Taste loss with obesity in mice and men.


Journal

International journal of obesity (2005)
ISSN: 1476-5497
Titre abrégé: Int J Obes (Lond)
Pays: England
ID NLM: 101256108

Informations de publication

Date de publication:
03 2020
Historique:
received: 10 04 2019
accepted: 30 06 2019
revised: 24 05 2019
pubmed: 8 8 2019
medline: 11 5 2021
entrez: 8 8 2019
Statut: ppublish

Résumé

Our sense of taste is critical in defining our food choices and habits. Located primarily in our tongue, taste buds are small assemblies of constantly renewing sensory cells, tasked with evaluating oral stimuli before the food we eat is consumed. Using both mice and a free-living human population, we tracked taste papilla abundancy with weight gain, to test for deficiencies in the taste system of obese mice and humans with increased adiposity. Mice fed a high-fat diet for 8 weeks expressed markers for all subtypes of taste cells at a lower level than chow-fed counterparts. This came alongside the loss of markers for taste cell proliferation (Ki-67) and development (β-catenin), as well as lower fungiform papillae density, consistent with earlier results showing lower circumvallate taste bud abundance in obese mice. Likewise, in a population of college students tracked through 4 years of college attendance, the change in density of fungiform papillae, which house taste buds in the anterior tongue, was negatively correlated with change in neck circumference, a marker of adiposity. These results highlight changes in taste during weight gain as a potentially important consideration in the study of obesity.

Sections du résumé

BACKGROUND
Our sense of taste is critical in defining our food choices and habits. Located primarily in our tongue, taste buds are small assemblies of constantly renewing sensory cells, tasked with evaluating oral stimuli before the food we eat is consumed.
METHODS
Using both mice and a free-living human population, we tracked taste papilla abundancy with weight gain, to test for deficiencies in the taste system of obese mice and humans with increased adiposity.
RESULTS
Mice fed a high-fat diet for 8 weeks expressed markers for all subtypes of taste cells at a lower level than chow-fed counterparts. This came alongside the loss of markers for taste cell proliferation (Ki-67) and development (β-catenin), as well as lower fungiform papillae density, consistent with earlier results showing lower circumvallate taste bud abundance in obese mice. Likewise, in a population of college students tracked through 4 years of college attendance, the change in density of fungiform papillae, which house taste buds in the anterior tongue, was negatively correlated with change in neck circumference, a marker of adiposity.
CONCLUSIONS
These results highlight changes in taste during weight gain as a potentially important consideration in the study of obesity.

Identifiants

pubmed: 31388094
doi: 10.1038/s41366-019-0429-6
pii: 10.1038/s41366-019-0429-6
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

739-743

Commentaires et corrections

Type : ErratumIn

Références

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Auteurs

Andrew Kaufman (A)

Department of Food Science, Cornell University, Ithaca, NY, 14853, USA.

Jennie Kim (J)

College of Human Ecology, Cornell University, Ithaca, NY, 14853, USA.

Corinna Noel (C)

Department of Food Science, Cornell University, Ithaca, NY, 14853, USA.

Robin Dando (R)

Department of Food Science, Cornell University, Ithaca, NY, 14853, USA. robin.dando@cornell.edu.

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