Effects of selective serotonin reuptake inhibitors on DNA damage in patients with depression.


Journal

Journal of psychopharmacology (Oxford, England)
ISSN: 1461-7285
Titre abrégé: J Psychopharmacol
Pays: United States
ID NLM: 8907828

Informations de publication

Date de publication:
11 2019
Historique:
pubmed: 27 9 2019
medline: 24 7 2020
entrez: 27 9 2019
Statut: ppublish

Résumé

The relationship between depression and increased oxidative stress is well known. DNA damage by oxidation factors is an important cause of the aging process in psychiatric disorders. Owing to the scarcity of human studies and high inconsistencies in studies of the effects of antidepressants on DNA damage, the current study was undertaken to investigate the effects of depression and its treatment on DNA damage. In a 15-week open-label study of citalopram ( DNA damage, 8-OHdG, IL-6 and expression of PARP1 were elevated in patients with depression compared with the healthy controls ( This is the first study on the effect of SSRIs on the DNA damage and some of the repair enzymes in depression. Based on the results, depression can cause increased DNA damage. This damage is followed by activation of compensatory mechanisms whereby the expression of DNA damage repair enzymes is elevated. Finally, the treatment of psychiatric disorder by antidepressants can lower the level of oxidative DNA damage.

Sections du résumé

BACKGROUND
The relationship between depression and increased oxidative stress is well known. DNA damage by oxidation factors is an important cause of the aging process in psychiatric disorders.
AIMS
Owing to the scarcity of human studies and high inconsistencies in studies of the effects of antidepressants on DNA damage, the current study was undertaken to investigate the effects of depression and its treatment on DNA damage.
METHODS
In a 15-week open-label study of citalopram (
RESULTS
DNA damage, 8-OHdG, IL-6 and expression of PARP1 were elevated in patients with depression compared with the healthy controls (
CONCLUSIONS
This is the first study on the effect of SSRIs on the DNA damage and some of the repair enzymes in depression. Based on the results, depression can cause increased DNA damage. This damage is followed by activation of compensatory mechanisms whereby the expression of DNA damage repair enzymes is elevated. Finally, the treatment of psychiatric disorder by antidepressants can lower the level of oxidative DNA damage.

Identifiants

pubmed: 31556787
doi: 10.1177/0269881119874461
doi:

Substances chimiques

Serotonin Uptake Inhibitors 0
Citalopram 0DHU5B8D6V
PARP1 protein, human EC 2.4.2.30
Poly (ADP-Ribose) Polymerase-1 EC 2.4.2.30
DNA Glycosylases EC 3.2.2.-
oxoguanine glycosylase 1, human EC 3.2.2.-
Sertraline QUC7NX6WMB

Types de publication

Comparative Study Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1364-1376

Auteurs

Mahnaz Ahmadimanesh (M)

Department of Pharmacodynamics and Toxicology, Mashhad University of Medical Sciences, Mashhad, Iran.

Mohammad Reza Abbaszadegan (MR)

Immunology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Dorsa Morshedi Rad (D)

Medical Genetics Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Adel Moallem (SA)

Department of Pharmacodynamics and Toxicology, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Pharmacology and Toxicology, Al Zahra University, Karbala, Iraq.

Amir Hooshang Mohammadpour (AH)

Department of Clinical Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Pharmaceutical Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran.

Mohammad Hossein Ghahremani (MH)

Department of Toxicology-Pharmacology, Faculty of Pharmacy, Tehran University of Medical Sciences, Tehran, Iran.

Farhad Farid Hosseini (F)

Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Fatemeh Behdani (F)

Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Ali Akhondpour Manteghi (A)

Psychiatry and Behavioral Sciences Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Paul Jowsey (P)

National Institute for Health Research (NIHR), Health Protection Research Unit for Chemical and Radiation Threats and Hazards, Institute of Cellular Medicine, Newcastle University, Newcastle Upon Tyne, UK.

Fatemeh Shabani Behbahani (F)

Department of Pharmacodynamics and Toxicology, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Mohammad Hassan Moallem (SMH)

School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Leila Etemad (L)

Pharmaceutical Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH