Morphological analysis of ischemia-reperfusion injury in a cold ischemia model of jejunal free flap for hypopharyngeal reconstruction.


Journal

Journal of plastic, reconstructive & aesthetic surgery : JPRAS
ISSN: 1878-0539
Titre abrégé: J Plast Reconstr Aesthet Surg
Pays: Netherlands
ID NLM: 101264239

Informations de publication

Date de publication:
Jan 2020
Historique:
received: 05 08 2018
revised: 17 07 2019
accepted: 27 07 2019
pubmed: 9 9 2019
medline: 23 6 2020
entrez: 9 9 2019
Statut: ppublish

Résumé

Jejunal free flap (JFF) reconstruction is a popular treatment option for advanced hypopharyngeal cancer. Several factors including ischemia-reperfusion injury (IRI) can cause mucosal damage and progressive flap necrosis. We investigated the development and time-related progression of morphological and cellular changes in patients with JFF reconstruction including cold preservation of the graft. Eleven patients were enrolled. Biopsies were taken during surgery from normally perfused tissue, before loop isolation (T0), at the end of back-table surgery (T1), immediately before reperfusion (T2), 15' after reperfusion (T3), and at the end of the digestive anastomoses (T4) and from the external monitor daily from the 1st to the 5th postoperative day (M1-M5). Histomorphological and immunohistochemical parameters in the intraoperative and postoperative samples were evaluated and compared. Delayed flap necrosis was observed in 2 patients. The cold ischemia phase did not negatively affect mucosal regeneration after reperfusion; morphological and cellular damage parameters returned to normal by the end of surgery or along the early postoperative period. Significant enterocyte replication activity was observed at the end of revascularization, which continued in the postoperative phase, leading to recovery of the epithelial morphological integrity and disappearance of apoptotic cells. An inflammatory infiltrate persisted in the M samples, and in a significant proportion of samples, mucosal fibrosis developed by the end of the postoperative observation. Cold perfusion and preservation of the JFF can effectively limit the negative effects of IRI and to prevent short- and medium-term complications that can compromise the final outcome.

Sections du résumé

BACKGROUND BACKGROUND
Jejunal free flap (JFF) reconstruction is a popular treatment option for advanced hypopharyngeal cancer. Several factors including ischemia-reperfusion injury (IRI) can cause mucosal damage and progressive flap necrosis. We investigated the development and time-related progression of morphological and cellular changes in patients with JFF reconstruction including cold preservation of the graft.
METHODS METHODS
Eleven patients were enrolled. Biopsies were taken during surgery from normally perfused tissue, before loop isolation (T0), at the end of back-table surgery (T1), immediately before reperfusion (T2), 15' after reperfusion (T3), and at the end of the digestive anastomoses (T4) and from the external monitor daily from the 1st to the 5th postoperative day (M1-M5). Histomorphological and immunohistochemical parameters in the intraoperative and postoperative samples were evaluated and compared.
RESULTS RESULTS
Delayed flap necrosis was observed in 2 patients. The cold ischemia phase did not negatively affect mucosal regeneration after reperfusion; morphological and cellular damage parameters returned to normal by the end of surgery or along the early postoperative period. Significant enterocyte replication activity was observed at the end of revascularization, which continued in the postoperative phase, leading to recovery of the epithelial morphological integrity and disappearance of apoptotic cells. An inflammatory infiltrate persisted in the M samples, and in a significant proportion of samples, mucosal fibrosis developed by the end of the postoperative observation.
CONCLUSION CONCLUSIONS
Cold perfusion and preservation of the JFF can effectively limit the negative effects of IRI and to prevent short- and medium-term complications that can compromise the final outcome.

Identifiants

pubmed: 31494055
pii: S1748-6815(19)30337-7
doi: 10.1016/j.bjps.2019.07.004
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

103-110

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Simone Mauramati (S)

Department of Otorhinolaryngology, University of Pavia, Foundation IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy. Electronic address: s.mauramati@smatteo.pv.it.

Patrizia Morbini (P)

Department of Molecular Medicine, Unit of Pathology, University of Pavia, Foundation IRCCS Policlinico San Matteo, Via Forlanini 16, 27100 Pavia, Italy.

Giuseppina Ferrario (G)

Department of Molecular Medicine, Unit of Pathology, University of Pavia, Foundation IRCCS Policlinico San Matteo, Via Forlanini 16, 27100 Pavia, Italy.

Mohamed Alnemr (M)

Department of Otorhinolaryngology, Faculty of Medicine, Zagazig University, Zagazig, Sharkia, Egypt.

Elona Luka (E)

Service of Clinical Epidemiology & Biometry, Fondazione IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy.

Antonio Occhini (A)

Department of Otorhinolaryngology, University of Pavia, Foundation IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy.

Giulia Bertino (G)

Department of Otorhinolaryngology, University of Pavia, Foundation IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy.

Catherine Klersy (C)

Service of Clinical Epidemiology & Biometry, Fondazione IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy.

Mario Alessiani (M)

ASST of Pavia, University of Pavia, C.so di Strada Nuova 5, 27100 Pavia, Italy.

Marco Benazzo (M)

Department of Otorhinolaryngology, University of Pavia, Foundation IRCCS Policlinico San Matteo, P.le Golgi 5, 27100 Pavia, Italy.

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