Reduced melatonin synthesis in pregnant night workers: Metabolic implications for offspring.


Journal

Medical hypotheses
ISSN: 1532-2777
Titre abrégé: Med Hypotheses
Pays: United States
ID NLM: 7505668

Informations de publication

Date de publication:
Nov 2019
Historique:
received: 07 10 2018
revised: 01 08 2019
accepted: 05 08 2019
pubmed: 20 8 2019
medline: 2 5 2020
entrez: 18 8 2019
Statut: ppublish

Résumé

Several novel animal studies have shown that intrauterine metabolic programming can be modified in the event of reduced melatonin synthesis during pregnancy, leading to glucose intolerance and insulin resistance in the offspring. It is therefore postulated that female night workers when pregnant may expose the offspring to unwanted health threats. This may be explained by the fact that melatonin is essential for regulating energy metabolism and can influence reproductive activity. Moreover, the circadian misalignment caused by shift work affects fertility and the fetus, increasing the risk of miscarriage, premature birth and low birth weight, phenomena observed in night workers. Thus, we hypothesize that light-induced melatonin suppression as a result of night work may alter intrauterine metabolic programming in pregnant women, potentially leading to metabolic disorders in their offspring.

Identifiants

pubmed: 31421432
pii: S0306-9877(18)31050-8
doi: 10.1016/j.mehy.2019.109353
pii:
doi:

Substances chimiques

Melatonin JL5DK93RCL

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

109353

Subventions

Organisme : Medical Research Council
ID : MR/N006321/1
Pays : United Kingdom

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

P A Nehme (PA)

School of Public Health, University of São Paulo, Brazil.

F Amaral (F)

Department of Physiology, Federal University of São Paulo, Brazil.

A Lowden (A)

Stress Research Institute, University of Stockholm, Sweden.

D J Skene (DJ)

Faculty of Health and Medical Sciences, University of Surrey, UK.

J Cipolla-Neto (J)

Department of Physiology and Biophysics Neurobiology Lab, Institute of Biomedical Sciences, University of São Paulo, Brazil.

C R C Moreno (CRC)

School of Public Health, University of São Paulo, Brazil; Stress Research Institute, University of Stockholm, Sweden. Electronic address: crmoreno@usp.br.

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Classifications MeSH