Functional capillary impairment in patients with ventricular assist devices.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
11 04 2019
Historique:
received: 22 08 2018
accepted: 29 03 2019
entrez: 13 4 2019
pubmed: 13 4 2019
medline: 2 10 2020
Statut: epublish

Résumé

The implantation of continuous - flow ventricular assist devices (VAD) is suggested to evoke angiodysplasia contributing to adverse events such as gastrointestinal bleeding. We evaluated in vivo capillary density and glycocalyx dimensions to investigate possible systemic microvascular changes in patients with chronic heart failure and VAD support vs. standard medical treatment. Forty-two patients with VAD support were compared to forty-one patients with ischemic and non-ischemic chronic heart failure (CHF) on standard pharmacotherapy and to a group of forty-two healthy subjects in a prospective cross-sectional study. Sublingual microcirculation was visualized using Sidestream Darkfield videomicroscopy and functional and perfused total capillary densities were quantified. Patients with VAD implantation were followed for one year and bleeding events were recorded. Median time after VAD implantation was 18 months. Patients were treated with centrifugal-flow devices (n = 31) or axial-flow devices (n = 11). Median functional capillary density was significantly lower in patients with VAD therapy as compared to CHF patients (196 vs. 255/mm

Identifiants

pubmed: 30976042
doi: 10.1038/s41598-019-42334-3
pii: 10.1038/s41598-019-42334-3
pmc: PMC6459831
doi:

Types de publication

Clinical Trial Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

5909

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Auteurs

Patricia P Wadowski (PP)

Division of Cardiothoracic and Vascular Anesthesia, Medical University of Vienna, Vienna, Austria.

Barbara Steinlechner (B)

Division of Cardiothoracic and Vascular Anesthesia, Medical University of Vienna, Vienna, Austria.

Daniel Zimpfer (D)

Department of Surgery, Division of Cardiac Surgery, Medical University of Vienna, Vienna, Austria.

Thomas Schlöglhofer (T)

Department of Surgery, Division of Cardiac Surgery, Medical University of Vienna, Vienna, Austria.
Department of Medical Physics and Biomedical Engineering, Medical University of Vienna, Vienna, Austria.
Ludwig-Boltzmann-Cluster for Cardiovascular Research, Vienna, Austria.

Heinrich Schima (H)

Department of Medical Physics and Biomedical Engineering, Medical University of Vienna, Vienna, Austria.
Ludwig-Boltzmann-Cluster for Cardiovascular Research, Vienna, Austria.

Martin Hülsmann (M)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Irene M Lang (IM)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Thomas Gremmel (T)

Department of Internal Medicine II, Division of Angiology, Medical University of Vienna, Vienna, Austria.

Renate Koppensteiner (R)

Department of Internal Medicine II, Division of Angiology, Medical University of Vienna, Vienna, Austria.

Sonja Zehetmayer (S)

Intitute of Medical Statistics, Medical University of Vienna, Vienna, Austria.

Constantin Weikert (C)

Department of Internal Medicine II, Division of Angiology, Medical University of Vienna, Vienna, Austria.

Joseph Pultar (J)

Department of Internal Medicine II, Division of Angiology, Medical University of Vienna, Vienna, Austria.

Bernd Jilma (B)

Department of Clinical Pharmacology, Medical University of Vienna, Vienna, Austria. bernd.jilma@meduniwien.ac.at.

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