Diffusing Capacity of Carbon Monoxide in Assessment of COPD.


Journal

Chest
ISSN: 1931-3543
Titre abrégé: Chest
Pays: United States
ID NLM: 0231335

Informations de publication

Date de publication:
12 2019
Historique:
received: 13 03 2019
revised: 10 06 2019
accepted: 14 06 2019
pubmed: 29 7 2019
medline: 29 5 2020
entrez: 29 7 2019
Statut: ppublish

Résumé

Diffusing capacity of the lung for carbon monoxide (Dlco) is inconsistently obtained in patients with COPD, and the added benefit of Dlco testing beyond that of more common tools is unknown. The goal of this study was to determine whether lower Dlco is associated with increased COPD morbidity independent of emphysema assessed via spirometry and CT imaging. Data for 1,806 participants with COPD from the Genetic Epidemiology of COPD (COPDGene) study 5-year visit were analyzed, including pulmonary function testing, quality of life, symptoms, exercise performance, and exacerbation rates. Dlco percent predicted was primarily analyzed as a continuous variable and additionally categorized into four groups: (1) Dlco and FEV In multivariable analyses, every 10% predicted decrease in Dlco was associated with symptoms and quality of life (COPD Assessment Test, 0.53 [P < .001]; St. George's Respiratory Questionnaire, 1.67 [P < .001]; Medical Outcomes Study Short Form 36 Physical Function, -0.89 [P < .001]), exercise performance (6-min walk distance, -45.35 feet; P < .001), and severe exacerbation rate (rate ratio, 1.14; P < .001). When categorized, severe impairment in Dlco alone, FEV Impairment in Dlco was associated with increased COPD symptoms, reduced exercise performance, and severe exacerbation risk even after accounting for spirometry and CT evidence of emphysema. These findings suggest that Dlco should be considered for inclusion in future multidimensional tools assessing COPD.

Sections du résumé

BACKGROUND
Diffusing capacity of the lung for carbon monoxide (Dlco) is inconsistently obtained in patients with COPD, and the added benefit of Dlco testing beyond that of more common tools is unknown.
OBJECTIVE
The goal of this study was to determine whether lower Dlco is associated with increased COPD morbidity independent of emphysema assessed via spirometry and CT imaging.
METHODS
Data for 1,806 participants with COPD from the Genetic Epidemiology of COPD (COPDGene) study 5-year visit were analyzed, including pulmonary function testing, quality of life, symptoms, exercise performance, and exacerbation rates. Dlco percent predicted was primarily analyzed as a continuous variable and additionally categorized into four groups: (1) Dlco and FEV
RESULTS
In multivariable analyses, every 10% predicted decrease in Dlco was associated with symptoms and quality of life (COPD Assessment Test, 0.53 [P < .001]; St. George's Respiratory Questionnaire, 1.67 [P < .001]; Medical Outcomes Study Short Form 36 Physical Function, -0.89 [P < .001]), exercise performance (6-min walk distance, -45.35 feet; P < .001), and severe exacerbation rate (rate ratio, 1.14; P < .001). When categorized, severe impairment in Dlco alone, FEV
CONCLUSIONS
Impairment in Dlco was associated with increased COPD symptoms, reduced exercise performance, and severe exacerbation risk even after accounting for spirometry and CT evidence of emphysema. These findings suggest that Dlco should be considered for inclusion in future multidimensional tools assessing COPD.

Identifiants

pubmed: 31352035
pii: S0012-3692(19)31377-7
doi: 10.1016/j.chest.2019.06.035
pmc: PMC7242635
pii:
doi:

Substances chimiques

Carbon Monoxide 7U1EE4V452

Banques de données

ClinicalTrials.gov
['NCT00608764']

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1111-1119

Subventions

Organisme : NHLBI NIH HHS
ID : T32 HL007534
Pays : United States
Organisme : NHLBI NIH HHS
ID : U01 HL089856
Pays : United States
Organisme : NHLBI NIH HHS
ID : U01 HL089897
Pays : United States
Organisme : NHLBI NIH HHS
ID : T32 HL007534-36
Pays : United States

Informations de copyright

Copyright © 2019. Published by Elsevier Inc.

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Auteurs

Aparna Balasubramanian (A)

Division of Pulmonary and Critical Care Medicine, Johns Hopkins University, Baltimore, MD.

Neil R MacIntyre (NR)

Division of Pulmonary and Critical Care Medicine, Duke University, Durham, NC.

Robert J Henderson (RJ)

Division of Pulmonary and Critical Care Medicine, Johns Hopkins University, Baltimore, MD.

Robert L Jensen (RL)

Division of Pulmonary and Critical Care Medicine, University of Utah, Salt Lake City, UT.

Gregory Kinney (G)

Department of Epidemiology, Colorado School of Public Health, University of Colorado, Denver, CO.

William W Stringer (WW)

Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center, Torrance, CA.

Craig P Hersh (CP)

Channing Division of Network Medicine, Brigham and Women's Hospital, Boston, MA.

Russell P Bowler (RP)

Division of Pulmonary, Critical Care and Sleep Medicine, National Jewish Health, Denver, CO.

Richard Casaburi (R)

Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center, Torrance, CA.

MeiLan K Han (MK)

Division of Pulmonary and Critical Care Medicine, University of Michigan, Ann Arbor, MI.

Janos Porszasz (J)

Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center, Torrance, CA.

R Graham Barr (RG)

Department of Epidemiology, Columbia University, New York, NY.

Barry J Make (BJ)

Division of Pulmonary, Critical Care and Sleep Medicine, National Jewish Health, Denver, CO.

Robert A Wise (RA)

Division of Pulmonary and Critical Care Medicine, Johns Hopkins University, Baltimore, MD.

Meredith C McCormack (MC)

Division of Pulmonary and Critical Care Medicine, Johns Hopkins University, Baltimore, MD. Electronic address: mmccor16@jhmi.edu.

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