Generation of Nppa-tagBFP reporter knock-in mouse line for studying cardiac chamber specification.


Journal

Genesis (New York, N.Y. : 2000)
ISSN: 1526-968X
Titre abrégé: Genesis
Pays: United States
ID NLM: 100931242

Informations de publication

Date de publication:
06 2019
Historique:
received: 20 01 2019
revised: 14 03 2019
accepted: 15 03 2019
pubmed: 29 3 2019
medline: 7 1 2020
entrez: 29 3 2019
Statut: ppublish

Résumé

Nppa is a cardiac hormone which plays critical roles in regulating salt-water balance. Its expression is restricted to the atria of the healthy post-natal heart. During heart development, spatio-temporal expression of Nppa is dynamically changed within the heart and becomes restricted to the atria upon birth. In contrast to its atrial specific expression after birth, Nppa is re-expressed in the adult ventricles in response to cardiac hypertrophy. To study cardiac chamber specification during development and pathological cardiac remodeling during heart disease, we generated a novel Nppa reporter mouse line by knocking-in a tagBFP reporter cassette into 3'-UTR of the Nppa gene without disrupting the endogenous gene. Our results demonstrated dynamic tagBFP expression in the developing heart, recapitulating the spatiotemporal expression pattern of endogenous Nppa. We also found that Nppa-tagBFP is induced in the ventricle during pathological remodeling. Taken together, Nppa-tagBFP reporter knock-in mouse model described in this article will serve as a valuable tool to study cardiac chamber specification during development as well as pathological cardiac remodeling.

Identifiants

pubmed: 30920727
doi: 10.1002/dvg.23294
pmc: PMC6826257
mid: NIHMS1056965
doi:

Substances chimiques

Nppa protein, mouse 0
Atrial Natriuretic Factor 85637-73-6

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e23294

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL119234
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL133290
Pays : United States
Organisme : NHLBI NIH HHS
ID : R03 HL140264
Pays : United States
Organisme : NIH HHS
ID : S10 OD021630
Pays : United States

Informations de copyright

© 2019 Wiley Periodicals, Inc.

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Auteurs

Zhentao Zhang (Z)

Department of Medicine, Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Department of Cell and Developmental Biology, Vanderbilt University, Nashville, Tennessee.
Vanderbilt Center for Stem Cell Biology, Vanderbilt University, Nashville, Tennessee.

Qinkun Zhang (Q)

Department of Medicine, Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Department of Pharmacology, Vanderbilt University, Nashville, Tennessee.

Hind Lal (H)

Department of Medicine, Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Department of Pharmacology, Vanderbilt University, Nashville, Tennessee.

Young-Jae Nam (YJ)

Department of Medicine, Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Department of Cell and Developmental Biology, Vanderbilt University, Nashville, Tennessee.
Vanderbilt Center for Stem Cell Biology, Vanderbilt University, Nashville, Tennessee.

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