Increased Serum Angiopoietin-Like 6 Ahead of Metabolic Syndrome in a Prospective Cohort Study.


Journal

Diabetes & metabolism journal
ISSN: 2233-6079
Titre abrégé: Diabetes Metab J
Pays: Korea (South)
ID NLM: 101556588

Informations de publication

Date de publication:
08 2019
Historique:
received: 16 05 2018
accepted: 21 12 2018
pubmed: 11 4 2019
medline: 11 4 2019
entrez: 11 4 2019
Statut: ppublish

Résumé

Despite being an anti-obesity hepatokine, the levels of serum angiopoietin-like 6 (ANGPTL6) are elevated in various metabolic diseases. Thus, ANGPTL6 expression may reflect metabolic burden and may have compensatory roles. This study investigated the association between serum ANGPTL6 levels and new-onset metabolic syndrome. In total, 221 participants without metabolic syndrome were randomly selected from a rural cohort in Korea. Baseline serum ANGPTL6 levels were measured using an enzyme-linked immunosorbent assay. Anthropometric and biochemical markers were analyzed before and after follow-up examinations. During an average follow-up period of 2.75 (interquartile range, 0.76) years, 82 participants (37.1%) presented new-onset metabolic syndrome and had higher ANGPTL6 levels before onset than those without metabolic syndrome (48.03±18.84 ng/mL vs. 64.75±43.35 ng/mL, Increased serum ANGPTL6 levels precede the development of metabolic syndrome and its components, including low high density lipoprotein, high triglyceride, and high glucose levels, which have an independent predictive value for metabolic syndrome.

Sections du résumé

BACKGROUND
Despite being an anti-obesity hepatokine, the levels of serum angiopoietin-like 6 (ANGPTL6) are elevated in various metabolic diseases. Thus, ANGPTL6 expression may reflect metabolic burden and may have compensatory roles. This study investigated the association between serum ANGPTL6 levels and new-onset metabolic syndrome.
METHODS
In total, 221 participants without metabolic syndrome were randomly selected from a rural cohort in Korea. Baseline serum ANGPTL6 levels were measured using an enzyme-linked immunosorbent assay. Anthropometric and biochemical markers were analyzed before and after follow-up examinations.
RESULTS
During an average follow-up period of 2.75 (interquartile range, 0.76) years, 82 participants (37.1%) presented new-onset metabolic syndrome and had higher ANGPTL6 levels before onset than those without metabolic syndrome (48.03±18.84 ng/mL vs. 64.75±43.35 ng/mL,
CONCLUSION
Increased serum ANGPTL6 levels precede the development of metabolic syndrome and its components, including low high density lipoprotein, high triglyceride, and high glucose levels, which have an independent predictive value for metabolic syndrome.

Identifiants

pubmed: 30968619
pii: 43.e25
doi: 10.4093/dmj.2018.0080
pmc: PMC6712233
doi:

Substances chimiques

ANGPTL6 protein, human 0
Angiopoietin-Like Protein 6 0
Angiopoietin-like Proteins 0
Biomarkers 0
Blood Glucose 0
Lipoproteins, HDL 0
Triglycerides 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Pagination

521-529

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 Korean Diabetes Association.

Déclaration de conflit d'intérêts

No potential conflict of interest relevant to this article was reported.

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Auteurs

Jun Namkung (J)

Department of Biochemistry, Yonsei University Wonju College of Medicine, Wonju, Korea.
Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea. junitive@yonsei.ac.kr.

Joon Hyung Sohn (JH)

Institute of Lifestyle Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.

Jae Seung Chang (JS)

Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea.
Institute of Lifestyle Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Physiology, Yonsei University Wonju College of Medicine, Wonju, Korea.

Sang Wook Park (SW)

Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Internal Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.
Institute of Genomic Cohort, Yonsei University Wonju College of Medicine, Wonju, Korea.

Jang Young Kim (JY)

Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Internal Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.
Institute of Genomic Cohort, Yonsei University Wonju College of Medicine, Wonju, Korea.

Sang Baek Koh (SB)

Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea.
Institute of Genomic Cohort, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Preventive Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.

In Deok Kong (ID)

Institute of Lifestyle Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Physiology, Yonsei University Wonju College of Medicine, Wonju, Korea.

Kyu Sang Park (KS)

Mitohormesis Research Center, Yonsei University Wonju College of Medicine, Wonju, Korea.
Institute of Lifestyle Medicine, Yonsei University Wonju College of Medicine, Wonju, Korea.
Department of Physiology, Yonsei University Wonju College of Medicine, Wonju, Korea. qsang@yonsei.ac.kr.

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