Pulmonary type-2 innate lymphoid cells in paediatric severe asthma: phenotype and response to steroids.


Journal

The European respiratory journal
ISSN: 1399-3003
Titre abrégé: Eur Respir J
Pays: England
ID NLM: 8803460

Informations de publication

Date de publication:
08 2019
Historique:
received: 26 09 2018
accepted: 26 05 2019
pubmed: 6 6 2019
medline: 30 9 2020
entrez: 6 6 2019
Statut: epublish

Résumé

Children with severe therapy-resistant asthma (STRA) have poor control despite maximal treatment, while those with difficult asthma (DA) have poor control from failure to implement basic management, including adherence to therapy. Although recognised as clinically distinct, the airway molecular phenotype, including the role of innate lymphoid cells (ILCs) and their response to steroids in DA and STRA is unknown.Immunophenotyping of sputum and blood ILCs and T-cells from STRA, DA and non-asthmatic controls was undertaken. Leukocytes were analysed longitudinally pre- and post-intramuscular triamcinolone in children with STRA. Cultured ILCs were evaluated to assess steroid responsiveness

Identifiants

pubmed: 31164437
pii: 13993003.01809-2018
doi: 10.1183/13993003.01809-2018
pmc: PMC6713888
pii:
doi:

Substances chimiques

IL17A protein, human 0
Interleukin-13 0
Interleukin-17 0
Steroids 0
Triamcinolone 1ZK20VI6TY

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 107059/Z/15/Z
Pays : United Kingdom

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright ©ERS 2019.

Déclaration de conflit d'intérêts

Conflict of interest: F. Puttur has nothing to disclose. Conflict of interest: L.G. Gregory has nothing to disclose. Conflict of interest: L. Denney has nothing to disclose. Conflict of interest: L. Fleming reports other funding from GSK, Sanofi, Novartis, Boehrringer Ingelheim and Astra Zeneca, outside the submitted work. Conflict of interest: A. Bush has nothing to disclose. Conflict of interest: C.M. Lloyd has nothing to disclose. Conflict of interest: S. Saglani has nothing to disclose. Conflict of interest: P. Nagakumar has nothing to disclose.

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Auteurs

Prasad Nagakumar (P)

National Heart and Lung Institute, Imperial College London, London, UK.
Respiratory Paediatrics, Royal Brompton Hospital and National Heart and Lung Institute, Imperial College London, London, UK.
Both authors contributed equally.

Franz Puttur (F)

National Heart and Lung Institute, Imperial College London, London, UK.
Both authors contributed equally.

Lisa G Gregory (LG)

National Heart and Lung Institute, Imperial College London, London, UK.

Laura Denney (L)

National Heart and Lung Institute, Imperial College London, London, UK.

Louise Fleming (L)

Respiratory Paediatrics, Royal Brompton Hospital and National Heart and Lung Institute, Imperial College London, London, UK.

Andrew Bush (A)

Respiratory Paediatrics, Royal Brompton Hospital and National Heart and Lung Institute, Imperial College London, London, UK.

Clare M Lloyd (CM)

National Heart and Lung Institute, Imperial College London, London, UK.
Both authors contributed equally.

Sejal Saglani (S)

National Heart and Lung Institute, Imperial College London, London, UK s.saglani@imperial.ac.uk.
Respiratory Paediatrics, Royal Brompton Hospital and National Heart and Lung Institute, Imperial College London, London, UK.
Both authors contributed equally.

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