Circulating endothelial glycocalyx components as a predictive marker of coronary artery lesions in Kawasaki disease.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
01 10 2019
Historique:
received: 24 01 2019
revised: 14 05 2019
accepted: 20 05 2019
pubmed: 16 6 2019
medline: 13 6 2020
entrez: 16 6 2019
Statut: ppublish

Résumé

Kawasaki disease (KD) is acute and self-limited vasculitis caused by unknown origin, and the critical complication in KD patients is coronary artery lesions (CALs). The endothelial glycocalyx is a network of membranes luminally covering the endothelium. This study aimed to evaluate the clinical utility of serum glycocalyx components as biomarkers of predicting the onset CALs in KD. Seventy subjects with complete type KD, 18 subjects as febrile control (FC), and 15 subjects as afebrile controls (AC) were enrolled. Medical, demographic, echocardiography, and laboratory data from the medical records were retrospectively analyzed. Serum syndecan-1 and hyaluronan levels prior to intravenous immunoglobulin (IVIG) therapy were measured at the acute phase, immediately after IVIG, the subacute phase, and the time of discharge at the convalescent phase. Serum syndecan-1 and hyaluronan levels were higher in the KD group than in the AC and FC groups at all three phases. Further, these levels were compared between KD patients with and without the development of CALs. Serum syndecan-1 and hyaluronan levels at the acute phase were significantly elevated in KD patients with the CALs than in those without CALs. Serum hyaluronan, not syndecan-1, was determined as the most contributory parameter to predict CALs by a multiple logistic analysis. Circulating syndecan-1 and hyaluronan can be useful biomarkers to predict the development of CALs in KD.

Sections du résumé

BACKGROUND
Kawasaki disease (KD) is acute and self-limited vasculitis caused by unknown origin, and the critical complication in KD patients is coronary artery lesions (CALs). The endothelial glycocalyx is a network of membranes luminally covering the endothelium. This study aimed to evaluate the clinical utility of serum glycocalyx components as biomarkers of predicting the onset CALs in KD.
METHODS
Seventy subjects with complete type KD, 18 subjects as febrile control (FC), and 15 subjects as afebrile controls (AC) were enrolled. Medical, demographic, echocardiography, and laboratory data from the medical records were retrospectively analyzed. Serum syndecan-1 and hyaluronan levels prior to intravenous immunoglobulin (IVIG) therapy were measured at the acute phase, immediately after IVIG, the subacute phase, and the time of discharge at the convalescent phase.
RESULTS
Serum syndecan-1 and hyaluronan levels were higher in the KD group than in the AC and FC groups at all three phases. Further, these levels were compared between KD patients with and without the development of CALs. Serum syndecan-1 and hyaluronan levels at the acute phase were significantly elevated in KD patients with the CALs than in those without CALs. Serum hyaluronan, not syndecan-1, was determined as the most contributory parameter to predict CALs by a multiple logistic analysis.
CONCLUSIONS
Circulating syndecan-1 and hyaluronan can be useful biomarkers to predict the development of CALs in KD.

Identifiants

pubmed: 31200965
pii: S0167-5273(19)30440-1
doi: 10.1016/j.ijcard.2019.05.045
pii:
doi:

Substances chimiques

Biomarkers 0
SDC1 protein, human 0
Syndecan-1 0
Hyaluronic Acid 9004-61-9

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

236-240

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Yuji Ohnishi (Y)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Hiroki Yasudo (H)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan. Electronic address: yasudo@yamaguchi-u.ac.jp.

Yasuo Suzuki (Y)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Takashi Furuta (T)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Chie Matsuguma (C)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Yoshihiro Azuma (Y)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Akiko Miyake (A)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan; Division of Pediatrics, Yamaguchi-ken Saiseikai Shimonoseki General Hospital, Shimonoseki, Japan.

Seigo Okada (S)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan; Division of Pediatrics, Yamaguchi-ken Saiseikai Shimonoseki General Hospital, Shimonoseki, Japan.

Kiyoshi Ichihara (K)

Department of Laboratory Sciences, Faculty of Health Sciences, Yamaguchi University Graduate School of Medicine, Ube, Japan.

Shouichi Ohga (S)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan; Department of Pediatrics, Kyushu University Graduate School of Medical Science, Fukuoka, Japan.

Shunji Hasegawa (S)

Department of Pediatrics, Yamaguchi University Graduate School of Medicine, Ube, Japan.

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Classifications MeSH