar 2019
Historique:
received: 03 07 2018
revised: 07 08 2018
accepted: 21 08 2018
pubmed: 12 3 2019
medline: 9 5 2019
entrez: 12 3 2019
Statut: ppublish

Résumé

Genetic studies have indicated possible involvement of the upregulated calcium-nuclear factor of activated T cells pathway in the pathogenesis of Kawasaki disease. We aimed to assess safety and efficacy of ciclosporin, an immunosuppressant targeting this pathway, for protection of patients with Kawasaki disease against coronary artery abnormalities. We did a randomised, open-label, blinded endpoints trial involving 22 hospitals in Japan between May 29, 2014, and Dec 27, 2016. Eligible patients predicted to be at higher risk for intravenous immunoglobulin (IVIG) resistance were randomly assigned to IVIG plus ciclosporin (5 mg/kg per day for 5 days; study treatment) or IVIG (conventional treatment) groups, stratified by risk score, age, and sex. The primary endpoint was incidence of coronary artery abnormalities using Japanese criteria during the 12-week trial, assessed in participants who received at least one dose of study drug and who visited the study institution at least once during treatment. This trial is registered to Center for Clinical Trials, Japan Medical Association, number JMA-IIA00174. We enrolled 175 participants. One patient withdrew consent after enrolment and was excluded and one patient (in the study treatment group) was excluded from analysis because of lost echocardiography data. Incidence of coronary artery abnormalities was lower in the study treatment group than in the conventional treatment group (12 [14%] of 86 patients vs 27 [31%] of 87 patients; risk ratio 0·46; 95% CI 0·25-0·86; p=0·010). No difference was found in the incidence of adverse events between the groups (9% vs 7%; p=0·78). Combined primary therapy with IVIG and ciclosporin was safe and effective for favourable coronary artery outcomes in Kawasaki disease patients who were predicted to be unresponsive to IVIG. Japan Agency for Medical Research and Development (grant CCT-B-2503).

Sections du résumé

BACKGROUND BACKGROUND
Genetic studies have indicated possible involvement of the upregulated calcium-nuclear factor of activated T cells pathway in the pathogenesis of Kawasaki disease. We aimed to assess safety and efficacy of ciclosporin, an immunosuppressant targeting this pathway, for protection of patients with Kawasaki disease against coronary artery abnormalities.
METHODS METHODS
We did a randomised, open-label, blinded endpoints trial involving 22 hospitals in Japan between May 29, 2014, and Dec 27, 2016. Eligible patients predicted to be at higher risk for intravenous immunoglobulin (IVIG) resistance were randomly assigned to IVIG plus ciclosporin (5 mg/kg per day for 5 days; study treatment) or IVIG (conventional treatment) groups, stratified by risk score, age, and sex. The primary endpoint was incidence of coronary artery abnormalities using Japanese criteria during the 12-week trial, assessed in participants who received at least one dose of study drug and who visited the study institution at least once during treatment. This trial is registered to Center for Clinical Trials, Japan Medical Association, number JMA-IIA00174.
FINDINGS RESULTS
We enrolled 175 participants. One patient withdrew consent after enrolment and was excluded and one patient (in the study treatment group) was excluded from analysis because of lost echocardiography data. Incidence of coronary artery abnormalities was lower in the study treatment group than in the conventional treatment group (12 [14%] of 86 patients vs 27 [31%] of 87 patients; risk ratio 0·46; 95% CI 0·25-0·86; p=0·010). No difference was found in the incidence of adverse events between the groups (9% vs 7%; p=0·78).
INTERPRETATION CONCLUSIONS
Combined primary therapy with IVIG and ciclosporin was safe and effective for favourable coronary artery outcomes in Kawasaki disease patients who were predicted to be unresponsive to IVIG.
FUNDING BACKGROUND
Japan Agency for Medical Research and Development (grant CCT-B-2503).

Identifiants

pubmed: 30853151
pii: S0140-6736(18)32003-8
doi: 10.1016/S0140-6736(18)32003-8
pii:
doi:

Substances chimiques

Immunoglobulins, Intravenous 0
Immunosuppressive Agents 0
Cyclosporine 83HN0GTJ6D

Types de publication

Clinical Trial, Phase III Comparative Study Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

