Targeted AAVP-based therapy in a mouse model of human glioblastoma: a comparison of cytotoxic versus suicide gene delivery strategies.


Journal

Cancer gene therapy
ISSN: 1476-5500
Titre abrégé: Cancer Gene Ther
Pays: England
ID NLM: 9432230

Informations de publication

Date de publication:
05 2020
Historique:
received: 19 12 2018
accepted: 27 04 2019
revised: 09 04 2019
pubmed: 28 5 2019
medline: 11 5 2021
entrez: 28 5 2019
Statut: ppublish

Résumé

Glioblastoma persists as a uniformly deadly diagnosis for patients and effective therapeutic options are gravely needed. Recently, targeted gene therapy approaches are reemerging as attractive experimental clinical agents. Our ligand-directed hybrid virus of adeno-associated virus and phage (AAVP) is a targeted gene delivery vector that has been used in several formulations displaying targeting ligand peptides to deliver clinically applicable transgenes. Here we compared different constructs side-by-side in a tumor model, an orthotopic model of xenograft human glioblastoma cells stereotactically implanted in immunodeficient mice. We have used divergent therapeutic strategies for two AAVP constructs, both displaying a double-cyclic RGD4C motif ligand specific for alpha V integrins expressed in tumor vascular endothelium, but carrying different genes of interest for the treatment of intracranial xenografted tumors. One construct delivered tumor necrosis factor (TNF), a purely cytotoxic gene for antitumor activity (RGD4C-AAVP-TNF); in the other construct, we delivered Herpes simplex virus thymidine kinase (HSVtk) for in tandem molecular-genetic imaging and targeted therapy (RGD4C-AAVP-HSVtk) utilizing ganciclovir (GCV) for a suicide gene therapy. Both AAVP constructs demonstrated antitumor activity, with damage to the tumor-associated neovasculature and induction of cell death evident after treatment. In addition, the ability to monitor transgene expression with a radiolabeled HSVtk substrate pre and post GCV treatment demonstrated the theranostic potential of RGD4C-AAVP-HSVtk. We conclude that targeted AAVP constructs delivering either cytotoxic TNF or theranostic HSVtk followed by suicide gene therapy with GCV have comparable preclinical efficacy, at least in this standard experimental model. The results presented here provide a blueprint for future studies of targeted gene delivery against human glioblastomas and other brain tumors.

Identifiants

pubmed: 31130731
doi: 10.1038/s41417-019-0101-2
pii: 10.1038/s41417-019-0101-2
pmc: PMC6879804
mid: NIHMS1528126
doi:

Substances chimiques

Antineoplastic Agents 0
TNF protein, human 0
Tumor Necrosis Factor-alpha 0
Viral Proteins 0
Thymidine Kinase EC 2.7.1.21
Ganciclovir P9G3CKZ4P5

Types de publication

Comparative Study Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

301-310

Subventions

Organisme : NCI NIH HHS
ID : R01 CA122568
Pays : United States

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Auteurs

Fernanda I Staquicini (FI)

Rutgers Cancer Institute of New Jersey and Division of Cancer Biology, Department of Radiation Oncology, Rutgers New Jersey Medical School, Newark, NJ, USA.

Tracey L Smith (TL)

Rutgers Cancer Institute of New Jersey and Division of Cancer Biology, Department of Radiation Oncology, Rutgers New Jersey Medical School, Newark, NJ, USA.

Fenny H F Tang (FHF)

Department of Biochemistry, Institute of Chemistry, University of São Paulo, São Paulo, São Paulo, Brazil.

Juri G Gelovani (JG)

Karmanos Cancer Institute, School of Medicine and Department of Biomedical Engineering, College of Engineering, Wayne State University, Detroit, MI, USA.

Ricardo J Giordano (RJ)

Department of Biochemistry, Institute of Chemistry, University of São Paulo, São Paulo, São Paulo, Brazil.

Steven K Libutti (SK)

Rutgers Cancer Institute of New Jersey and Department of Surgery, Rutgers Robert Wood Johnson Medical School, New Brunswick, NJ, USA.

Richard L Sidman (RL)

Department of Neurology, Harvard Medical School, Boston, MA, USA.

Webster K Cavenee (WK)

Ludwig Institute for Cancer Research, University of California-San Diego, La Jolla, CA, USA.

Wadih Arap (W)

Rutgers Cancer Institute of New Jersey and Division of Hematology/Oncology, Department of Medicine, Rutgers New Jersey Medical School, Newark, NJ, USA. wadih.arap@rutgers.edu.

Renata Pasqualini (R)

Rutgers Cancer Institute of New Jersey and Division of Cancer Biology, Department of Radiation Oncology, Rutgers New Jersey Medical School, Newark, NJ, USA. renata.pasqualini@rutgers.edu.

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