KRAS mutational status impacts pathologic response to pre-hepatectomy chemotherapy: a study from the International Genetic Consortium for Liver Metastases.


Journal

HPB : the official journal of the International Hepato Pancreato Biliary Association
ISSN: 1477-2574
Titre abrégé: HPB (Oxford)
Pays: England
ID NLM: 100900921

Informations de publication

Date de publication:
11 2019
Historique:
received: 29 08 2018
revised: 08 01 2019
accepted: 14 03 2019
pubmed: 14 4 2019
medline: 16 7 2020
entrez: 14 4 2019
Statut: ppublish

Résumé

A major response to pre-hepatectomy chemotherapy has been associated with improved survival in patients who undergo resection of colorectal liver metastases (CRLM). However, the role of tumor biology, as exemplified by overall and codon-specific KRAS mutational status, in predicting response to chemotherapy is not well defined. Pathologic response was characterized as minor or major depending on the percentage of remnant viable cells (>50% vs <50%, respectively). Multivariable logistic regression was used to identify factors associated with major response. 319 patients met inclusion criteria. 229 patients had a KRAS wild-type (wtKRAS) tumor and 90 harbored KRAS mutations (mutKRAS). A major pathologic response was more commonly noted in patients with wtKRAS compared to mutKRAS (48.5% vs 33.3%, P = 0.01) and wtKRAS status remained independently associated with a major response (P = 0.04). On a codon-specific level, major pathologic response occurred less frequently in those with codon 13 mutations (17.7%) compared to those with codon 12 (35.4%), and other KRAS mutations (33.3%). Importantly, codon 13 mutations were independently associated with minor pathologic response (P = 0.023). Patients with wtKRAS tumors appear to have the highest likelihood of experiencing a major response after preoperative chemotherapy. Future studies in "all-comer" cohorts are needed to confirm these findings and further investigate the response of codon 13 mutations.

Sections du résumé

BACKGROUND
A major response to pre-hepatectomy chemotherapy has been associated with improved survival in patients who undergo resection of colorectal liver metastases (CRLM). However, the role of tumor biology, as exemplified by overall and codon-specific KRAS mutational status, in predicting response to chemotherapy is not well defined.
METHODS
Pathologic response was characterized as minor or major depending on the percentage of remnant viable cells (>50% vs <50%, respectively). Multivariable logistic regression was used to identify factors associated with major response.
RESULTS
319 patients met inclusion criteria. 229 patients had a KRAS wild-type (wtKRAS) tumor and 90 harbored KRAS mutations (mutKRAS). A major pathologic response was more commonly noted in patients with wtKRAS compared to mutKRAS (48.5% vs 33.3%, P = 0.01) and wtKRAS status remained independently associated with a major response (P = 0.04). On a codon-specific level, major pathologic response occurred less frequently in those with codon 13 mutations (17.7%) compared to those with codon 12 (35.4%), and other KRAS mutations (33.3%). Importantly, codon 13 mutations were independently associated with minor pathologic response (P = 0.023).
CONCLUSIONS
Patients with wtKRAS tumors appear to have the highest likelihood of experiencing a major response after preoperative chemotherapy. Future studies in "all-comer" cohorts are needed to confirm these findings and further investigate the response of codon 13 mutations.

Identifiants

pubmed: 30979646
pii: S1365-182X(19)30493-9
doi: 10.1016/j.hpb.2019.03.368
pii:
doi:

Substances chimiques

Biomarkers, Tumor 0
Codon 0
KRAS protein, human 0
Proto-Oncogene Proteins p21(ras) EC 3.6.5.2

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1527-1534

Informations de copyright

Copyright © 2019 International Hepato-Pancreato-Biliary Association Inc. Published by Elsevier Ltd. All rights reserved.

Auteurs

Georgios A Margonis (GA)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Neda Amini (N)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Nikolaos Andreatos (N)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Kazunari Sasaki (K)

Department of General Surgery, Digestive Disease Institute, Cleveland Clinic, Cleveland, OH, USA.

Jack McVey (J)

Department of General Surgery, Digestive Disease Institute, Cleveland Clinic, Cleveland, OH, USA.

Muhammad B Mirza (MB)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Samuel Warner (S)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Stefan Buettner (S)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Carlotta Barbon (C)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Jane Wang (J)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Alessandra Pulvirenti (A)

Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Anastasios Angelou (A)

First Surgery Department, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.

Carsten Kamphues (C)

Department of General, Visceral and Vascular Surgery, Charite Campus Benjamin Franklin, Berlin, Germany.

Efstathios Antoniou (E)

First Surgery Department, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.

Emmanouil Pikoulis (E)

First Surgery Department, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.

Timothy M Pawlik (TM)

Department of Surgery, Wexner Medical Center at the Ohio State University, Columbus, OH, United States.

Klaus Kaczirek (K)

Department of General Surgery, Medical University of Vienna, Vienna, Austria.

George Poultsides (G)

Department of Surgery, Stanford University School of Medicine, Stanford, CA, USA.

Doris Wagner (D)

Department of General Surgery, Medical University of Graz, Graz, Austria.

Itaru Endo (I)

Department of Department of Gastroenterological Surgery, Yokohama City University, Yokohama, Japan.

Katsunori Imai (K)

Department of Gastroenterological Surgery, Graduate School of Life Sciences Kumamoto University, Kumamoto, Japan.

Federico Aucejo (F)

Department of General Surgery, Digestive Disease Institute, Cleveland Clinic, Cleveland, OH, USA.

Martin E Kreis (ME)

Department of General, Visceral and Vascular Surgery, Charite Campus Benjamin Franklin, Berlin, Germany.

Christopher L Wolfgang (CL)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA.

Matthew J Weiss (MJ)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, USA. Electronic address: mweiss5@jhmi.edu.

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Classifications MeSH