Pravastatin combination with sorafenib does not improve survival in advanced hepatocellular carcinoma.


Journal

Journal of hepatology
ISSN: 1600-0641
Titre abrégé: J Hepatol
Pays: Netherlands
ID NLM: 8503886

Informations de publication

Date de publication:
09 2019
Historique:
received: 30 10 2018
revised: 23 04 2019
accepted: 30 04 2019
pubmed: 28 5 2019
medline: 15 12 2020
entrez: 25 5 2019
Statut: ppublish

Résumé

Sorafenib is the standard of care for advanced hepatocellular carcinoma (HCC). Combining sorafenib with another treatment, to improve overall survival (OS) within an acceptable safety profile, might be the next step forward in the management of patients with advanced HCC. We aimed to assess whether a combination of sorafenib and a statin improved survival in patients with HCC. The objective of the PRODIGE-11 trial was to compare the respective clinical outcomes of the sorafenib-pravastatin combination (arm A) versus sorafenib alone (arm B) in patients with advanced HCC. Child-Pugh A patients with advanced HCC who were naive to systemic treatment (n = 323) were randomly assigned to sorafenib-pravastatin combination (n = 162) or sorafenib alone (n = 161). The primary endpoint was OS; secondary endpoints were progression-free survival, time to tumor progression, time to treatment failure, safety, and quality of life. After randomization, 312 patients received at least 1 dose of study treatment. After a median follow-up of 35 months, 269 patients died (arm A: 135; arm B: 134) with no difference in median OS between treatments arms (10.7 months vs. 10.5 months; hazard ratio = 1.00; p = 0.975); no difference was observed in secondary survival endpoints either. In the univariate analysis, the significant prognostic factors for OS were CLIP score, performance status, and quality of life scores. The multivariate analysis showed that the only prognostic factor for OS was the CLIP score. The main toxicity was diarrhea (which was severe in 11% of patients in arm A, and 8.9% in arm B), while severe nausea-vomiting was rare, and no toxicity-related deaths were reported. Adding pravastatin to sorafenib did not improve survival in patients with advanced HCC. Sorafenib has proven efficacy for the treatment of patients with advanced hepatocellular carcinoma. However, overall survival remains poor in these patients, so we were interested to see if the addition of a statin, pravastatin, improved outcomes in patients with advanced HCC. This randomized-controlled trial demonstrated that the sorafenib-pravastatin combination did not improve overall survival in this study population compared to sorafenib alone. Clinical trial number: NCT01075555.

Sections du résumé

BACKGROUND & AIMS
Sorafenib is the standard of care for advanced hepatocellular carcinoma (HCC). Combining sorafenib with another treatment, to improve overall survival (OS) within an acceptable safety profile, might be the next step forward in the management of patients with advanced HCC. We aimed to assess whether a combination of sorafenib and a statin improved survival in patients with HCC.
METHODS
The objective of the PRODIGE-11 trial was to compare the respective clinical outcomes of the sorafenib-pravastatin combination (arm A) versus sorafenib alone (arm B) in patients with advanced HCC. Child-Pugh A patients with advanced HCC who were naive to systemic treatment (n = 323) were randomly assigned to sorafenib-pravastatin combination (n = 162) or sorafenib alone (n = 161). The primary endpoint was OS; secondary endpoints were progression-free survival, time to tumor progression, time to treatment failure, safety, and quality of life.
RESULTS
After randomization, 312 patients received at least 1 dose of study treatment. After a median follow-up of 35 months, 269 patients died (arm A: 135; arm B: 134) with no difference in median OS between treatments arms (10.7 months vs. 10.5 months; hazard ratio = 1.00; p = 0.975); no difference was observed in secondary survival endpoints either. In the univariate analysis, the significant prognostic factors for OS were CLIP score, performance status, and quality of life scores. The multivariate analysis showed that the only prognostic factor for OS was the CLIP score. The main toxicity was diarrhea (which was severe in 11% of patients in arm A, and 8.9% in arm B), while severe nausea-vomiting was rare, and no toxicity-related deaths were reported.
CONCLUSION
Adding pravastatin to sorafenib did not improve survival in patients with advanced HCC.
LAY SUMMARY
Sorafenib has proven efficacy for the treatment of patients with advanced hepatocellular carcinoma. However, overall survival remains poor in these patients, so we were interested to see if the addition of a statin, pravastatin, improved outcomes in patients with advanced HCC. This randomized-controlled trial demonstrated that the sorafenib-pravastatin combination did not improve overall survival in this study population compared to sorafenib alone. Clinical trial number: NCT01075555.

