Deciphering the BSE-type specific cell and tissue tropisms of atypical (H and L) and classical BSE.
Atypical BSE
PrP
cattle
central nervous system
glial cells
neurons
origin of BSE
peripheral nervous system
Journal
Prion
ISSN: 1933-690X
Titre abrégé: Prion
Pays: United States
ID NLM: 101472305
Informations de publication
Date de publication:
01 2019
01 2019
Historique:
entrez:
4
9
2019
pubmed:
4
9
2019
medline:
30
4
2020
Statut:
ppublish
Résumé
After the discovery of two atypical bovine spongiform encephalopathy (BSE) forms in France and Italy designated H- and L-BSE, the question arose whether these new forms differed from classical BSE (C-BSE) in their pathogenesis. Samples collected from cattle in the clinical stage of BSE during an intracranial challenge study with L- and H-BSE were analysed using biochemical and histological methods as well as in a transgenic mouse bioassay. Our results generally confirmed what had been described for C-BSE to be true also for both atypical BSE forms, namely the restriction of the pathological prion protein (PrP
Identifiants
pubmed: 31476957
doi: 10.1080/19336896.2019.1651180
pmc: PMC6746549
doi:
Substances chimiques
PrPSc Proteins
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
160-172Références
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