GDF-15 in solid vs non-solid treatment-naïve malignancies.


Journal

European journal of clinical investigation
ISSN: 1365-2362
Titre abrégé: Eur J Clin Invest
Pays: England
ID NLM: 0245331

Informations de publication

Date de publication:
Nov 2019
Historique:
received: 07 10 2018
revised: 30 07 2019
accepted: 25 08 2019
pubmed: 30 8 2019
medline: 10 6 2020
entrez: 30 8 2019
Statut: ppublish

Résumé

GDF-15 is an established cardiovascular risk marker but is equally implicated in tumour biology. Elevated levels of GDF-15 have indeed been observed in distinct tumour entities. This study aimed to explore the relation of GDF-15 to other cardiac biomarkers and the general association of GDF-15 on prognosis in an unselected cohort of treatment-naïve cancer patients. We prospectively enrolled 555 consecutive patients at time of diagnosis of malignant disease prior receiving anticancer therapy. Plasma GDF-15 concentrations were determined alongside other cardiac and routine laboratory markers. All-cause mortality was defined as primary endpoint. GDF-15 levels were 338 ng/L (IQR:205-534) for the total cohort, and values were comparable for different tumour entities except breast cancer. Metastatic disease was characterized by higher plasma GDF-15 [435 ng/L (IQR:279-614) vs 266 ng/L (IQR:175-427), P < .001]. GDF-15 correlated positively with inflammatory status reflected by CRP, SAA and IL-6 [r = .31, P < .001, r = .23, P < .001 and r = .14, P = .002] and cardiac biomarkers as NT-proBNP, hsTnT, MR-proADM and CT-proET-1 [r = .46; r = .46; r = .59 and r = .50; P < .001 for all]. GDF-15 was significantly associated with all-cause mortality after multivariate adjustment [adj.HR for ln(GDF-15) 1.78, 95%CI:1.47-2.16, P < .001]. There was a significant interaction between solid and haematological malignancies with loss of association of GDF-15 with outcome in myelodysplastic and myeloproliferative disease. Elevated plasma GDF-15 is associated with progressing disease severity and poor prognosis in solid tumours of treatment-naïve cancer patients. GDF-15 increase is accompanied by worsening systemic inflammation and a subclinical functional impairment of different organs including the heart. GDF-15 represents a promising target for our pathophysiologic understanding in cardio-oncology linking conditions of both cardiac and neoplastic disease.

Identifiants

pubmed: 31463975
doi: 10.1111/eci.13168
pmc: PMC6899906
doi:

Substances chimiques

C-terminal proendothelin-1 0
Endothelin-1 0
GDF15 protein, human 0
Glycopeptides 0
Growth Differentiation Factor 15 0
IL6 protein, human 0
Interleukin-6 0
Peptide Fragments 0
Protein Precursors 0
Serum Amyloid A Protein 0
Troponin T 0
copeptins 0
mid-regional pro-adrenomedullin, human 0
pro-brain natriuretic peptide (1-76) 0
Natriuretic Peptide, Brain 114471-18-0
Adrenomedullin 148498-78-6
C-Reactive Protein 9007-41-4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13168

Subventions

Organisme : Thermo Fisher Scientific
ID : This study was supported by an unrestricted grant

Informations de copyright

© 2019 The Authors. European Journal of Clinical Investigation published by John Wiley & Sons Ltd on behalf of Stichting European Society for Clinical Investigation Journal Foundation.

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Auteurs

Henrike Arfsten (H)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Anna Cho (A)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Claudia Freitag (C)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Markus Raderer (M)

Department of Internal Medicine I, Division of Oncology and Hematology, Medical University of Vienna, Vienna, Austria.

Georg Goliasch (G)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Philipp E Bartko (PE)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Raphael Wurm (R)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Guido Strunk (G)

Complexity Research, Vienna, Austria.
FH Campus Vienna, Vienna, Austria.
Technical University Dortmund, Dortmund, Germany.

Heinz Gisslinger (H)

Department of Internal Medicine I, Division of Oncology and Hematology, Medical University of Vienna, Vienna, Austria.

Christine Marosi (C)

Department of Internal Medicine I, Division of Oncology and Hematology, Medical University of Vienna, Vienna, Austria.

Gabriela Kornek (G)

Department of Internal Medicine I, Division of Oncology and Hematology, Medical University of Vienna, Vienna, Austria.

Christoph Zielinski (C)

Department of Internal Medicine I, Division of Oncology and Hematology, Medical University of Vienna, Vienna, Austria.

Martin Hülsmann (M)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Noemi Pavo (N)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

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Classifications MeSH