Functions of estrogen and estrogen receptor signaling on skeletal muscle.


Journal

The Journal of steroid biochemistry and molecular biology
ISSN: 1879-1220
Titre abrégé: J Steroid Biochem Mol Biol
Pays: England
ID NLM: 9015483

Informations de publication

Date de publication:
07 2019
Historique:
received: 27 03 2019
accepted: 03 05 2019
pubmed: 9 5 2019
medline: 21 11 2019
entrez: 9 5 2019
Statut: ppublish

Résumé

Activity of estrogen, a sex steroid hormone, is not only limited to the reproductive organs but also involves other organs and tissues, including skeletal muscle. In postmenopausal women, estrogen decline causes endocrine and metabolic dysfunction, leading to a predisposition to osteoporosis, metabolic syndrome, and decreased muscle mass and strength. The decline in skeletal muscle mass often associates with sarcopenia, a popular condition observed in fragile elder people. In addition, varying estrogen levels associated with the menstrual phases may modulate exercise performance in women. Estrogen is thus considered to play a crucial role in skeletal muscle homeostasis and exercise capacity, although its precise mechanisms remain to be elucidated. In this article, we review the role of estrogen in the skeletal muscle, outlining the proposed molecular mechanisms. We especially focus on the current understanding of estrogen actions on mitochondria metabolism in skeletal muscle.

Identifiants

pubmed: 31067490
pii: S0960-0760(19)30185-2
doi: 10.1016/j.jsbmb.2019.105375
pii:
doi:

Substances chimiques

Estrogens 0
Receptors, Estrogen 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

105375

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Kazuhiro Ikeda (K)

Division of Gene Regulation and Signal Transduction, Research Center for Genomic Medicine, Saitama Medical University, Saitama, Japan.

Kuniko Horie-Inoue (K)

Division of Gene Regulation and Signal Transduction, Research Center for Genomic Medicine, Saitama Medical University, Saitama, Japan.

Satoshi Inoue (S)

Division of Gene Regulation and Signal Transduction, Research Center for Genomic Medicine, Saitama Medical University, Saitama, Japan; Department of Systems Aging Science and Medicine, Tokyo Metropolitan Institute of Gerontology, Tokyo, Japan. Electronic address: sinoue@tmig.or.jp.

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Classifications MeSH