Serial C-Reactive Protein Measurements in Newborn Infants without Evidence of Early-Onset Infection.


Journal

Neonatology
ISSN: 1661-7819
Titre abrégé: Neonatology
Pays: Switzerland
ID NLM: 101286577

Informations de publication

Date de publication:
2019
Historique:
received: 02 10 2018
accepted: 25 01 2019
pubmed: 22 5 2019
medline: 22 5 2020
entrez: 22 5 2019
Statut: ppublish

Résumé

C-reactive protein (CRP) is used to assist the diagnosis and monitoring of newborn infection. Little is known about CRP activity after birth in the absence of infection. The aim of this work was to describe postnatal CRP responses in the first days of life in asymptomatic infants with a negative blood culture. Data were collected from infants who had a blood culture taken at <72 h of age in a UK maternity hospital. All CRP values and their time from birth were recorded. Infants with signs of infection, positive blood culture, or major congenital anomalies were excluded. Infants were analysed by gestation (greater or less than 37 weeks). Normalised CRP curves were generated by linear interpolation and centile curves were derived. Comparisons of median CRP values between groups were made by Mann-Whitney U test at 24, 36, and 48 h. During the study period a total of 219 babies were screened. After exclusions, 73 infants (58 term, 15 preterm) were analysed. In asymptomatic term neonates the CRP (mg/L) peaked at 9.4 after 34.6 h. In preterm babies the CRP peak was 1.75 at 43 h. The median (IQR) values were higher in the term group at 24 and 36 h: 2.5 (1-10.5) versus 0 (0-2.2; p = 0.02) and 3 (0-8.6) versus 0 (0-2.8; p = 0.031). A CRP rise was demonstrated in term and preterm infants without evidence of infection. This rise was greatest in term infants. CRP values must be interpreted in the context of an infant's clinical condition and not used alone to guide clinical decision making.

Sections du résumé

BACKGROUND
C-reactive protein (CRP) is used to assist the diagnosis and monitoring of newborn infection. Little is known about CRP activity after birth in the absence of infection.
OBJECTIVE
The aim of this work was to describe postnatal CRP responses in the first days of life in asymptomatic infants with a negative blood culture.
METHODS
Data were collected from infants who had a blood culture taken at <72 h of age in a UK maternity hospital. All CRP values and their time from birth were recorded. Infants with signs of infection, positive blood culture, or major congenital anomalies were excluded. Infants were analysed by gestation (greater or less than 37 weeks). Normalised CRP curves were generated by linear interpolation and centile curves were derived. Comparisons of median CRP values between groups were made by Mann-Whitney U test at 24, 36, and 48 h.
RESULTS
During the study period a total of 219 babies were screened. After exclusions, 73 infants (58 term, 15 preterm) were analysed. In asymptomatic term neonates the CRP (mg/L) peaked at 9.4 after 34.6 h. In preterm babies the CRP peak was 1.75 at 43 h. The median (IQR) values were higher in the term group at 24 and 36 h: 2.5 (1-10.5) versus 0 (0-2.2; p = 0.02) and 3 (0-8.6) versus 0 (0-2.8; p = 0.031).
CONCLUSIONS
A CRP rise was demonstrated in term and preterm infants without evidence of infection. This rise was greatest in term infants. CRP values must be interpreted in the context of an infant's clinical condition and not used alone to guide clinical decision making.

Identifiants

pubmed: 31112949
pii: 000497237
doi: 10.1159/000497237
doi:

Substances chimiques

C-Reactive Protein 9007-41-4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

85-91

Informations de copyright

© 2019 S. Karger AG, Basel.

Auteurs

Kathryn Macallister (K)

Neonatal Directorate, King Edward Memorial and Perth Children's Hospitals, Women and Newborn Health Service, Perth, Washington, Australia.
Neonatal Department, Gloucestershire Royal Hospital NHS Foundation Trust, Gloucester, United Kingdom.

Adam Smith-Collins (A)

Regional Neonatal intensive Care Unit, St. Michael's Hospital, University Hospital Bristol, NHS Foundation Trust, Bristol, United Kingdom.
Department of Neonatal Neuroscience, School of Clinical Sciences, University of Bristol, Bristol, United Kingdom.

Helen Gillet (H)

Regional Neonatal intensive Care Unit, St. Michael's Hospital, University Hospital Bristol, NHS Foundation Trust, Bristol, United Kingdom.

Linze Hamilton (L)

Regional Neonatal intensive Care Unit, St. Michael's Hospital, University Hospital Bristol, NHS Foundation Trust, Bristol, United Kingdom.

Jonathan Davis (J)

Neonatal Directorate, King Edward Memorial and Perth Children's Hospitals, Women and Newborn Health Service, Perth, Washington, Australia, jonathan.davis@uwa.edu.au.
Centre for Newborn Research and Education, School of Child and Paediatric Health, University of Western Australia, Perth, Washington, Australia, jonathan.davis@uwa.edu.au.

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