Glucose stimulates somatostatin secretion in pancreatic δ-cells by cAMP-dependent intracellular Ca


Journal

The Journal of general physiology
ISSN: 1540-7748
Titre abrégé: J Gen Physiol
Pays: United States
ID NLM: 2985110R

Informations de publication

Date de publication:
02 09 2019
Historique:
received: 18 02 2019
revised: 11 05 2019
accepted: 09 07 2019
pubmed: 31 7 2019
medline: 5 9 2020
entrez: 31 7 2019
Statut: ppublish

Résumé

Somatostatin secretion from pancreatic islet δ-cells is stimulated by elevated glucose levels, but the underlying mechanisms have only partially been elucidated. Here we show that glucose-induced somatostatin secretion (GISS) involves both membrane potential-dependent and -independent pathways. Although glucose-induced electrical activity triggers somatostatin release, the sugar also stimulates GISS via a cAMP-dependent stimulation of CICR and exocytosis of somatostatin. The latter effect is more quantitatively important and in mouse islets depolarized by 70 mM extracellular K

Identifiants

pubmed: 31358556
pii: jgp.201912351
doi: 10.1085/jgp.201912351
pmc: PMC6719402
doi:

Substances chimiques

Adjuvants, Immunologic 0
Guanine Nucleotide Exchange Factors 0
Rapgef4 protein, mouse 0
Colforsin 1F7A44V6OU
Somatostatin 51110-01-1
Thapsigargin 67526-95-8
Cyclic AMP E0399OZS9N
Glucose IY9XDZ35W2
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1094-1115

Subventions

Organisme : Medical Research Council
ID : MC_UU_00014/3
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_UU_00014/5
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_UU_12012/3
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 095531/Z/11/Z
Pays : United Kingdom

Informations de copyright

© 2019 Denwood et al.

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Auteurs

Geoffrey Denwood (G)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK.

Andrei Tarasov (A)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK.
School of Life and Medical Sciences, University of Hertfordshire, Hatfield, UK.

Albert Salehi (A)

Institute of Neuroscience and Physiology, Department of Physiology, Metabolic Research Unit, University of Goteborg, Göteborg, Sweden.

Elisa Vergari (E)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK.

Reshma Ramracheya (R)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK.

Harumi Takahashi (H)

Division of Molecular and Metabolic Medicine, Kobe University Graduate School of Medicine, Kobe, Japan.

Viacheslav O Nikolaev (VO)

Institute of Experimental Cardiovascular Research, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Susumo Seino (S)

Division of Molecular and Metabolic Medicine, Kobe University Graduate School of Medicine, Kobe, Japan.

Fiona Gribble (F)

Institute of Metabolic Science, University of Cambridge, Addenbrook's Hospital, Cambridge, UK.

Frank Reimann (F)

Institute of Metabolic Science, University of Cambridge, Addenbrook's Hospital, Cambridge, UK.

Patrik Rorsman (P)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK.
Institute of Neuroscience and Physiology, Department of Physiology, Metabolic Research Unit, University of Goteborg, Göteborg, Sweden.

Quan Zhang (Q)

Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK quan.zhang@ocdem.ox.ac.uk.

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