Family history of prostate cancer and the incidence of ERG- and phosphatase and tensin homolog-defined prostate cancer.


Journal

International journal of cancer
ISSN: 1097-0215
Titre abrégé: Int J Cancer
Pays: United States
ID NLM: 0042124

Informations de publication

Date de publication:
15 05 2020
Historique:
received: 27 01 2019
revised: 18 06 2019
accepted: 28 06 2019
pubmed: 19 7 2019
medline: 23 9 2020
entrez: 19 7 2019
Statut: ppublish

Résumé

Family history is among the strongest known risk factors for prostate cancer (PCa). Emerging data suggest molecular subtypes of PCa, including two somatic genetic aberrations: fusions of androgen-regulated promoters with ERG and, separately, phosphatase and tensin homolog (PTEN) loss. We examined associations between family history and incidence of these subtypes in 44,126 men from the prospective Health Professionals Follow-up Study. ERG and PTEN status were assessed by immunohistochemistry. Multivariable competing risks models were used to estimate hazard ratios (HR) and 95% confidence intervals (CI) for associations between self-reported family history of PCa and molecular subtypes of disease. Thirteen percent of men had a positive family history of PCa at baseline. During a median follow-up of 18.5 years, 5,511 PCa cases were diagnosed. Among them, 888 were assayed for ERG status (47% ERG-positive) and 715 were assayed for PTEN loss (14% PTEN null). Family history was more strongly associated with risk of ERG-negative (HR: 2.15; 95% CI: 1.71-2.70) than ERG-positive (HR: 1.49; 95% CI: 1.13-1.95) disease (p

Identifiants

pubmed: 31318977
doi: 10.1002/ijc.32577
pmc: PMC7905843
mid: NIHMS1669003
doi:

Substances chimiques

Biomarkers, Tumor 0
ERG protein, human 0
Transcriptional Regulator ERG 0
PTEN Phosphohydrolase EC 3.1.3.67
PTEN protein, human EC 3.1.3.67

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

2694-2702

Subventions

Organisme : NCI NIH HHS
ID : P50 CA090381
Pays : United States
Organisme : NCI NIH HHS
ID : U01 CA167552
Pays : United States
Organisme : NIH HHS
ID : P50 CA090381
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NIH HHS
ID : U01 CA167552
Pays : United States
Organisme : NIH HHS
ID : R25 CA112355
Pays : United States
Organisme : NCI NIH HHS
ID : U01 CA113913
Pays : United States
Organisme : NCI NIH HHS
ID : T32 CA009001
Pays : United States
Organisme : NIH HHS
ID : R01 CA141298
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA141298
Pays : United States
Organisme : NIH HHS
ID : R01 CA136578
Pays : United States
Organisme : NCI NIH HHS
ID : R25 CA112355
Pays : United States
Organisme : NIH HHS
ID : T32 CA09001
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA136578
Pays : United States

Informations de copyright

© 2019 UICC.

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Auteurs

Dana Hashim (D)

Icahn School of Medicine at Mount Sinai, Tisch Cancer Institute, New York, NY.

Amparo G Gonzalez-Feliciano (AG)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.

Thomas U Ahearn (TU)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD.

Andreas Pettersson (A)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Clinical Epidemiology Unit, Department of Medicine Solna, Karolinska Institutet, Stockholm, Sweden.

Lauren Barber (L)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.

Claire H Pernar (CH)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.

Ericka M Ebot (EM)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.

Masis Isikbay (M)

Department of Surgery, University of California, San Francisco, San Francisco, CA.

Stephen P Finn (SP)

Department of Histopathology, St. James's Hospital and Trinity College Dublin Medical School, Dublin, Ireland.

Edward L Giovannucci (EL)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Department of Nutrition, Harvard T.H. Chan School of Public Health, Boston, MA.
Channing Division of Network Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA.

Rosina T Lis (RT)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA.

Massimo Loda (M)

Channing Division of Network Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA.
Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA.

Giovanni Parmigiani (G)

Department of Biostatistics, Harvard T.H. Chan School of Public Health, Boston, MA.
Department of Biostatistics and Computational Biology, Dana-Farber Cancer Institute, Boston, MA.

Tamara Lotan (T)

Department of Pathology, Johns Hopkins Bayview Medical Center, Baltimore, MD.

Philip W Kantoff (PW)

Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY.

Lorelei A Mucci (LA)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Channing Division of Network Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA.

Rebecca E Graff (RE)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA.
Department of Epidemiology and Biostatistics, University of California, San Francisco, San Francisco, CA.

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Classifications MeSH