Defective Renal Angiotensin III and AT


Journal

Journal of the American Heart Association
ISSN: 2047-9980
Titre abrégé: J Am Heart Assoc
Pays: England
ID NLM: 101580524

Informations de publication

Date de publication:
07 05 2019
Historique:
entrez: 2 5 2019
pubmed: 2 5 2019
medline: 4 8 2020
Statut: ppublish

Résumé

Background Previous studies demonstrated that angiotensin (Ang) III , not Ang II , is the predominant endogenous agonist for Ang type-2 receptor ( AT

Identifiants

pubmed: 31039659
doi: 10.1161/JAHA.119.012016
pmc: PMC6512109
doi:

Substances chimiques

Receptor, Angiotensin, Type 2 0
Slc9a3 protein, rat 0
Sodium-Hydrogen Exchanger 3 0
Angiotensin III 12687-51-3
Sodium-Potassium-Exchanging ATPase EC 7.2.2.13

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

e012016

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL128189
Pays : United States

Commentaires et corrections

Type : CommentIn

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Auteurs

Brandon A Kemp (BA)

1 Division of Endocrinology and Metabolism Department of Medicine University of Virginia School of Medicine Charlottesville VA.

Nancy L Howell (NL)

1 Division of Endocrinology and Metabolism Department of Medicine University of Virginia School of Medicine Charlottesville VA.

Susanna R Keller (SR)

1 Division of Endocrinology and Metabolism Department of Medicine University of Virginia School of Medicine Charlottesville VA.

John J Gildea (JJ)

2 Department of Pathology University of Virginia School of Medicine Charlottesville VA.

Weijian Shao (W)

3 Department of Physiology and Hypertension and Renal Center Tulane University School of Medicine New Orleans LA.

Luis Gabriel Navar (LG)

3 Department of Physiology and Hypertension and Renal Center Tulane University School of Medicine New Orleans LA.

Robert M Carey (RM)

1 Division of Endocrinology and Metabolism Department of Medicine University of Virginia School of Medicine Charlottesville VA.

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Classifications MeSH