Niemann-Pick Type A Disease: Behavior of Neutral Sphingomyelinase and Vitamin D Receptor.
Niemann-Pick type A disease
Toll-like receptors
neutral sphingomyelinase
vitamin D receptor
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
13 May 2019
13 May 2019
Historique:
received:
22
03
2019
revised:
03
05
2019
accepted:
07
05
2019
entrez:
16
5
2019
pubmed:
16
5
2019
medline:
4
9
2019
Statut:
epublish
Résumé
Sphingomyelinase (SMase) is responsible for the breakdown of sphingomyelin (SM) with production of ceramide. The absence of acid sphingomyelinase (aSMase) causes abnormal synapse formation in Niemann-Pick type A (NPA) disease. Because high levels of ceramide in the NPA brain were demonstrated, the involvement of other SMases were supposed. In the present study we focused the attention on the neurogenic niches in the hippocampal
Identifiants
pubmed: 31086057
pii: ijms20092365
doi: 10.3390/ijms20092365
pmc: PMC6539364
pii:
doi:
Substances chimiques
Receptors, Calcitriol
0
Toll-Like Receptor 2
0
Toll-Like Receptor 7
0
Toll-Like Receptor 8
0
Sphingomyelin Phosphodiesterase
EC 3.1.4.12
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Références
Science. 2000 Feb 25;287(5457):1433-8
pubmed: 10688783
Biochim Biophys Acta. 2000 Apr 12;1484(2-3):129-41
pubmed: 10760463
Neuroscience. 2002;109(2):371-88
pubmed: 11801372
Biol Cell. 2004 Oct;96(8):657-67
pubmed: 15519699
Brain Res Bull. 2005 Mar 15;65(2):141-8
pubmed: 15763180
J Biol Chem. 2005 May 13;280(19):19196-204
pubmed: 15769735
Neurology. 2006 Jan 24;66(2):228-32
pubmed: 16434659
Nat Cell Biol. 2007 Sep;9(9):1081-8
pubmed: 17704767
PLoS One. 2009;4(4):e5310
pubmed: 19390577
Endocrinol Metab Clin North Am. 2010 Jun;39(2):255-69, table of contents
pubmed: 20511050
Proc Natl Acad Sci U S A. 2010 Aug 31;107(35):15625-30
pubmed: 20713712
Mol Biol Cell. 2011 Sep;22(17):3022-31
pubmed: 21737687
PLoS One. 2012;7(5):e37767
pubmed: 22666391
Handb Clin Neurol. 2013;113:1717-21
pubmed: 23622394
J Chem Neuroanat. 2013 Sep;52:1-8
pubmed: 23660496
EMBO Mol Med. 2014 Mar;6(3):398-413
pubmed: 24448491
Front Neurosci. 2014 Aug 28;8:272
pubmed: 25221470
Int J Mol Sci. 2015 Jan 22;16(2):2307-19
pubmed: 25622250
Neuropharmacology. 2016 Apr;103:211-21
pubmed: 26686389
J Lipid Res. 2016 Sep;57(9):1644-58
pubmed: 27402802
Neurochem Res. 2017 Feb;42(2):493-500
pubmed: 28108849
Mol Genet Metab. 2017 Jan - Feb;120(1-2):27-33
pubmed: 28164782
Int J Mol Sci. 2017 Jul 05;18(7):null
pubmed: 28678158
Scand J Immunol. 2017 Nov;86(5):377-388
pubmed: 28889482
Biochimie. 2018 Mar;146:35-42
pubmed: 29158006
Toxicol In Vitro. 2018 Apr;48:45-52
pubmed: 29273543
Mediators Inflamm. 2017;2017:2470950
pubmed: 29343884
Front Cell Neurosci. 2018 Feb 07;12:31
pubmed: 29467625
J Mol Neurosci. 2018 Apr;64(4):611-618
pubmed: 29589201
Sci Rep. 2018 Jun 27;8(1):9741
pubmed: 29950608
PLoS One. 2018 Jul 6;13(7):e0200344
pubmed: 29979789
Int J Mol Sci. 2018 Nov 01;19(11):null
pubmed: 30388783
Cell. 1995 Jun 30;81(7):1053-61
pubmed: 7600574
Nat Genet. 1995 Jul;10(3):288-93
pubmed: 7670466