Ticagrelor versus placebo for the reduction of vaso-occlusive crises in pediatric sickle cell disease: Rationale and design of a randomized, double-blind, parallel-group, multicenter phase 3 study (HESTIA3).


Journal

Contemporary clinical trials
ISSN: 1559-2030
Titre abrégé: Contemp Clin Trials
Pays: United States
ID NLM: 101242342

Informations de publication

Date de publication:
10 2019
Historique:
received: 15 03 2019
revised: 19 08 2019
accepted: 20 08 2019
pubmed: 26 8 2019
medline: 22 10 2020
entrez: 26 8 2019
Statut: ppublish

Résumé

An unmet need for therapies exists to reduce sickle cell disease (SCD) complications in pediatric patients. Activated platelets contribute to the formation of cellular aggregates during sickling and vaso-occlusive crises (VOCs). Ticagrelor is an oral, direct-acting, and reversible adenosine diphosphate P2Y Approximately 180 patients (aged ≥ 2 to <18 years) with SCD (≥ 2 VOCs in the prior year) from 18 countries will be randomized 1:1 to ticagrelor or placebo. Primary endpoint: number of VOCs (a composite endpoint of painful crises and/or acute chest syndrome); key secondary endpoints: hospitalizations, pain intensity and analgesic use during VOCs, acceptability of formulation, and health-related quality of life. The weight-based doses of ticagrelor are set by modeling and simulation. Platelet inhibition data, measured by the vasodilator-stimulated phosphoprotein assay, will be collected for exploratory purposes. HESTIA3 aims to demonstrate that using greater target platelet inhibition than previous studies on SCD, ticagrelor will decrease the frequency of VOC in pediatric patients. Trial Identifier: NCT03615924; EudraCT2017-002421-38.

Sections du résumé

BACKGROUND
An unmet need for therapies exists to reduce sickle cell disease (SCD) complications in pediatric patients. Activated platelets contribute to the formation of cellular aggregates during sickling and vaso-occlusive crises (VOCs). Ticagrelor is an oral, direct-acting, and reversible adenosine diphosphate P2Y
METHODS
Approximately 180 patients (aged ≥ 2 to <18 years) with SCD (≥ 2 VOCs in the prior year) from 18 countries will be randomized 1:1 to ticagrelor or placebo. Primary endpoint: number of VOCs (a composite endpoint of painful crises and/or acute chest syndrome); key secondary endpoints: hospitalizations, pain intensity and analgesic use during VOCs, acceptability of formulation, and health-related quality of life. The weight-based doses of ticagrelor are set by modeling and simulation. Platelet inhibition data, measured by the vasodilator-stimulated phosphoprotein assay, will be collected for exploratory purposes.
CONCLUSIONS
HESTIA3 aims to demonstrate that using greater target platelet inhibition than previous studies on SCD, ticagrelor will decrease the frequency of VOC in pediatric patients. Trial Identifier: NCT03615924; EudraCT2017-002421-38.

Identifiants

pubmed: 31446143
pii: S1551-7144(19)30550-6
doi: 10.1016/j.cct.2019.105835
pii:
doi:

Substances chimiques

Platelet Aggregation Inhibitors 0
Ticagrelor GLH0314RVC

Banques de données

ClinicalTrials.gov
['NCT03615924']
EudraCT
['2017-002421-38']

Types de publication

Clinical Trial Protocol Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

105835

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Matthew M Heeney (MM)

Division of Hematology/Oncology, Boston Children's Hospital, Harvard Medical School, 300 Longwood Ave, Boston, MA 02115, USA. Electronic address: matthew.heeney@childrens.harvard.edu.

Miguel R Abboud (MR)

American University of Beirut Medical Center, Cairo Street, Beirut, Lebanon. Electronic address: ma56@aub.edu.lb.

Carl Amilon (C)

BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Pepparedsleden 1, Mölndal 431 83, Sweden. Electronic address: carl.amilon@astrazeneca.com.

Marielle Andersson (M)

BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Pepparedsleden 1, Mölndal 431 83, Sweden. Electronic address: Marielle.Andersson@astrazeneca.com.

Jessie Githanga (J)

Hematology and Blood Transfusion Unit, Department of Human Pathology, University of Nairobi, P. O. Box 19676, Nairobi 00202, Kenya. Electronic address: jessie.githanga@uonbi.ac.ke.

Baba Inusa (B)

Paediatric Hematology, Guy's and St Thomas' NHS Trust, Evelina London Children's Hospital, Westminster Bridge Rd, Lambeth, London SE1 7EH, UK. Electronic address: Baba.Inusa@gstt.nhs.uk.

Julie Kanter (J)

Hematology-Oncology, Department of Medicine, University of Alabama at Birmingham, 2000 6th Avenue S, Birmingham, AL 35233, USA. Electronic address: kanter@musc.edu.

Maria Leonsson-Zachrisson (M)

BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Pepparedsleden 1, Mölndal 431 83, Sweden. Electronic address: Maria.Leonsson-Zachrisson@astrazeneca.com.

Alan D Michelson (AD)

Center for Platelet Research Studies, Division of Hematology/Oncology, Boston Children's Hospital, Harvard Medical School, 300 Longwood Avenue, Karp 08213, Boston, MA 02115, USA. Electronic address: Alan.Michelson@childrens.harvard.edu.

Anders R Berggren (AR)

BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Pepparedsleden 1, Mölndal 431 83, Sweden. Electronic address: anders.r.berggren@astrazeneca.com.

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