No correlation between Torque Teno virus viral load and BK virus replication after kidney transplantation.


Journal

Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology
ISSN: 1873-5967
Titre abrégé: J Clin Virol
Pays: Netherlands
ID NLM: 9815671

Informations de publication

Date de publication:
07 2019
Historique:
received: 11 02 2019
revised: 20 03 2019
accepted: 30 03 2019
pubmed: 16 4 2019
medline: 19 6 2020
entrez: 16 4 2019
Statut: ppublish

Résumé

Assessment of the intensity of immunosuppression in transplant recipients to estimate the risk of rejection and infection is not entirely satisfactory at the present time. Determination of Torque teno virus (TTV) viral load appears to be a promising tool in this setting. We evaluated the level of replication and kinetics of TTV during the first three months after kidney transplantation compared to BK virus replication. In a retrospective cohort of 116 renal transplant recipients, TTV viral load gradually increased during the first three months post-transplantation with no significant difference or discriminatory threshold between patients with and without BK virus replication. However, the level of TTV replication appeared to be indirectly related to the risk of BK virus replication, particularly according to the induction treatment used (antithymocyte globulin: ATG or basiliximab). Among patients receiving ATG, those receiving cyclosporine had significantly lower TTV viral loads (p < 0.01) with threefold lower reactivation of BKPyV (13 vs 37%) 3 months post-transplantation. Similarly, among the women in our cohort, TTV viral load was significantly higher in women receiving ATG (6.58 ± 1.57 versus 4.62 ± 2.0 log The multiparametric variation of TTV viral load does not appear to be individually appropriate for the early detection or monitoring of possible post-transplant BKPyV virus reactivation in renal transplant recipients.

Sections du résumé

BACKGROUND
Assessment of the intensity of immunosuppression in transplant recipients to estimate the risk of rejection and infection is not entirely satisfactory at the present time. Determination of Torque teno virus (TTV) viral load appears to be a promising tool in this setting.
OBJECTIVES
We evaluated the level of replication and kinetics of TTV during the first three months after kidney transplantation compared to BK virus replication.
RESULTS
In a retrospective cohort of 116 renal transplant recipients, TTV viral load gradually increased during the first three months post-transplantation with no significant difference or discriminatory threshold between patients with and without BK virus replication. However, the level of TTV replication appeared to be indirectly related to the risk of BK virus replication, particularly according to the induction treatment used (antithymocyte globulin: ATG or basiliximab). Among patients receiving ATG, those receiving cyclosporine had significantly lower TTV viral loads (p < 0.01) with threefold lower reactivation of BKPyV (13 vs 37%) 3 months post-transplantation. Similarly, among the women in our cohort, TTV viral load was significantly higher in women receiving ATG (6.58 ± 1.57 versus 4.62 ± 2.0 log
CONCLUSION
The multiparametric variation of TTV viral load does not appear to be individually appropriate for the early detection or monitoring of possible post-transplant BKPyV virus reactivation in renal transplant recipients.

Identifiants

pubmed: 30986626
pii: S1386-6532(19)30074-5
doi: 10.1016/j.jcv.2019.03.018
pii:
doi:

Substances chimiques

Biomarkers 0
DNA, Viral 0
Immunosuppressive Agents 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

4-6

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Lynda Handala (L)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Véronique Descamps (V)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Virginie Morel (V)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Sandrine Castelain (S)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Catherine François (C)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Gilles Duverlie (G)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

François Helle (F)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France.

Etienne Brochot (E)

Department of Virology, Amiens University Medical Center, Amiens, France; AGIR Research Unit, EA4294, Jules Verne University of Picardie, Amiens, France. Electronic address: etienne.brochot@u-picardie.fr.

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