Plasma tryptophan and kynurenine in females with temporomandibular disorders and fibromyalgia-An exploratory pilot study.

fibromyalgia kynurenine pathway pain psychological symptoms temporomandibular disorders tryptophan metabolism

Journal

Journal of oral rehabilitation
ISSN: 1365-2842
Titre abrégé: J Oral Rehabil
Pays: England
ID NLM: 0433604

Informations de publication

Date de publication:
Feb 2020
Historique:
received: 27 05 2019
revised: 30 08 2019
accepted: 11 09 2019
pubmed: 24 9 2019
medline: 21 1 2020
entrez: 24 9 2019
Statut: ppublish

Résumé

Both temporomandibular disorders myalgia (TMDM) and fibromyalgia (FM) have been linked to central and peripheral changes in serotonin availability. The precursor of serotonin, tryptophan (TRP), is mainly catabolised via another pathway to produce kynurenine (KYN), but whether changes of this pathway are present in TMDM and FM are still unclear. The aim was to explore blood plasma concentrations of TRP and KYN in TMDM and FM in an attempt to identify novel associations for future research. Plasma of 113 female participants (17 TMDM, 40 FM and 56 healthy pain-free controls) were analysed for TRP and KYN concentrations. The degradation of TRP via the KYN pathway was indicated by the KYN to TRP ratio (KYN/TRP). Pain intensities were assessed with the Graded Chronic Pain Scale (GCPS) and Visual Analogue Scale (VAS). Psychological symptoms were evaluated using the Hospital Anxiety and Depression Scale (HADS), Patient Health Questionnaire (PHQ-9) and General Anxiety Disorder scale (GAD-7). In TMDM there was a negative correlation between TRP and pain intensity (r The association between KYN/TRP and pain intensity as well as anxiety ratings in this small exploratory study may indicate that KYN/TRP could be a relevant indicator for symptom severity in TMDM and FM. Further investigations of the KYN pathway in chronic myalgia are warranted.

Sections du résumé

BACKGROUND BACKGROUND
Both temporomandibular disorders myalgia (TMDM) and fibromyalgia (FM) have been linked to central and peripheral changes in serotonin availability. The precursor of serotonin, tryptophan (TRP), is mainly catabolised via another pathway to produce kynurenine (KYN), but whether changes of this pathway are present in TMDM and FM are still unclear.
OBJECTIVE OBJECTIVE
The aim was to explore blood plasma concentrations of TRP and KYN in TMDM and FM in an attempt to identify novel associations for future research.
METHODS METHODS
Plasma of 113 female participants (17 TMDM, 40 FM and 56 healthy pain-free controls) were analysed for TRP and KYN concentrations. The degradation of TRP via the KYN pathway was indicated by the KYN to TRP ratio (KYN/TRP). Pain intensities were assessed with the Graded Chronic Pain Scale (GCPS) and Visual Analogue Scale (VAS). Psychological symptoms were evaluated using the Hospital Anxiety and Depression Scale (HADS), Patient Health Questionnaire (PHQ-9) and General Anxiety Disorder scale (GAD-7).
RESULTS RESULTS
In TMDM there was a negative correlation between TRP and pain intensity (r
CONCLUSION CONCLUSIONS
The association between KYN/TRP and pain intensity as well as anxiety ratings in this small exploratory study may indicate that KYN/TRP could be a relevant indicator for symptom severity in TMDM and FM. Further investigations of the KYN pathway in chronic myalgia are warranted.

Identifiants

pubmed: 31545529
doi: 10.1111/joor.12892
doi:

Substances chimiques

Kynurenine 343-65-7
Tryptophan 8DUH1N11BX

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

150-157

Subventions

Organisme : Swedish Rheumatism Association
ID : R861271
Organisme : Stockholm County Council
ID : SLL20180426
Organisme : Karolinska Institutet
ID : 2-281-2017

Informations de copyright

© 2019 John Wiley & Sons Ltd.

Références

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Auteurs

Golnaz Barjandi (G)

Department of Dental Medicine, Karolinska Institutet & Scandinavian Center for Oral Neurosciences, Huddinge, Sweden.

Sofia Louca Jounger (S)

Department of Dental Medicine, Karolinska Institutet & Scandinavian Center for Oral Neurosciences, Huddinge, Sweden.

Monika Löfgren (M)

Department of Clinical Sciences, Karolinska Institutet, Stockholm, Sweden.
Department of Rehabilitation Medicine, Danderyd Hospital, Stockholm, Sweden.

Indre Bileviciute-Ljungar (I)

Department of Clinical Sciences, Karolinska Institutet, Stockholm, Sweden.
Department of Rehabilitation Medicine, Danderyd Hospital, Stockholm, Sweden.

Eva Kosek (E)

Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.
Department of Neuroradiology, Karolinska University Hospital, Stockholm, Sweden.

Malin Ernberg (M)

Department of Dental Medicine, Karolinska Institutet & Scandinavian Center for Oral Neurosciences, Huddinge, Sweden.

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Classifications MeSH