Passive leg-lifting in heart failure patients predicts exercise-induced rise in left ventricular filling pressures.


Journal

Clinical research in cardiology : official journal of the German Cardiac Society
ISSN: 1861-0692
Titre abrégé: Clin Res Cardiol
Pays: Germany
ID NLM: 101264123

Informations de publication

Date de publication:
Apr 2020
Historique:
received: 22 03 2019
accepted: 18 07 2019
pubmed: 2 8 2019
medline: 29 12 2020
entrez: 2 8 2019
Statut: ppublish

Résumé

The aim of this study was to assess PCWP with passive leg-lifting (PLL) and exercise, in two groups of patients presenting with normal left ventricular ejection fraction (LVEF); one group with elevated NT-proBNP (eBNP), and one with normal NT-proBNP (nBNP) plasma concentration. Fifty-one patients with eBNP (NT-proBNP ≥ 125 ng/l) and LVEF > 50%, were investigated and compared with 34 patients with nBNP (NT-proBNP < 125 ng/l) and LVEF > 50%. Both groups underwent right heart catheterization (RHC) at rest, PLL and exercise. From RHC, mean pulmonary arterial pressure (mPAP), cardiac output (CO), and PCWP were measured. All nBNP patients had PCWP < 15 mmHg at rest, and a PCWP of < 25 mmHg with PLL and during exercise. Patients with eBNP had higher (p < 0.01) resting mPAP, PCWP, and mPAP/CO. These values increased with exercise; however, CO increased less in comparison with nBNP patients (p = 0.001). 20% of patients with eBNP had a PCWP > 15 mmHg at rest, this percentage increased to 47% with PLL and 41% had a PCWP > 25 mmHg during exercise. Of those with PCWP > 25 mmHg during exercise, 91% had a PCWP > 15 mmHg with PLL. A PCWP > 15 mmHg on PLL had a 91% sensitivity and 92% specificity in predicting exercise-induced PCWP of > 25 mmHg. In patients presenting with eBNP, PLL can predict which patients will develop elevated PCWP with exercise. These findings highlight the role of stress assessment.

Identifiants

pubmed: 31368000
doi: 10.1007/s00392-019-01531-w
pii: 10.1007/s00392-019-01531-w
pmc: PMC7098926
doi:

Substances chimiques

Biomarkers 0
Peptide Fragments 0
pro-brain natriuretic peptide (1-76) 0
Natriuretic Peptide, Brain 114471-18-0

Types de publication

Comparative Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

498-507

Références

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Auteurs

E Tossavainen (E)

Department of Public Health and Clinical Medicine, Cardiology, Umeå University, S-90185, Umeå, Sweden. Erik.tossavainen@vll.se.

G Wikström (G)

Department of Medical Sciences,Cardiology, Uppsala University, Uppsala, Sweden.

M Y Henein (MY)

Department of Public Health and Clinical Medicine, Cardiology, Umeå University, S-90185, Umeå, Sweden.

M Lundqvist (M)

Department of Medical Sciences,Cardiology, Uppsala University, Uppsala, Sweden.

U Wiklund (U)

Department of Radiation Sciences, Biomedical Engineering, Umeå University, Umeå, Sweden.

P Lindqvist (P)

Department of Surgical and Perioperative Sciences, Clinical Physiology, Umeå University, Umeå, Sweden.

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