Prospective CYP2C19-Guided Voriconazole Prophylaxis in Patients With Neutropenic Acute Myeloid Leukemia Reduces the Incidence of Subtherapeutic Antifungal Plasma Concentrations.


Journal

Clinical pharmacology and therapeutics
ISSN: 1532-6535
Titre abrégé: Clin Pharmacol Ther
Pays: United States
ID NLM: 0372741

Informations de publication

Date de publication:
03 2020
Historique:
received: 04 06 2019
accepted: 14 09 2019
pubmed: 25 9 2019
medline: 29 10 2020
entrez: 25 9 2019
Statut: ppublish

Résumé

A risk mitigation strategy was implemented to determine if a higher prophylactic voriconazole dosage in patients with CYP2C19 rapid metabolizer neutropenic acute myeloid leukemia (AML) reduces the incidence of subtherapeutic trough concentrations. Patients with AML (n = 263) were preemptively genotyped for CYP2C19*2, *3, and *17 alleles as part of a single-center prospective, interventional, quality improvement study. CYP2C19 rapid metabolizers (CYP2C19*1/*17) were recommended to receive interventional voriconazole 300 mg twice daily, ultrarapid metabolizers (CYP2C19*17/*17) were recommended to avoid voriconazole, and all others received the standard prophylactic dosage of 200 mg twice daily. In this real-world setting, 202 patients (76.8%) were prescribed prophylactic voriconazole, and of these patients 176 (87.1%) received CYP2C19-guided prophylactic dosing. Voriconazole trough concentrations were obtained for 41 of the 58 (70.7%) CYP2C19 rapid metabolizers prescribed prophylactic voriconazole. Interventional voriconazole resulted in higher plasma trough concentrations (median 2.7 μg/mL) compared with the standard prophylactic dosage (median 0.6 μg/mL; P = 0.001). Subtherapeutic concentrations were avoided in 83.8% of CYP2C19 rapid metabolizers receiving interventional dosage compared to 46.2% receiving standard dosage (P = 0.02). CYP2C19 genotyping to preemptively guide prophylactic voriconazole dosing is feasible and may be a potential strategy for reducing the risk of subtherapeutic trough concentrations that potentiate breakthrough fungal infections.

Identifiants

pubmed: 31549389
doi: 10.1002/cpt.1641
pmc: PMC7018540
mid: NIHMS1051680
doi:

Substances chimiques

Antifungal Agents 0
CYP2C19 protein, human EC 1.14.14.1
Cytochrome P-450 CYP2C19 EC 1.14.14.1
Voriconazole JFU09I87TR

Types de publication

Comparative Study Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

563-570

Subventions

Organisme : NCI NIH HHS
ID : P30 CA076292
Pays : United States

Informations de copyright

© 2019 The Authors Clinical Pharmacology & Therapeutics © 2019 American Society for Clinical Pharmacology and Therapeutics.

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Auteurs

J Kevin Hicks (JK)

Department of Individualized Cancer Management, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Rod E Quilitz (RE)

Department of Pharmacy, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Rami S Komrokji (RS)

Department of Malignant Hematology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Timothy E Kubal (TE)

Department of Malignant Hematology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Jeffrey E Lancet (JE)

Department of Malignant Hematology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Yanina Pasikhova (Y)

Department of Pharmacy, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Dahui Qin (D)

Department of Pathology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Wonhee So (W)

Department of Pharmacy, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Gisela Caceres (G)

Department of Pathology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Kerry Kelly (K)

Department of Clinical Informatics, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Yasmina S Salchert (YS)

Department of Pathology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Kevin Shahbazian (K)

Department of Individualized Cancer Management, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Farnoosh Abbas-Aghababazadeh (F)

Department of Biostatistics and Bioinformatics, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Brooke L Fridley (BL)

Department of Biostatistics and Bioinformatics, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Ana P Velez (AP)

Department of Infectious Disease, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

Howard L McLeod (HL)

Department of Individualized Cancer Management, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

John N Greene (JN)

Department of Infectious Disease, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida, USA.

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Classifications MeSH