Rotational symmetry of the structured Chip/LDB-SSDP core module of the Wnt enhanceosome.


Journal

Proceedings of the National Academy of Sciences of the United States of America
ISSN: 1091-6490
Titre abrégé: Proc Natl Acad Sci U S A
Pays: United States
ID NLM: 7505876

Informations de publication

Date de publication:
15 10 2019
Historique:
pubmed: 2 10 2019
medline: 31 3 2020
entrez: 2 10 2019
Statut: ppublish

Résumé

The Chip/LIM-domain binding protein (LDB)-single-stranded DNA-binding protein (SSDP) (ChiLS) complex controls numerous cell-fate decisions in animal cells, by mediating transcription of developmental control genes via remote enhancers. ChiLS is recruited to these enhancers by lineage-specific LIM-domain proteins that bind to its Chip/LDB subunit. ChiLS recently emerged as the core module of the Wnt enhanceosome, a multiprotein complex that primes developmental control genes for timely Wnt responses. ChiLS binds to NPFxD motifs within Pygopus (Pygo) and the Osa/ARID1A subunit of the BAF chromatin remodeling complex, which could synergize with LIM proteins in tethering ChiLS to enhancers. Chip/LDB and SSDP both contain N-terminal dimerization domains that constitute the bulk of their structured cores. Here, we report the crystal structures of these dimerization domains, in part aided by DARPin chaperones. We conducted systematic surface scanning by structure-designed mutations, followed by in vitro and in vivo binding assays, to determine conserved surface residues required for binding between Chip/LDB, SSDP, and Pygo-NPFxD. Based on this, and on the 4:2 (SSDP-Chip/LDB) stoichiometry of ChiLS, we derive a highly constrained structural model for this complex, which adopts a rotationally symmetrical SSDP

Identifiants

pubmed: 31570581
pii: 1912705116
doi: 10.1073/pnas.1912705116
pmc: PMC6800368
doi:

Substances chimiques

DNA-Binding Proteins 0
LDB1 protein, human 0
LDB2 protein, human 0
LIM Domain Proteins 0
Multiprotein Complexes 0
SSBP3 protein, human 0
Transcription Factors 0
Wnt Proteins 0

Banques de données

PDB
['6S9R', '6D9T', '6S9S']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

20977-20983

Subventions

Organisme : Medical Research Council
ID : MC_U105192713
Pays : United Kingdom
Organisme : Medical Research Council
ID : U105192713
Pays : United Kingdom
Organisme : Cancer Research UK
ID : C7379/A24639
Pays : United Kingdom
Organisme : Cancer Research UK
ID : C7379/A15291
Pays : United Kingdom

Informations de copyright

Copyright © 2019 the Author(s). Published by PNAS.

Déclaration de conflit d'intérêts

The authors declare no competing interest.

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Auteurs

Miha Renko (M)

Medical Research Council Laboratory of Molecular Biology, CB2 0QH Cambridge, United Kingdom.

Marc Fiedler (M)

Medical Research Council Laboratory of Molecular Biology, CB2 0QH Cambridge, United Kingdom.

Trevor J Rutherford (TJ)

Medical Research Council Laboratory of Molecular Biology, CB2 0QH Cambridge, United Kingdom.

Jonas V Schaefer (JV)

Department of Biochemistry, University of Zurich, 8057 Zurich, Switzerland.

Andreas Plückthun (A)

Department of Biochemistry, University of Zurich, 8057 Zurich, Switzerland.

Mariann Bienz (M)

Medical Research Council Laboratory of Molecular Biology, CB2 0QH Cambridge, United Kingdom; mb2@mrc-lmb.cam.ac.uk.

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Classifications MeSH