Low Golimumab Trough Levels at Week 6 Are Associated With Poor Clinical, Endoscopic and Histological Outcomes in Ulcerative Colitis Patients: Pharmacokinetic and Pharmacodynamic Sub-analysis of the Evolution Study.


Journal

Journal of Crohn's & colitis
ISSN: 1876-4479
Titre abrégé: J Crohns Colitis
Pays: England
ID NLM: 101318676

Informations de publication

Date de publication:
28 Oct 2019
Historique:
pubmed: 17 4 2019
medline: 31 3 2020
entrez: 17 4 2019
Statut: ppublish

Résumé

Golimumab has an established exposure-response relationship in patients with ulcerative colitis [UC]. However, the association of serum golimumab trough levels [TL] with objective markers of disease activity, such as endoscopic and histological activity scores and concentrations of biomarkers, remains less understood. This report describes the relationship of serum golimumab TL at the end of the induction period [Week 6] with clinical, endoscopic, histological, and biomarker parameters. This was an open-label, uncontrolled, prospective and interventional study. Moderate to severely active UC patients naïve to biologic therapy were treated with golimumab. Serum golimumab TL and faecal calprotectin levels were measured at baseline [Week 0 of induction] and Week 6. A total of 34 patients completed the induction phase [Week 6] and were included in this analysis. Overall, 47.1% and 14.7% of patients achieved clinical response and remission with significantly higher serum golimumab TL in patients with early response or remission [3.7 μg/mL vs 1.3 μg/mL, p = 0.0013; and 3.1 μg/mL vs 1.7 μg/mL, p = 0.0164, respectively]. In addition, golimumab TL were significantly higher in patients achieving histological remission [4.2 μg/mL vs 1.7 μg/mL, p = 0.0049]. Week 6 golimumab TL were inversely correlated with the total Mayo score [rs = -0.546; p = 0.0008], the Mayo endoscopic subscore [rs = -0.381; p = 0.0262], the Geboes histological activity score [rs = -0.464; p = 0.0057], and faecal calprotectin levels [rs = -0.497; p = 0.0044]. A higher early exposure to golimumab is associated with a better objective response in active UC patients and appears to drive the outcome at Week 6.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
Golimumab has an established exposure-response relationship in patients with ulcerative colitis [UC]. However, the association of serum golimumab trough levels [TL] with objective markers of disease activity, such as endoscopic and histological activity scores and concentrations of biomarkers, remains less understood. This report describes the relationship of serum golimumab TL at the end of the induction period [Week 6] with clinical, endoscopic, histological, and biomarker parameters.
METHODS METHODS
This was an open-label, uncontrolled, prospective and interventional study. Moderate to severely active UC patients naïve to biologic therapy were treated with golimumab. Serum golimumab TL and faecal calprotectin levels were measured at baseline [Week 0 of induction] and Week 6.
RESULTS RESULTS
A total of 34 patients completed the induction phase [Week 6] and were included in this analysis. Overall, 47.1% and 14.7% of patients achieved clinical response and remission with significantly higher serum golimumab TL in patients with early response or remission [3.7 μg/mL vs 1.3 μg/mL, p = 0.0013; and 3.1 μg/mL vs 1.7 μg/mL, p = 0.0164, respectively]. In addition, golimumab TL were significantly higher in patients achieving histological remission [4.2 μg/mL vs 1.7 μg/mL, p = 0.0049]. Week 6 golimumab TL were inversely correlated with the total Mayo score [rs = -0.546; p = 0.0008], the Mayo endoscopic subscore [rs = -0.381; p = 0.0262], the Geboes histological activity score [rs = -0.464; p = 0.0057], and faecal calprotectin levels [rs = -0.497; p = 0.0044].
CONCLUSIONS CONCLUSIONS
A higher early exposure to golimumab is associated with a better objective response in active UC patients and appears to drive the outcome at Week 6.

Identifiants

pubmed: 30989180
pii: 5466449
doi: 10.1093/ecco-jcc/jjz071
doi:

Substances chimiques

Antibodies, Monoclonal 0
Biomarkers 0
Gastrointestinal Agents 0
Glucocorticoids 0
Immunosuppressive Agents 0
Leukocyte L1 Antigen Complex 0
C-Reactive Protein 9007-41-4
golimumab 91X1KLU43E

Types de publication

Clinical Trial Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1387-1393

Informations de copyright

Copyright © 2019 European Crohn’s and Colitis Organisation (ECCO). Published by Oxford University Press. All rights reserved. For permissions, please email: journals.permissions@oup.com.

Auteurs

F Magro (F)

Centro Hospitalar São João, Departamento de Gastrenterologia, Porto, Portugal.
Faculdade de Medicina, Universidade do Porto, Porto, Portugal.

S Lopes (S)

Centro Hospitalar São João, Departamento de Gastrenterologia, Porto, Portugal.

M Silva (M)

Centro Hospitalar São João, Departamento de Gastrenterologia, Porto, Portugal.

R Coelho (R)

Centro Hospitalar São João, Departamento de Gastrenterologia, Porto, Portugal.

F Portela (F)

Centro Hospitalar Universitário de Coimbra, Departamento de Gastrenterologia, Coimbra, Portugal.

D Branquinho (D)

Centro Hospitalar Universitário de Coimbra, Departamento de Gastrenterologia, Coimbra, Portugal.

L Correia (L)

Centro Hospitalar Lisboa Norte, Departamento de Gastrenterologia, Lisboa, Portugal.

S Fernandes (S)

Centro Hospitalar Lisboa Norte, Departamento de Gastrenterologia, Lisboa, Portugal.

M Cravo (M)

Hospital Beatriz Ângelo, Departamento de Gastrenterologia, Loures, Portugal.

P Caldeira (P)

Centro Hospitalar Universitário do Algarve, Departamento de Ciências Biomédicas e Medicina, Universidade do Algarve, Algarve Biomedical Centre, Universidade do Algarve, Faro, Portugal.

H T Sousa (HT)

Centro Hospitalar Universitário do Algarve, Departamento de Ciências Biomédicas e Medicina, Universidade do Algarve, Algarve Biomedical Centre, Universidade do Algarve, Faro, Portugal.

M Patita (M)

Hospital Garcia de Orta, Departamento de Gastrenterologia, Almada, Portugal.

P Lago (P)

Centro Hospitalar do Porto, Departamento de Gastrenterologia, Porto, Portugal.

J Ramos (J)

Centro Hospitalar Lisboa Central, Departamento de Gastrenterologia, Lisboa, Portugal.

J Afonso (J)

Faculdade de Medicina, Universidade do Porto, Porto, Portugal.

I Redondo (I)

MSD Portugal, Medical Affairs, Paço de Arcos, Portugal.

P Machado (P)

MSD Portugal, Medical Affairs, Paço de Arcos, Portugal.

F Cornillie (F)

Merck Sharp & Dohme, Kriens, Switzerland.

J Lopes (J)

Faculdade de Medicina, Universidade do Porto, Porto, Portugal.

F Carneiro (F)

Faculdade de Medicina, Universidade do Porto, Porto, Portugal.
Instituto de Patologia e Imunologia Molecular da Universidade do Porto [Ipatimup], i3S-Instituto de Investigação e Inovação em Saúde, Universidade do Porto, Porto, Portugal.

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Classifications MeSH