The Impact of Coronary Artery Disease and Statins on Survival After Liver Transplantation.


Journal

Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society
ISSN: 1527-6473
Titre abrégé: Liver Transpl
Pays: United States
ID NLM: 100909185

Informations de publication

Date de publication:
10 2019
Historique:
received: 12 05 2019
accepted: 03 07 2019
pubmed: 26 7 2019
medline: 12 9 2020
entrez: 26 7 2019
Statut: ppublish

Résumé

Cardiovascular disease (CVD) is a major contributor to longterm mortality after liver transplantation (LT) necessitating aggressive modification of CVD risk. However, it is unclear how coronary artery disease (CAD) and the development of dyslipidemia following LT impacts clinical outcomes and how management of these factors may impact survival. Patients undergoing LT at Virginia Commonwealth University from January 2007 to January 2017 were included (n = 495). CAD and risk factors in all potential liver transplantation recipients (LTRs) over the age of 50 years were evaluated via coronary angiography. The impact of pre-LT CAD after transplantation was evaluated via a survival analysis. Additionally, factors associated with new-onset dyslipidemia, statin use, and mortality were assessed using multiple logistic regression or Cox proportional hazards models. The mean age of the cohort was 55.3 ± 9.3 years at the time of LT, and median follow-up was 4.5 years. CAD was noted in 129 (26.1%) patients during the pre-LT evaluation. The presence or severity of pre-LT CAD did not impact post-LT survival. Dyslipidemia was present in 96 patients at LT, and 157 patients developed new-onset dyslipidemia after LT. Statins were underused as only 45.7% of patients with known CAD were on therapy. In patients with new-onset dyslipidemia, statin therapy was initiated in 111 (71.1%), and median time to initiation of statin therapy was 2.5 years. Statin use conferred survival benefit (hazard ratio, 0.25; 95% confidence interval, 0.12-0.49) and was well tolerated with only 12% of patients developing an adverse event requiring the cessation of therapy. In conclusion, pre-LT CAD did not impact survival after LT, potentially suggesting a role of accelerated atherosclerosis that may not be captured on pre-LT testing. Although statin therapy confers survival benefit, it is underused in LTRs.

Identifiants

pubmed: 31344758
doi: 10.1002/lt.25613
pmc: PMC6754286
mid: NIHMS1043471
doi:

Substances chimiques

Hydroxymethylglutaryl-CoA Reductase Inhibitors 0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1514-1523

Subventions

Organisme : NCATS NIH HHS
ID : UL1 TR002649
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 by the American Association for the Study of Liver Diseases.

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Auteurs

Samarth S Patel (SS)

Division of Gastroenterology, Hepatology and Nutrition, Virginia Commonwealth University, Richmond, VA.

Viviana A Rodriguez (VA)

Departments of Biostatistics, Virginia Commonwealth University, Richmond, VA.

Mohammad B Siddiqui (MB)

Division of Gastroenterology, Hepatology and Nutrition, Virginia Commonwealth University, Richmond, VA.

Masoud Faridnia (M)

Departments of Internal Medicine, Virginia Commonwealth University, Richmond, VA.

Fei-Pi Lin (FP)

School of Medicine, Virginia Commonwealth University, Richmond, VA.

Anchalia Chandrakumaran (A)

Departments of Internal Medicine, Virginia Commonwealth University, Richmond, VA.

John Laurenzano (J)

School of Medicine, Virginia Commonwealth University, Richmond, VA.

Joseph Clinton (J)

School of Medicine, Virginia Commonwealth University, Richmond, VA.

Gurukripa N Kowlgi (GN)

Division of Cardiology, Department of Internal Medicine, Virginia Commonwealth University, Richmond, VA.

Danielle Kirkman (D)

Department of Kinesiology and Health Sciences, Virginia Commonwealth University, Richmond, VA.

Adam P Sima (AP)

Departments of Biostatistics, Virginia Commonwealth University, Richmond, VA.

Erika Liptrap (E)

School of Medicine, Virginia Commonwealth University, Richmond, VA.

Chandra Bhati (C)

Division of Transplant Surgery, Department of Surgery, Virginia Commonwealth University, Richmond, VA.

Mohammad Shadab Siddiqui (MS)

Division of Gastroenterology, Hepatology and Nutrition, Virginia Commonwealth University, Richmond, VA.

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