Brain region-specific regulation of histone acetylation and efflux transporters in mice.


Journal

Journal of biochemical and molecular toxicology
ISSN: 1099-0461
Titre abrégé: J Biochem Mol Toxicol
Pays: United States
ID NLM: 9717231

Informations de publication

Date de publication:
Jun 2019
Historique:
received: 21 12 2018
revised: 12 02 2019
accepted: 19 02 2019
pubmed: 22 3 2019
medline: 6 8 2020
entrez: 22 3 2019
Statut: ppublish

Résumé

Multidrug resistance protein 1 (MDR1) and breast cancer resistance protein (BCRP) protect the brain by restricting the passage of chemicals across the blood-brain barrier. Prior studies have demonstrated the epigenetic regulation of MDR1 and BCRP in cancer cells treated with histone deacetylase (HDAC) inhibitors that enhance histone acetylation and gene transcription. In the present study, we tested the in vivo effects of two HDAC inhibitors, valproic acid (VPA; 400 mg/kg) and apicidin (5 mg/kg), on Mdr1 and Bcrp transporter expression in brain regions of adult male mice injected intraperitoneally daily for 7 days. VPA increased Mdr1 protein expression in the striatum (70%) and Bcrp protein in the midbrain (30%). Apicidin enhanced striatal Mdr1 protein (30%) and hippocampal Bcrp protein (20%). Transporter induction correlated with increased histone H3 acetylation in discrete brain regions. In conclusion, HDAC inhibitors upregulate transporter proteins in vivo, which may be important in regulating regional xenobiotic disposition within the brain.

Identifiants

pubmed: 30897286
doi: 10.1002/jbt.22318
pmc: PMC6754812
mid: NIHMS1013746
doi:

Substances chimiques

ATP Binding Cassette Transporter, Subfamily B, Member 1 0
ATP Binding Cassette Transporter, Subfamily G, Member 2 0
Abcg2 protein, mouse 0
Histone Deacetylase Inhibitors 0
Histones 0
Peptides, Cyclic 0
apicidin 0
Valproic Acid 614OI1Z5WI
Histone Deacetylases EC 3.5.1.98

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e22318

Subventions

Organisme : National Institutes of Health
Organisme : NIEHS NIH HHS
ID : P30 ES005022
Pays : United States
Organisme : NIEHS NIH HHS
ID : R01 ES021800
Pays : United States
Organisme : National Institute of Environmental Health Sciences
ID : P30ES005022
Organisme : National Institute of Environmental Health Sciences
ID : R01ES021800

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Références

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Auteurs

Dahea You (D)

School of Graduate Studies, Joint Graduate Program in Toxicology, Rutgers, The State University of New Jersey, Piscataway, New Jersey.

Hye Min Shin (HM)

Department of Pharmacology and Toxicology, Rutgers, The State University of New Jersey, Ernest Mario School of Pharmacy, Piscataway, New Jersey.

Fatimah Mosaad (F)

Department of Pharmacology and Toxicology, Rutgers, The State University of New Jersey, Ernest Mario School of Pharmacy, Piscataway, New Jersey.

Jason R Richardson (JR)

Division of Toxicology, Environmental and Occupational Health Sciences Institute, Piscataway, New Jersey.
Department of Environmental Health Sciences, Robert Stempel School of Public Health and Social Work, Florida International University, Miami, Florida.

Lauren M Aleksunes (LM)

Department of Pharmacology and Toxicology, Rutgers, The State University of New Jersey, Ernest Mario School of Pharmacy, Piscataway, New Jersey.
Division of Toxicology, Environmental and Occupational Health Sciences Institute, Piscataway, New Jersey.

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Classifications MeSH