Gene signature-MELD score and alcohol relapse determine long-term prognosis of patients with severe alcoholic hepatitis.


Journal

Liver international : official journal of the International Association for the Study of the Liver
ISSN: 1478-3231
Titre abrégé: Liver Int
Pays: United States
ID NLM: 101160857

Informations de publication

Date de publication:
03 2020
Historique:
received: 18 06 2019
revised: 03 09 2019
accepted: 19 09 2019
pubmed: 1 10 2019
medline: 22 6 2021
entrez: 1 10 2019
Statut: ppublish

Résumé

The gene-signature-model for end stage liver disease (gs-MELD) score has been shown to be a strong predictor of 6-month survival in severe alcoholic hepatitis (AH). Currently, only a few studies have evaluated the long-term prognosis of patients with severe AH. To assess the prognostic value of the gs-MELD score at 5 years in patients with severe AH. Forty-eight consecutive patients with AH (25 males, median age 52 years [95% IC: 48-56]) were included. The median gs-MELD score was 2.6 (95% CI: 2.2-3.0). According to the gs-MELD score, 22 patients (46%) were considered to have a poor prognosis. During a median follow-up of 29 months (95% CI: 4-43), 19 patients (40%) were abstinent and 24 patients (50%) died. At 5 years, rates of survival were 61% (95% CI: 41-81) and 26% (95% CI: 11-55) in patients with low and high gs-MELD scores (P = .001), and 81% (95% CI: 58-96) and 22% (95% CI: 10-47) in abstainers and in consumers (P < .001) respectively. In multivariable competing risk regression modelling, gs-MELD score (subdistribution hazard ratio: 5.78, 95% CI: 2.17-15.38, P < .001) and recurrent alcohol consumption (subdistribution hazard ratio: 12.18, 95% CI: 3.16-46.95, P < .001) were independently associated with 5-year mortality. Both gs-MELD score and alcohol consumption drive AH long-term prognosis. The gs-MELD score may guide the development of molecularly targeted therapies in AH.

Sections du résumé

BACKGROUND
The gene-signature-model for end stage liver disease (gs-MELD) score has been shown to be a strong predictor of 6-month survival in severe alcoholic hepatitis (AH). Currently, only a few studies have evaluated the long-term prognosis of patients with severe AH.
AIM
To assess the prognostic value of the gs-MELD score at 5 years in patients with severe AH.
METHODS
Forty-eight consecutive patients with AH (25 males, median age 52 years [95% IC: 48-56]) were included.
RESULTS
The median gs-MELD score was 2.6 (95% CI: 2.2-3.0). According to the gs-MELD score, 22 patients (46%) were considered to have a poor prognosis. During a median follow-up of 29 months (95% CI: 4-43), 19 patients (40%) were abstinent and 24 patients (50%) died. At 5 years, rates of survival were 61% (95% CI: 41-81) and 26% (95% CI: 11-55) in patients with low and high gs-MELD scores (P = .001), and 81% (95% CI: 58-96) and 22% (95% CI: 10-47) in abstainers and in consumers (P < .001) respectively. In multivariable competing risk regression modelling, gs-MELD score (subdistribution hazard ratio: 5.78, 95% CI: 2.17-15.38, P < .001) and recurrent alcohol consumption (subdistribution hazard ratio: 12.18, 95% CI: 3.16-46.95, P < .001) were independently associated with 5-year mortality.
CONCLUSIONS
Both gs-MELD score and alcohol consumption drive AH long-term prognosis. The gs-MELD score may guide the development of molecularly targeted therapies in AH.

Identifiants

pubmed: 31568650
doi: 10.1111/liv.14265
pmc: PMC7056530
mid: NIHMS1052830
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

565-570

Subventions

Organisme : European Research Council
ID : 671231
Pays : International
Organisme : NCI NIH HHS
ID : R01 CA233794
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK099558
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2019 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

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Auteurs

Pierre Deltenre (P)

Department of Gastroenterology, Hepatopancreatology, and Digestive Oncology, C.U.B. Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.
Department of Gastroenterology and Hepatology, Clinique St Luc, Bouge, Belgium.

Eric Trépo (E)

Department of Gastroenterology, Hepatopancreatology, and Digestive Oncology, C.U.B. Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.
Laboratory of Experimental Gastroenterology, Université Libre de Bruxelles, Brussels, Belgium.

Naoto Fujiwara (N)

Division of Digestive and Liver Diseases, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.

Nicolas Goossens (N)

Division of Digestive and Liver Diseases, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Division of Gastroenterology and Hepatology, Geneva University Hospital, Geneva, Switzerland.

Astrid Marot (A)

Department of Gastroenterology and Hepatology, CHU UCL Namur, Université Catholique de Louvain, Yvoir, Belgium.

Margaux Dubois (M)

Division of Gastroenterology and Hepatology, Centre Hospitalier Universitaire Vaudois, University of Lausanne, Lausanne, Switzerland.

Laurent Spahr (L)

Division of Gastroenterology and Hepatology, Geneva University Hospital, Geneva, Switzerland.

Jean Henrion (J)

Department of Gastroenterology and Hepatology, Hôpital de Jolimont, Haine-Saint-Paul, Belgium.

Christophe Moreno (C)

Department of Gastroenterology, Hepatopancreatology, and Digestive Oncology, C.U.B. Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.
Laboratory of Experimental Gastroenterology, Université Libre de Bruxelles, Brussels, Belgium.

Yujin Hoshida (Y)

Division of Digestive and Liver Diseases, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.

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Classifications MeSH