Retrospective Validation of the REVEAL 2.0 Risk Score With the Australian and New Zealand Pulmonary Hypertension Registry Cohort.


Journal

Chest
ISSN: 1931-3543
Titre abrégé: Chest
Pays: United States
ID NLM: 0231335

Informations de publication

Date de publication:
01 2020
Historique:
received: 14 05 2019
revised: 13 08 2019
accepted: 31 08 2019
pubmed: 30 9 2019
medline: 22 7 2020
entrez: 30 9 2019
Statut: ppublish

Résumé

Pulmonary arterial hypertension (PAH) prognosis has improved with targeted therapies; however, the long-term outlook remains poor. Objective multiparametric risk assessment is recommended to identify patients at risk of early morbidity and mortality, and for optimization of treatment. The US Registry to Evaluate Early and Long-Term PAH Disease Management (REVEAL) 2.0 risk score is a new model proposed for the follow-up of patients with PAH but has not been externally validated. The REVEAL 2.0 risk score was applied to a mixed prevalent and incident cohort of patients with PAH (n = 1,011) from the Pulmonary Hypertension Society of Australia and New Zealand (PHSANZ) Registry. Kaplan-Meier survival was estimated for each REVEAL 2.0 risk score strata and for a simplified three-category (low, intermediate, and high risk) model. Sensitivity analysis was performed on an incident-only cohort. The REVEAL 2.0 model effectively discriminated risk in the large external PHSANZ Registry cohort, with a C statistic of 0.74 (both for full eight-tier and three-category models). When applied to incident cases only, the C statistic was 0.73. The three-category REVEAL 2.0 model demonstrated robust separation of 12- and 60-month survival estimates (all risk category comparisons P < .001). Although the full eight-tier REVEAL 2.0 model separated patients at low, intermediate, and high risk, survival estimates overlapped within some of the intermediate- and high-risk strata. The REVEAL 2.0 risk score was validated in a large external cohort from the PHSANZ Registry. The REVEAL 2.0 model can be applied for risk assessment of patients with PAH at follow-up. The simplified three-category model may be preferred for clinical use and for future comparison with other prognostic models.

Sections du résumé

BACKGROUND
Pulmonary arterial hypertension (PAH) prognosis has improved with targeted therapies; however, the long-term outlook remains poor. Objective multiparametric risk assessment is recommended to identify patients at risk of early morbidity and mortality, and for optimization of treatment. The US Registry to Evaluate Early and Long-Term PAH Disease Management (REVEAL) 2.0 risk score is a new model proposed for the follow-up of patients with PAH but has not been externally validated.
METHODS
The REVEAL 2.0 risk score was applied to a mixed prevalent and incident cohort of patients with PAH (n = 1,011) from the Pulmonary Hypertension Society of Australia and New Zealand (PHSANZ) Registry. Kaplan-Meier survival was estimated for each REVEAL 2.0 risk score strata and for a simplified three-category (low, intermediate, and high risk) model. Sensitivity analysis was performed on an incident-only cohort.
RESULTS
The REVEAL 2.0 model effectively discriminated risk in the large external PHSANZ Registry cohort, with a C statistic of 0.74 (both for full eight-tier and three-category models). When applied to incident cases only, the C statistic was 0.73. The three-category REVEAL 2.0 model demonstrated robust separation of 12- and 60-month survival estimates (all risk category comparisons P < .001). Although the full eight-tier REVEAL 2.0 model separated patients at low, intermediate, and high risk, survival estimates overlapped within some of the intermediate- and high-risk strata.
CONCLUSIONS
The REVEAL 2.0 risk score was validated in a large external cohort from the PHSANZ Registry. The REVEAL 2.0 model can be applied for risk assessment of patients with PAH at follow-up. The simplified three-category model may be preferred for clinical use and for future comparison with other prognostic models.

