Human cardiac myosin light chain 4 (MYL4) mosaic expression patterns vary by sex.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
03 09 2019
Historique:
received: 16 05 2019
accepted: 21 08 2019
entrez: 5 9 2019
pubmed: 5 9 2019
medline: 30 10 2020
Statut: epublish

Résumé

Sex disparities modulate cardiac function, although the proteins and mechanisms remain to be elucidated. We recently demonstrated a mosaic pattern of protein expression in the heart for over 100 proteins. Here we investigate one of these proteins, myosin light chain 4 (MYL4), which is important for contractile functions by increasing force production. We assayed the expression pattern of MYL4 across 756 ventricular myocardial samples from 668 individuals utilizing a semi-automated Cell Profiler method on five tissue microarrays (TMAs) of cardiac tissues across a diverse set of diseases. The percentage of MYL4 positive cells was significantly higher in male subjects independently across all five TMAs, regardless of disease state (p = 8.66e-15). Higher MYL4 expression was also modestly associated with hypertrophic cardiomyopathy (p = 6.3e-04). MYL4 expression did not associate with sudden cardiac death or other cardiomyopathies. This study demonstrates a new mosaic pattern of protein expression that underlies sex disparities in the human heart.

Identifiants

pubmed: 31481666
doi: 10.1038/s41598-019-49191-0
pii: 10.1038/s41598-019-49191-0
pmc: PMC6722118
doi:

Substances chimiques

MYL4 protein, human 0
Myosin Light Chains 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

12681

Subventions

Organisme : NIGMS NIH HHS
ID : T32 GM007814
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA006973
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM130564
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL137811
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL131573
Pays : United States

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Auteurs

Tony Y Wang (TY)

Division of Cardiovascular Pathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Dan E Arking (DE)

McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Joseph J Maleszewski (JJ)

Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, USA.

Karen Fox-Talbot (K)

Division of Cardiovascular Pathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Tim O Nieuwenhuis (TO)

Division of Cardiovascular Pathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.
McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Lakshmi Santhanam (L)

Department of Anesthesiology and Critical Care Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Renu Virmani (R)

CVPath Institute, Gaithersburg, Maryland, USA.

Avi Z Rosenberg (AZ)

Division of Renal Pathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Marc K Halushka (MK)

Division of Cardiovascular Pathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD, USA. mhalush1@jhmi.edu.

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