Cardiac syncope recurrence in type 2 diabetes mellitus patients vs. normoglycemics patients: The CARVAS study.


Journal

Diabetes research and clinical practice
ISSN: 1872-8227
Titre abrégé: Diabetes Res Clin Pract
Pays: Ireland
ID NLM: 8508335

Informations de publication

Date de publication:
May 2019
Historique:
received: 19 02 2019
revised: 01 04 2019
accepted: 12 04 2019
pubmed: 21 4 2019
medline: 14 8 2019
entrez: 21 4 2019
Statut: ppublish

Résumé

Cardiac autonomic dysfunction might lead to higher vaso vagal syncope (VVS) recurrence rate in type 2 diabetes mellitus (T2DM) patients vs. non diabetics patients. VVS recurrence might be due to alterations of autonomic system function, as assessed by heart rate variability (HRV). To date, in this study we investigated the correlation between HRV alterations and VVS recurrence at 12 months of follow up in T2DM vs. non T2DM patients. In a prospective multicenter study we studied a propensity score matching (PSM) analysis of 121 T2DM vs. 121 non T2DM patients affected by VVS. T2DM vs. non T2DM patients had at baseline a higher rate of HRV dysfunction, and this was linked to higher rate of VVS recurrence at 12 months of follow up (p < 0.05). Blood pressure alterations and lower LF/HF ratio were linked to higher rate of all cause syncope recurrence, and of vasodepressor, cardio inhibitory, and mixed syncope recurrence (p < 0.05). Anti hypertensive drug therapies increased the number of vasodepressor and mixed syncope events (p < 0.05); alterations of heart rate increased syncope recurrence and mixed syncope recurrence events (p < 0.05). Finally, T2DM was linked to higher rate of VVS recurrence, and specifically of vasodepressor and mixed VVS recurrence (p < 0.05). T2DM patients have alterations of the autonomic nervous system, as result of cardiac autonomic neuropathy. However, T2DM diagnosis and autonomic dysfunction assessed by HRV alterations predicted VVS recurrence.

Sections du résumé

STUDY HYPOTHESIS OBJECTIVE
Cardiac autonomic dysfunction might lead to higher vaso vagal syncope (VVS) recurrence rate in type 2 diabetes mellitus (T2DM) patients vs. non diabetics patients.
BACKGROUND BACKGROUND
VVS recurrence might be due to alterations of autonomic system function, as assessed by heart rate variability (HRV). To date, in this study we investigated the correlation between HRV alterations and VVS recurrence at 12 months of follow up in T2DM vs. non T2DM patients.
MATERIALS AND METHODS METHODS
In a prospective multicenter study we studied a propensity score matching (PSM) analysis of 121 T2DM vs. 121 non T2DM patients affected by VVS.
RESULTS RESULTS
T2DM vs. non T2DM patients had at baseline a higher rate of HRV dysfunction, and this was linked to higher rate of VVS recurrence at 12 months of follow up (p < 0.05). Blood pressure alterations and lower LF/HF ratio were linked to higher rate of all cause syncope recurrence, and of vasodepressor, cardio inhibitory, and mixed syncope recurrence (p < 0.05). Anti hypertensive drug therapies increased the number of vasodepressor and mixed syncope events (p < 0.05); alterations of heart rate increased syncope recurrence and mixed syncope recurrence events (p < 0.05). Finally, T2DM was linked to higher rate of VVS recurrence, and specifically of vasodepressor and mixed VVS recurrence (p < 0.05).
CONCLUSIONS CONCLUSIONS
T2DM patients have alterations of the autonomic nervous system, as result of cardiac autonomic neuropathy. However, T2DM diagnosis and autonomic dysfunction assessed by HRV alterations predicted VVS recurrence.

Identifiants

pubmed: 31004672
pii: S0168-8227(19)30257-8
doi: 10.1016/j.diabres.2019.04.015
pii:
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Pagination

152-162

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Celestino Sardu (C)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: drsarducele@gmail.com.

Pasquale Paolisso (P)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy.

Matteo Santamaria (M)

Cardiovascular and Arrhythmias Department, Juan Paul II Research and Care Foundation, Campobasso, Italy. Electronic address: matteo.santamaria@fgps.it.

Cosimo Sacra (C)

Cardiovascular and Arrhythmias Department, Juan Paul II Research and Care Foundation, Campobasso, Italy. Electronic address: cosimo.sacra@fgps.it.

Gorizio Pieretti (G)

Department of Precision Medicine, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: gorizio.pieretti@unicampania.it.

Maria Rosaria Rizzo (MR)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: mariarosaria.rizzo@unicampania.it.

Michelangela Barbieri (M)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: michelangela.barbieri@unicampania.it.

Lucia Scisciola (L)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: lucia.scisciola@unicampania.it.

Gianfranco Nicoletti (G)

Department of Precision Medicine, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: Gianfranco.nicoletti@unicampania.it.

Giuseppe Paolisso (G)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: giuseppe.paolisso@unicampania.it.

Raffaele Marfella (R)

Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy. Electronic address: raffaele.marfella@unicampania.it.

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