1128-1137

Investigateurs

Hiromichi Hamada (H)
Takafumi Honda (T)
Kumi Yasukawa (K)
Katsuki Hirai (K)
Nishihara Takahiro (N)
Takashi Soga (T)
Hideoki Yajima (H)
Hideyasu Ooto (H)
You Umeda (Y)
Hiroyuki Suzuki (H)
Takashi Takeuchi (T)
Tomohiro Suenaga (T)
Nobuyuki Kakimoto (N)
Yukiko Ishiguchi (Y)
Masahiro Kamada (M)
Yoshiaki Okuma (Y)
Nobuyuki Takada (N)
Shinichi Suwabe (S)
Masaaki Yanai (M)
Yasushi Ueno (Y)
Natsuko Nishi (N)
Junichi Sato (J)
Yuuko Saito (Y)
Yutaka Kitani (Y)
Mami Nakayashiro (M)
Taisuke Nabeshima (T)
Kiyotaka Takefuta (K)
Mamoru Ayusawa (M)
Takahiro Nakamura (T)
Akiko Komori (A)
Masataka Kato (M)
Ryota Ebata (R)
Naoki Saito (N)
Kentaro Okunushi (K)
Hironobu Kobayashi (H)
Eiichi Yamamoto (E)
Takeshi Nakano (T)
Yuichi Nomura (Y)
Kiminori Masuda (K)
Yuya Hashimura (Y)
Hirotaka Minami (H)
Takamichi Uchiyama (T)
You Okizuka (Y)
Kazunobu Ouchi (K)
Naoki Ohno (N)
Satoko Ogita (S)
Hiroshi Masuda (H)
Hiroshi Ono (H)
Akira Ishiguro (A)
Shinichi Takatsuki (S)
Tsutomu Saji (T)
Fukiko Ichida (F)
Keiichi Hirono (K)
Sayaka Ozawa (S)
Tadashi Ariga (T)
Atsuhito Takeda (A)
Gaku Izumi (G)
Takashi Higaki (T)
Akio Otsuki (A)
Akira Hata (A)
Hideki Hanaoka (H)
Hiroyuki Suzuki (H)
Masaru Terai (M)
Hiromichi Hamada (H)
Yoshihiro Onouchi (Y)
Ryota Ebata (R)
Moe Terauchi (M)
Yasuhisa Fujii (Y)
Takatoshi Sato (T)
Reiko Aoyagi (R)
Michiko Hanawa (M)
Tadami Fujiwara (T)
Shigeto Fuse (S)
Yoshitomo Okajima (Y)
Shunji Kurotobi (S)

Commentaires et corrections

Type : CommentIn
Type : ErratumIn
Type : ErratumIn
Type : CommentIn

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Hiromichi Hamada (H)

Graduate School of Medicine, Chiba University, Chiba, Japan; Department of Pediatrics, Yachiyo Medical Center, Tokyo Women's Medical University, Chiba, Japan.

Hiroyuki Suzuki (H)

Department of Pediatrics, Wakayama Medical University of Medicine, Wakayama, Japan.

Yoshihiro Onouchi (Y)

Department of Public Health, Chiba University, Chiba, Japan.

Ryota Ebata (R)

Department of Pediatrics, Chiba University, Chiba, Japan.

Masaru Terai (M)

Department of Pediatrics, Yachiyo Medical Center, Tokyo Women's Medical University, Chiba, Japan.

Shigeto Fuse (S)

Department of Pediatrics, NTT Sapporo Medical Center, Sapporo, Japan.

Yoshitomo Okajima (Y)

Department of Pediatrics, Chiba Cardiovascular Center, Chiba, Japan.

Shunji Kurotobi (S)

Oriono Clinic, Sakai, Japan.

Katsuki Hirai (K)

Department of Pediatrics, Japanese Red Cross Kumamoto Hospital, Kumamoto, Japan.

Takashi Soga (T)

Department of Pediatrics, Showa University Northern Yokohama Hospital, Yokohama, Japan.

Yukiko Ishiguchi (Y)

Department of Pediatric Cardiology, Hiroshima City Hiroshima Citizens Hospital, Hiroshima, Japan.

Yoshiaki Okuma (Y)

Department of Pediatrics, National Center for Global Health and Medicine, Tokyo, Japan.

Nobuyuki Takada (N)

Department of Pediatrics, Kimitsu Chuo Hospital, Chiba, Japan.

Masaaki Yanai (M)

Department of Pediatrics, Kumamoto Regional Medical Center, Kumamoto, Japan.

Junichi Sato (J)

Department of Pediatrics, Funabashi Municipal Medical Center, Funabashi, Japan.

Mami Nakayashiro (M)

Department of Pediatric Cardiology, Okinawa Prefectural Nanbu Medical Center and Children's Medical Center, Okinawa, Japan.

Mamoru Ayusawa (M)

Department of Pediatrics, Nihon University Itabashi Hospital, Tokyo, Japan.

Eiichi Yamamoto (E)

Department of Pediatrics, Ehime Prefectural Central Hospital, Ehime, Japan.

Yuichi Nomura (Y)

Department of Pediatrics, Kagoshima City Hospital, Kagoshima, Japan.

Yuya Hashimura (Y)

Department of Pediatrics, Takatsuki General Hospital, Osaka, Japan.

Kazunobu Ouchi (K)

Department of Pediatrics, Kawasaki Medical School Hospital, Okayama, Japan.

Hiroshi Masuda (H)

Department of General Pediatrics & Interdisciplinary Medicine, National Center for Child Health and Development, Tokyo, Japan.

Shinichi Takatsuki (S)

Department of Pediatrics, Toho University, Tokyo, Japan.

Keiichi Hirono (K)

Department of Pediatrics, Graduate School of Medicine, University of Toyama, Toyama, Japan.

Tadashi Ariga (T)

Department of Pediatrics, Hokkaido University Hospital, Sapporo, Japan.

Takashi Higaki (T)

Department of Regional Pediatrics and Perinatology, Graduate School of Medicine, Ehime University, Ehime, Japan.

Akio Otsuki (A)

Department of Pediatrics, Komatsu Municipal Hospital, Ishikawa, Japan.

Moe Terauchi (M)

Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Reiko Aoyagi (R)

Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Takatoshi Sato (T)

Department of Pediatrics, Funabashi Municipal Medical Center, Funabashi, Japan; Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Yasuhisa Fujii (Y)

Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Tadami Fujiwara (T)

Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Hideki Hanaoka (H)

Clinical Research Centre, Chiba University Hospital, Chiba, Japan.

Akira Hata (A)

Department of Public Health, Chiba University, Chiba, Japan. Electronic address: ahata@faculty.chiba-u.jp.

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