MUNC13-1 heterozygosity does not alter voluntary ethanol consumption or sensitivity in mice.


Journal

Alcohol (Fayetteville, N.Y.)
ISSN: 1873-6823
Titre abrégé: Alcohol
Pays: United States
ID NLM: 8502311

Informations de publication

Date de publication:
03 2020
Historique:
received: 12 02 2019
revised: 12 06 2019
accepted: 21 06 2019
pubmed: 3 7 2019
medline: 9 1 2021
entrez: 3 7 2019
Statut: ppublish

Résumé

The role of the munc13-1 presynaptic protein in alcohol-related behaviors has been little-studied, despite being a known site of action for ethanol binding. Munc13-1 is an active zone protein that plays a vital role in vesicle maturation and the release of neurotransmitters in excitatory neurons. Ethanol binds munc13-1, which decreases its functionality. In Drosophila, loss of the homologous protein Dunc13 is associated with an increase in ethanol preference, and is associated with a resistance to sedation following ethanol exposure. The current study assessed the effects of munc13-1 heterozygosity on ethanol sensitivity and consumption in mice, as well as on learning and anxiety-like behaviors, which can influence alcohol intake. Wild-type and mutant mice underwent 6 cycles of drinking-in-the-dark (DID) as well as rotarod testing following ethanol injection, to probe for differences in ethanol consumption and sensitivity, respectively. We did not detect genotype-based differences in our measures of anxiety, spatial learning, ethanol consumption, or ethanol sensitivity. However, heterozygotes showed increased use of a spatial navigation strategy in a dual-solution water maze, as opposed to a stimulus-response strategy. To summarize, although reduction of Dunc13 in flies produces clear effects on ethanol consumption and sensitivity, heterozygosity for munc13-1 does not, potentially due to compensatory adaptation by other munc-13 isoforms.

Identifiants

pubmed: 31265903
pii: S0741-8329(19)30013-8
doi: 10.1016/j.alcohol.2019.06.007
pmc: PMC7043798
mid: NIHMS1552684
pii:
doi:

Substances chimiques

Nerve Tissue Proteins 0
Unc13a protein, mouse 0
Ethanol 3K9958V90M

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

89-97

Subventions

Organisme : NIAAA NIH HHS
ID : R01 AA022414
Pays : United States

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

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Auteurs

Jessica I Wooden (JI)

Department of Psychology, University of Houston, Houston, TX 77204, United States.

Kyle Schuller (K)

Department of Psychology, University of Houston, Houston, TX 77204, United States.

Gregg Roman (G)

Department of Biology, University of Mississippi, Oxford, MS 38677, United States.

Joydip Das (J)

Department of Pharmacological and Pharmaceutical Sciences, College of Pharmacy, University of Houston, Houston, TX 77204, United States.

J Leigh Leasure (JL)

Department of Psychology, University of Houston, Houston, TX 77204, United States; Department of Biology & Biochemistry, University of Houston, Houston, TX 77204, United States. Electronic address: jlleasure@uh.edu.

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Classifications MeSH