Identifiants

pubmed: 31563497
pii: S0012-3692(19)33934-0
doi: 10.1016/j.chest.2019.08.2203
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Validation Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

162-172

Informations de copyright

Crown Copyright © 2019. Published by Elsevier Inc. All rights reserved.

Auteurs

James J Anderson (JJ)

Advanced Lung Disease Unit, Fiona Stanley Hospital, Perth, WA, Australia; Respiratory Department, Sunshine Coast University Hospital, Birtinya, QLD, Australia. Electronic address: james.anderson2@health.qld.gov.au.

Edmund M Lau (EM)

Department of Respiratory Medicine, Royal Prince Alfred Hospital, Sydney, NSW, Australia; Sydney Medical School, University of Sydney, Camperdown, NSW, Australia.

Melanie Lavender (M)

Advanced Lung Disease Unit, Fiona Stanley Hospital, Perth, WA, Australia.

Raymond Benza (R)

Allegheny General Hospital, Pittsburgh, PA.

David S Celermajer (DS)

Sydney Medical School, University of Sydney, Camperdown, NSW, Australia; Department of Cardiology, Royal Prince Alfred Hospital, Sydney, NSW, Australia.

Nicholas Collins (N)

John Hunter Hospital, Newcastle, NSW, Australia.

Carolyn Corrigan (C)

Heart and Lung Transplant Unit, St Vincent's Hospital, Sydney, NSW, Australia.

Nathan Dwyer (N)

Cardiology Department, Royal Hobart Hospital, Hobart, TAS, Australia.

John Feenstra (J)

Thoracic Medicine, The Prince Charles Hospital, Brisbane, QLD, Australia.

Mark Horrigan (M)

The Austin Hospital, Melbourne, VIC, Australia.

Dominic Keating (D)

Respiratory Medicine, Alfred Hospital, Melbourne, VIC, Australia; Monash University, Melbourne, VIC, Australia.

Fiona Kermeen (F)

Thoracic Medicine, The Prince Charles Hospital, Brisbane, QLD, Australia.

Eugene Kotlyar (E)

Heart and Lung Transplant Unit, St Vincent's Hospital, Sydney, NSW, Australia; University of New South Wales, Sydney, NSW, Australia.

Tanya McWilliams (T)

Greenlane Respiratory Services, Auckland City Hospital, Auckland, New Zealand.

Bronwen Rhodes (B)

Department of Respiratory Medicine, Christchurch Hospital, Christchurch, New Zealand.

Peter Steele (P)

Department of Cardiovascular Services, Royal Adelaide Hospital, Adelaide, SA, Australia.

Vivek Thakkar (V)

Department of Clinical Medicine, Macquarie University, Macquarie Park, NSW, Australia; Department of Rheumatology, Liverpool Hospital, Liverpool, NSW, Australia.

Trevor Williams (T)

Respiratory Medicine, Alfred Hospital, Melbourne, VIC, Australia; Monash University, Melbourne, VIC, Australia.

Helen Whitford (H)

Respiratory Medicine, Alfred Hospital, Melbourne, VIC, Australia; Monash University, Melbourne, VIC, Australia.

Kenneth Whyte (K)

Greenlane Respiratory Services, Auckland City Hospital, Auckland, New Zealand; NZ Respiratory and Sleep Institute, Auckland, New Zealand.

Robert Weintraub (R)

Royal Children's Hospital, Melbourne, VIC, Australia; Murdoch Children's Research Institute, Melbourne, VIC, Australia; University of Melbourne, Melbourne, VIC, Australia.

Jeremy P Wrobel (JP)

Advanced Lung Disease Unit, Fiona Stanley Hospital, Perth, WA, Australia; School of Medicine, University of Notre Dame, Fremantle, WA, Australia.

Anne Keogh (A)

Heart and Lung Transplant Unit, St Vincent's Hospital, Sydney, NSW, Australia; University of New South Wales, Sydney, NSW, Australia.

Geoff Strange (G)

School of Medicine, University of Notre Dame, Fremantle, WA, Australia.

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