Identifiants

pubmed: 31125576
pii: S0168-8278(19)30289-2
doi: 10.1016/j.jhep.2019.04.021
pii:
doi:

Substances chimiques

Antineoplastic Agents 0
Sorafenib 9ZOQ3TZI87
Pravastatin KXO2KT9N0G

Banques de données

ClinicalTrials.gov
['NCT01075555']

Types de publication

Clinical Trial, Phase III Comparative Study Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

516-522

Informations de copyright

Copyright © 2019 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

Auteurs

Jean-Louis Jouve (JL)

Department of Hepato-Gastroenterology, University Hospital F. Mitterrand, Dijon, France. Electronic address: jean-louis.jouve@chu-dijon.fr.

Thierry Lecomte (T)

Department of Hepato-Gastroenterology, University Hospital Trousseau, Tours, France.

Olivier Bouché (O)

Department of Hepato-Gastroenterology, Robert Debré Hospital, Reims, France.

Emilie Barbier (E)

Fédération Française de Cancérologie Digestive (FFCD), Dijon, France.

Faiza Khemissa Akouz (F)

Department of Hepato-Gastroenterology, Saint-Jean Hospital, Perpignan, France.

Ghassan Riachi (G)

Department of Hepato-Gastroenterology, University Hospital Charles Nicolle, Rouen, France.

Eric Nguyen Khac (E)

Department of Hepato-Gastroenterology, University Hospital Nord, Amiens, France.

Isabelle Ollivier-Hourmand (I)

Department of Hepato-Gastroenterology, University Hospital Côte de Nacre, Caen, France.

Maryline Debette-Gratien (M)

Department of Hepato-Gastroenterology, University Hospital Dupuytren, Limoges, France.

Roger Faroux (R)

Department of Hepato-Gastroenterology, Les Oudairies Hospital, La Roche-sur-Yon, France.

Anne-Laure Villing (AL)

Department of Medical Oncology, Auxerre Hospital, Auxerre, France.

Julien Vergniol (J)

Department of Hepato-Gastroenterology, University Hospital Haut-Lévêque, Pessac, France.

Jean-François Ramee (JF)

Department of Medical Oncology, Centre Catherine de Sienne, Nantes, France.

Jean-Pierre Bronowicki (JP)

INSERM U954, University of Lorraine and University Hospital of Nancy, Vandoeuvre-les-Nancy, France.

Jean-François Seitz (JF)

Department of Oncology and Hepato-Gastroenterology, University Hospital La Timone, Marseille, France.

Jean-Louis Legoux (JL)

Department of Hepato-Gastroenterology, La Source Hospital, Orléans, France.

Jacques Denis (J)

Department of Hepato-Gastroenterology, Louise Michel Hospital, Evry, France.

Sylvain Manfredi (S)

Department of Hepato-Gastroenterology, University Hospital F. Mitterrand, Dijon, France; INSERM U1231, University of Bourgogne - Franche-Comté, Faculté de Médecine, Dijon, France.

Jean-Marc Phelip (JM)

Department of Hepato-Gastroenterology, University Hospital of Saint-Etienne - Hôpital Nord, Saint-Priest-en-Jarez, France.

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