Network basis of the dysexecutive and posterior cortical cognitive profiles in Parkinson's disease.


Journal

Movement disorders : official journal of the Movement Disorder Society
ISSN: 1531-8257
Titre abrégé: Mov Disord
Pays: United States
ID NLM: 8610688

Informations de publication

Date de publication:
06 2019
Historique:
received: 26 12 2018
revised: 04 03 2019
accepted: 06 03 2019
pubmed: 30 3 2019
medline: 14 3 2020
entrez: 30 3 2019
Statut: ppublish

Résumé

The dual syndrome hypothesis of cognitive impairment in PD suggests that two cognitive profiles exist with distinct pathological mechanisms and a differential risk for further cognitive decline. How these profiles relate to network dysfunction has never been explicitly characterized. First, to assess intranetwork functional connectivity while considering global connectivity, and second, to relate network connectivity with measures of the dysexecutive and posterior cortical profiles. Eighty-two subjects with idiopathic PD and 37 age-matched controls underwent resting-state functional MRI and comprehensive neuropsychological assessment. Intranetwork and global connectivity was compared between groups. Measures of the dysexecutive and posterior cortical profiles were related to network connectivity while considering demographic and disease-related covariates. PD subjects show decreased connectivity within several cortical networks. However, only the sensorimotor network displayed a loss of connectivity independent of the observed decreased global connectivity. The dysexecutive factor was independently related to increased motor severity, less education, and decreased connectivity in the sensorimotor network. The posterior cortical factor was related to increased age, less education, decreased connectivity in the central executive network, as well as increased connectivity in the temporal network. Our results provide evidence supporting a network-specific process of degeneration in the sensorimotor network which contributes to the dysexecutive cognitive profile. In contrast, connectivity of the temporal and central executive network is related to the posterior cortical profile, representing a distinct network signature of this syndrome. © 2019 International Parkinson and Movement Disorder Society.

Sections du résumé

BACKGROUND
The dual syndrome hypothesis of cognitive impairment in PD suggests that two cognitive profiles exist with distinct pathological mechanisms and a differential risk for further cognitive decline. How these profiles relate to network dysfunction has never been explicitly characterized.
OBJECTIVE
First, to assess intranetwork functional connectivity while considering global connectivity, and second, to relate network connectivity with measures of the dysexecutive and posterior cortical profiles.
METHODS
Eighty-two subjects with idiopathic PD and 37 age-matched controls underwent resting-state functional MRI and comprehensive neuropsychological assessment. Intranetwork and global connectivity was compared between groups. Measures of the dysexecutive and posterior cortical profiles were related to network connectivity while considering demographic and disease-related covariates.
RESULTS
PD subjects show decreased connectivity within several cortical networks. However, only the sensorimotor network displayed a loss of connectivity independent of the observed decreased global connectivity. The dysexecutive factor was independently related to increased motor severity, less education, and decreased connectivity in the sensorimotor network. The posterior cortical factor was related to increased age, less education, decreased connectivity in the central executive network, as well as increased connectivity in the temporal network.
CONCLUSIONS
Our results provide evidence supporting a network-specific process of degeneration in the sensorimotor network which contributes to the dysexecutive cognitive profile. In contrast, connectivity of the temporal and central executive network is related to the posterior cortical profile, representing a distinct network signature of this syndrome. © 2019 International Parkinson and Movement Disorder Society.

Identifiants

pubmed: 30924964
doi: 10.1002/mds.27674
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

893-902

Subventions

Organisme : CIHR
ID : MOP‐126017
Pays : Canada

Informations de copyright

© 2019 International Parkinson and Movement Disorder Society.

Auteurs

Stefan Lang (S)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.

Alexandru Hanganu (A)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.
Centre de Recherche, Institut Universitaire de Gériatrie de Montréal, Montreal, QC, Canada.

Liu Shi Gan (LS)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.

Mekale Kibreab (M)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.

Noémie Auclair-Ouellet (N)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
McGill University School of Communication Sciences and Disorders, Montreal, Canada.

Tazrina Alrazi (T)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.

Mehrafarin Ramezani (M)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.

Jenelle Cheetham (J)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.

Tracy Hammer (T)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.

Iris Kathol (I)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.

Justyna Sarna (J)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.

Oury Monchi (O)

Cumming School of Medicine, Hotchkiss Brain Institute, Calgary, AB, Canada.
Department of Clinical Neurosciences and Department of Radiology, University of Calgary, Calgary, AB, Canada.
Centre de Recherche, Institut Universitaire de Gériatrie de Montréal, Montreal, QC, Canada.
Department of Neurology, Montreal General Hospital, Montreal, QC, Canada.
Department of Radiology, Radio-Oncology, and Nuclear Medicine, Université de Montréal, Montreal, QC, Canada.

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